What is pathophysiology of Prostate cancer?

['مڠهادڤي لاڤورن اين']

ڤاتوڤيسيوولوڬي كسرطان پروستات اداله اڤ؟

Pathophysiology of prostate cancer merujuk kepada mekanisme dan proses yang mendasari yang mengarah pada perkembangan dan perkembangan penyakit.

سرطان پروستات اداله تومور خبيث يڠ برمولا دري سلوله غدة پروستات، يڠ اداله اورڬان يڠ ڤيڠيت، بربنديڠ كاج يڠ دتڠڬهكن د سڬالا كيس دالم اورڠ.

غدة البروستاتا مڠنتوڠي سيليۏيد سيمين، يڠ ممڤوپاڠي دان ممڤرتنڤقكن سڤرم.

سبب يڠ tepat dari kanker prostat tidak sepenuhnya dipahami, tapi beberapa faktor diketahui untuk meningkatkan risiko mengembangkan penyakit.

ڽن ترماسوق umur, sejarah keluarga, ras, dan mutasi genetik tertentu.

سرطان پروستات بياساڽ دالم اورڠ يڠ لاما، دڠن ماجوريتي كيس دجاديكن دالم اورڠ يڠ لبيه دري 65 تاهون.

سلاين ايت، اورڠ٢ يڠ اد سجاره كسر ڤروستات دالم كاوسن كاوسنڽ، سام اد اورڠ٢ افريقي امريك دان اورڠ٢ منچاري كريبي.

ڤاتوڤيسيوولوڬي كسرن ڤروستات مڠورڠكن ڤمباڠونن دان ڤمباڠونن سلول يڠ تيدق دڤڠاروه دالم غدة ڤروستات.

ڤميوتاسي اين موڠكين ممباوا كبواتأن سل يڠ تيدق دڤرچايا، ممباوا كڤد ڤمبنتوق تومور.

كاتيك تومور برومڤا، اي بوليه مڠهادڤي تييسو دان اورڬان يڠ ڤاليڠ ڤاليڠ، سڤرتي بولڠ، ريكتوم، دان غدد ليمف يڠ ڤاليڠ ڤاليڠ.

دالم ببراڤ كيس، sel kanker بوليه برڤاڬاي دري tumor primer دان برڤاڬاي كڤد باڬي-باڬي لاين درڤد جالن darah اتاو sistem limfatik، ساتو ڤروسيس يڠ دكنلي سباڬاي metastasis.

ستله بسرڽ مڠمبيل، موڠكين لبيه سوده دڤلاجري.

سرطان پروستات توروت دتاثيركن اوليه فاكتور هورمون، خصوصڽ هورمون اندروجين تيستوسترون.

تستوسترون موڠكين ممڤوڽاءي ڤمبڠكڠن سيل بسر ڤروستات، دان باڽق ڤڠڬوناءن بسر ڤروستات مڠهدڤي ممڤوڽاءي ڤڠوربانن هرمون اين اتاو ممڤوڽاءي ايكوڽ.

باختصار، patofisiologi kanker prostat melibatkan pertumbuhan dan pembelahan sel yang tidak terkontrol di dalam kelenjar prostat, yang dapat dipengaruhi oleh faktor genetik, hormonal, dan lingkungan.

ڤمهمن ميكنزم ڤاسر كريتاڽ اداله ڤنتيڠ اونتوق ممباڠونكن ڤلاجرن يڠ ايكفيكتيف دان ممڤربيتاهوكن كاوسن اونتوق مريک دڠن كسر ڤروستات.

['ريفرنس']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Zobniw CM, Causebrook A, Fong MK: Clinical use of abiraterone in the treatment of metastatic castration-resistant prostate cancer. Res Rep Urol. 2014, 6 (): 97-105.

Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.

Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.

Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.

Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.

Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.

Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.

['ڤڠهادڤن: ميديك']

['ويب سايت اين دبريكن اونتوق ڤڠاجرن دان معلومت سهيڠڬ دان تيدق ممڤوڽاءي نصيحت ميديكل اتاو ڤرخدمتن ڤروفيسيونل.']

['معلومت يڠ دبريكن تيدق بوليه دڬوناكن اونتوق ممڤوڽاءي اتاو ممباوا ڤروبليم كصيحتن اتاو كمرضن، دان اورڠ-اورڠ يڠ ممڤوڽاءي نصيحت ميديكل ڤرسوداران بوليه مڠيكوت دوكتور يڠ مڠيكوت.']

['سوده دتنتوكن، jaring saraf يڠ ممبنتو جواڤن كڤد سوألن، تيدق ساڠت ڤاليڠ ڤاليڠ ڤاليڠ ڤاليڠ ڤاليڠ كتيک دتڠكڤ كڤد كونتينن نوميريک. سڤرتي، ڤندودوق اورڠ يڠ دڤرچاياڬوسكن دڠن كمرڬانن خاص.']

['Slalu seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. Jika anda pikir anda mungkin mengalami keadaan darurat medis, hubungi 911 atau pergi ke ruang gawat darurat terdekat segera. Tidak ada hubungan dokter-pasien yang dibuat oleh situs web ini atau penggunaannya. BioMedLib maupun karyawannya, maupun kontributor situs web ini, tidak membuat representasi apapun, secara eksplisit atau implisit, sehubungan dengan informasi yang disediakan di sini atau penggunaannya. BioMedLib tidak akan memberikan jaminan, baik secara eksplisit maupun implisit, sehubungan dengan informasi yang disediakan di sini atau penggunaannya. BioMedLib tidak akan memberikan jaminan, baik secara eksplisit maupun implisit, sehubungan dengan informasi yang disediakan di sini atau penggunaannya. BioMedLib tidak akan memberikan jaminan, baik secara eksplisit maupun implisit, sehubungan dengan informasi yang disediakan di sini atau penggunaannya.']

['ڤڠهادڤن: حق ڤنوليس']

['Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the DMCA) ممبري ڤڠاجرن اونتوق ڤميمڤين حق ڤنوليس يڠ مڠڬنتيكن بهاوا ماتريال يڠ دڤرچايا?? ي د انتارنيت مڠڬوناكن حقڽ دالم ڤرلمباڬان حق ڤنوليس اميريكا.']

['اڤابيلا ديري مڠڬنتيكن دڠن كصيحتن يڠ باءيق بهاوا ستياڤ كونتين اتاو ماتريال يڠ دڤاوڤونكن دڠن ڤڠڬوناان ويب اتاو ڤرخدمتن ديري مڠڬوناكن حق ڤنوليسن ديري، ديري (اتاو اڬين ديري) موڠكين مڠوروسكن كيت ساتو كپاتا ن مڠمبيل ستياڤ كونتين اتاو ماتريال دڤاوڤون اتاو كلوارڬ كيت.']

['ڤڠاجرن هاروس دجوال دڠن توليسن ملالوءي ايميل (تڠه سيكشن "Kontakt" اونتوق ايديريس ايميل)']

['DMCA ممرلوكن ڤڠاجرن ڤڠهيناان حق ڤنوليسن ترماسوق معلومت يڠ منوروت: (1) ڤنوليسن كراجا?? ن حق ڤنوليسن يڠ منجادي موضوع ڤڠهيناان ترماسوق معلومت (2) ڤنوليسن كونتينن يڠ ترماسوق ڤڠهيناان دان معلومت يڠ ممڤرلوكن كيت منچاري معلومت (3) معلومت ڤڠاتوهن كيت ترماسوق ايدريس، نومبر تيليفون دان ايدريس ايميل (4) كپاتا?? ن كيت يڠ ممڤرلوكن كڤد كيت كران كيت برهارڤ ڤڠهيناان يڠ دڤنوليسكن تيدق دأوتوريساسي اوليه ڤملكن حق ڤنوليسن اتاو اڬينڽ اتاو اوليه ڤڠڬونا?? ن ايستري.']

['(5) pernyataan awak, ditandatangani di bawah hukuman sumpah palsu, bahwa informasi dalam pemberitahuan adalah akurat dan awak memiliki wewenang untuk menegakkan hak cipta yang diklaim dilanggar; ']

['ڠن (6) ساتو ڤنوليسن فيزيک اتاو ايليکترونيک دري ڤميمڤين حق اوڤاي اتاو سأورڠ يڠ دأذنکن اونتوق مڠݢوناکن ڤميمڤين حق اوڤاي. ']

['كران تيدق ممڤوڽاءي سموا معلومت يڠ دڤرتوان اكن ممباوا كڤد ڤڠڬوناان كڤد كڤدأنن.']

['ڤرتوبوهن']

['Tulung kirimi kami email dengan pertanyaan/usulan apapun.']

What is pathophysiology of prostate cancer?

The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.

Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.

The prostate gland produces seminal fluid, which nourishes and transports sperm.

The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.

These include age, family history, race, and certain genetic mutations.

Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.

Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.

The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.

This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.

These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.

As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.

In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.

Once the cancer has spread, it can be more difficult to treat.

Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.

Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.

In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.

Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.

Disclaimer: medical

This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.

The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.

Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.

Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.

Disclaimer: copyright

The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.