What is pathophysiology of Heart attack?

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Kunas ataque cardíaco ukan fisiopatología ukaxa?

Infarto de miocardio sat sutinchata infarto de miocardio ukan fisiopatología ukax janchijan mä chiqapar wila puriñ jark't'i, ukatx células del corazón ukanakaw jiwi.

Akax mä arterias coronarias ukan mä coágulo de sangre ukan jark'antatapata, ukax aterosclerosis ukan uñsti, uka usux placas ukanakaw arterias ukanakanx jilxati.

Uka placanjja, colesterol, materias grasas, células de desecho, calcio ukat fibrina ukanakaw utji.

Kunapachati uka placax phallki ukhaxa, mä coágulo de sangre ukaw utji, ukasti arteriarux jark'xäspawa, ukat oxigeno-mp phuqt'at wilax janiw músculo cardíaco ukar purkaspati.

Aka oxigeno pist'awinxa, células musculares del corazón ukanakaxa jiwarawayxiwa, ukatsti infarto de miocardio ukaruw puriyi.

Kunjam usuchjatas jikjjatasini ukjja, kawkhakamas arteriasajj jan waltʼki ukat qhawqha tiempos usutäjje ukampiw yatjjatapjjani.

Chuyma usuxa puraka usumpi, janchi usumpi, samsuñ usumpi, chuyma usumpi, ch'usasiñampi, amparanakana, kunka, laka, qhipäxana usumpiw utjaraki.

Usutapatjja, jankʼakiw wilajj wasitat chuymar mantani, ukatakisti qollirinakaw utji.

Wali wakiskiriwa amuyaña fisiopatología de un ataque cardíaco ukax wali ch'amawa ukatx walja factores ukanakaw utji, genéticos, estilo de vida ukat factores ambientales ukanaka.

Chuyma usu (infarto) puriñatakix wilancha (presión) usu, colesterol usu, cigarro usu, diabetes usu, lik'i usu, jan ch'ama usu, wila masinakans chuyma usu utjatapaw utji.

Aka jan walt'äwinakax mä ataque cardíaco ukamp jan aynacht'aykaspati.

['Qillqatanaka']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.

Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .

Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.

LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.

Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.

Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.

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["5) jumati aka yatiyawx chiqpachapun yatiyatama, ukhamarak uka derechos de autor ukanakxat arxatirjamax janiw chiqaparux arxatktati sasin sañatak mä arsüwi qillqantäta, ukarux k'ari juramentompiw phuqhatani."]

['Ukjamaraki (6) derechos de autor ukanin sutinakapamp qillqantat chimpupa jan ukax electrónica chimpupa.']

["Janitix taqi uka yatiyawinakax qillqatäkanixa, janiw jank'as jank'as jan walt'awix askichatäkaniti."]

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What is pathophysiology of heart attack?

The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.

This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.

The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.

When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.

This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.

The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.

Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.

Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.

It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.

Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.

Managing these risk factors can help reduce the likelihood of experiencing a heart attack.

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