علائم حمله قلبي مي تواند شامل درد قفسه سينه يا ناراحتي، تنگی نفس، حالت تهوع، سرگيجه و درد در بازو، گردن، فک يا پشت باشد.
ڤلاجرن ڤڠاجرن jantung biasanyamemaksudkan memulihkan aliran darah ke otot jantung secepat mungkin, baik melalui pengobatan atau prosedur seperti angioplasty dan stenting atau operasi bypass arteri koroner.
فاكتور-فاكتور ريسيكو انتاك jantung termasuk tekanan darah tinggi, kolesterol tinggi, merokok, diabetes, obesity, kurangnya aktivitas fisik, dan riwayat keluarga penyakit jantung.
ڤڠاجرن فکتور-فکتور risiko ngini kawa membantu mengurangi kemungkinan mengalami serangan jantung.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['ڤڠاجرن: حق ڤنوليس']
['Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the DMCA) ممبري ڤڠاجرن كڤد ڤڠڬونا حق ڤنوليسن يڠ مڠڬنتيكن ماتريال يڠ دڤرچايا?? ي د انتارنيت مڠڬوناكن حقڽ دالم ڤرلمباڬان حق ڤنوليسن اميريكا.']
['(5) بيانيه اي از طرف شما، با امضاي شما تحت مجازات شهادت دروغين، که اطلاعات در اطلاعيه دقيق است و شما اختيار داريد حقوق كپي رايت را كه ادعا مي شود نقض شده است، اجرا كنيد؛']
['ڠڬوناكن ڤڠڬوناءن حق ڤنوليس اتاو اورڠ يڠ دڤرلوكن اونتوق مڠڬوناكن ڤڠڬوناءن حق ڤنوليس.']
['Tulung kirimi kami surel dengan apa jua pertanyaan/cadangan.']
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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