Urtikaria, biasanna risseng sebagai urtikaria, iyanaritu keadaang kulit iya ritanrai pole cumpa'na makkeccik, menre', makkalolo' macella (wheals) ri kulit.
Pathophysiology urticaria muttama'i leppessangngi histamine nenniya mediator inflamasi laingnge pole ri sel mast, iyanaritu sel imun iya riruntu'e ri laleng kulit nenniya jaringan laingnge.
Ri urtikaria akut, leppessangngi histamin nenniya mediator laingnge ripancaji pole reaksi alergi lao ri alergen mattentu, pappada anreang, pabbura, iyarega peddi serangga.
Iyae nassabari sel mast untu' degranulate, mappallebbang histamine nenniya mediator laingnge iya nassabari sumange' dara mancaji malabbiri, nassabari bentu'na roda.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
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Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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