Pathophysiology nyau prostate ɗon nufa dabareji be dabareji je ɗon yahda be yahde nyau ndu.
Kansaru prostate lati nyau je ɗon ummina ɓikkoy ɓaandu je ɗon mari gland prostate, je lati bana ɓaandu pamarron je ɗon mari irin walnut je ɗon les ɓernde ɓii fuffu ha worɓe.
Prostate ɗon wurtina ndiyam iyam, je ɗon hokka nyamdu e ɗon yahida be iyam.
Dalila je ɗon hokka nyau prostate je ɗon mari nyau man famɗa, amma kujeji jur ɗon aandi ngam ɓesdugo sababu heɓugo nyau man.
Ndu larni duɓɓi, tariya iyalji, laral, be fe'itolji tagdi feere.
Kansaru prostate ɓuri nyaɗugo ha nder worɓe je ɗon mari duuɓi, ɓurna fu ɗon wara ha nder worɓe je mari duuɓi 65.
Fahin bo'o, worɓe je mari tariya nyau man ha nder iyalji maɓɓe ɗon mari sababu jur, bana non bo'o worɓe African American be worɓe je ɗon mari asli Caribbean.
Pathophysiology cancer prostate ɗon larna jahol be sandirol cellji je ɗon wala hakkilgo ha nder prostate.
Ndu footi wona ngam saanjolji tagdi je ɗon hokka sababuji maunugo feere mala darnugo tagdi je ɗon darna gilɗi.
Tagle ndu footi waɗa ɓesdugo taklon je wala dabare, je footi waɗa no ɓundu fuɗɗata.
Bana no ɓundu ɗon mawna, footi memma ɓaandu be kujeji je ɗon ha ɓada, bana ɓuulol ɓaandu, rectum, be lymph nodeji je ɗon ha ɓada.
Ha wakati feere, ɓikkoy ɓaandu cancer footi salla diga fuɗɗam ɓaandu e sankita ha wakkereji ɓaandu feere gal iyam mala dabare lymphatic, yahde je ɗon aandin bana metastasis.
To nyau man sankiti, footi lati saɗirma hurgugo.
Nyau prostate footi bo'o memma gal kujeji je ɗon hokka ɓaandu, ɓurna fu androgen hormone testosterone.
Testosterone footi ummina jahol ɓikkoy cancer prostate, e hurgolji ɗuɗɗum ngam cancer prostate ɗon ɗaɓɓa ustugo yahde hormone ndu mala darnugo kuɗe maɗa.
Ha nder limtol, pathophysiology cancer prostate larni jahol be sandirol cellji je wala aynol ha nder prostate, je footi memma gal sababuji renol, hormonji be wakkere.
Fahmugo dabareji nyau ndu lati kanduɗum ngam wurtinugo hurgolji je ɗon naffa e yahde nyauɓe je mari nyau prostate.
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Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.
Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.
Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.
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['(5) matinolji je on windi, je on windi ha les bone seedugo fewre, je matinolji ha nder matinolji man lati goonga e je on mari baawɗe ngam aynugo hakkeji je mbiyotto ɗon luuta;']
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What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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