Maladi alzayme a se yon pwogresis maladi neurodegenerative ki afekte sèvo a, ki lakòz pèt memwa, bès mantal, ak chanjman konpòtman.
Yo pa fin byen konprann sa ki lakòz maladi alzayme a, men yo kwè gen yon seri bagay ki makònen ak jenetik, anviwònman ak fason moun viv.
Gen kèk faktè ki ka kontribye nan devlopman maladi alzayme a:
1. Jenetik: Yo idantifye sèten jèn ki ogmante risk pou yon moun gen maladi alzayme a, sitou jèn ki rele apolipoprotein E (APOE) a.
2. Laj: Risk pou yon moun gen maladi alzayme a ogmante avèk laj, e pifò moun ki gen maladi sa a se apre yo fin gen 65 an.
3. Istwa fanmi: Si gen moun nan fanmi w ki te gen maladi alzayme a, sa ka fè w gen plis chans pou w vin gen maladi sa a tou.
4. Blesi nan tèt: Si yon moun te konn gen yon seri blesi nan tèt li, sitou sa ki lakòz li pèdi konsyans, sa ka fè li gen plis chans pou l gen maladi alzayme a.
5. Faktè ki ka fè moun gen maladi kè ak nan venn: Gen kèk maladi tankou tansyon wo, kolestewòl ki wo ak dyabèt ki ka fè moun gen plis chans pou yo gen maladi alzayme a.
6. Fason moun viv: Lè yon moun chita, lè li pa manje byen e lè li pa gen bonjan aktivite nan lespri l, sa ka fè li gen plis chans pou l gen maladi alzayme a.
7. Enflamasyon: Yon enflamasyon kwonik nan sèvo a ka fè moun vin gen maladi alzayme a.
8. Estrès ki lakòz maladi ki rele " oxidative stress ": Lè gen yon move balans ant pwodiksyon radikal lib yo ak kapasite kò a pou l elimine yo, sa ka fè moun nan vin gen maladi alzayme a.
9. plak amiloyid ak anmele neurofibrillary: akimilasyon plak amiloyid ak anmele neurofibrillary nan sèvo a se yon mak de maladi alzayme a, men wòl yo jwe nan devlopman kondisyon an pa konprann nèt.
Li enpòtan pou note ke kòz maladi alzayme a konplèks e li gen anpil chans pou li enplike yon konbinezon faktè sa yo.
Rechèch ap kontinye pou konprann pi byen mekanis ki kache yo epi devlope tretman efikas pou kondisyon devastatè sa a.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
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['(5) yon deklarasyon ou siyen sou penalite fo temwayaj ke enfòmasyon ki nan notifikasyon an egzat e ke ou gen otorite pou fè respekte dwa otè yo ke yo reklame ke yo vyole yo;']
['e (6) yon siyati fizik oubyen elektwonik mèt copyright la oubyen yon moun ki otorize pou aji sou non mèt copyright la. ']
['Si ou pa bay tout enfòmasyon ki anwo yo, sa ka fè ke nou pa trete plent ou a byen vit.']
['Kontak']
['Tanpri voye nou yon imèl avèk nenpòt kesyon / sijesyon.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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