Alzheimer ana gaw, shawng nnan na bawnu hpe hkra ai, dumn'ta hten ai, myit n'gun yawm ai hte akyang lailen galai shai ai lam ni byin shangun ai, rawt jat wa ai neurodegenerative ana re.
Alzheimer ana byin ai lam hpe atsawm n chye lu tim, masha a aru arat, shingra masa hte asak hkrung lam a majaw byin ai ngu sawn la ma ai.
Alzheimer ana byin wa hkra galaw ya ai lam ni gaw:
1. Htinggaw hpan (genetics): Alzheimer ana byin wa hkra grau sawng shangun ai, lawu na lam ni hpe chye lu ai.
Asak: Asak kaba wa ai hte maren, Alzheimer ana byin chye ai lam grau grau law wa nna, masha law malawng gaw asak 65 ning jan ai hpang hta, dai ana hpe mu lu ai.
3. Dinghku hta ana byin ai lam: Dinghku hta ana byin ai lam nga yang, ana byin chye ai lam grau law wa chye ai.
4. Baw hta hkala ai lam: Labau hta baw hta hkala ai lam byin wa ai, grau nna myit n mat ai lam byin wa ai gaw, Alzheimer ana byin wa na matu grau shoihpa nga ai.
5. Myit masin ana byin chye ai lam: Sai rawt ai, sai hta cholesterol law ai hte, ana zinli byin chye ai.
6. Hkrun lam masa: Hkrun lam hta n hkawm ai, n hkru ai lusha ni sha ai, myit n rawt ai lam ni gaw, Alzheimer ana byin wa hkra grau galaw ya chye ai.
7. Hkum hkrang machyi ai lam: Myit hta ana byin ai lam gaw, Alzheimer ana byin wa hkra galaw ya chye ai.
8. Oxidative stress: Free radical ni hpe hkum hkrang hta n nga hkra galaw ai majaw, dai ni hpe lu sha kau ya ai lam n galaw ai majaw, Alzheimer ana byin wa chye ai.
Amyloid plaque hte neurofibrillary tangles: Amyloid plaque hte neurofibrillary tangles ni gaw, baw hta law htam wa ai majaw, Alzheimer ana byin wa ai.
Alzheimer ana byin wa ai lam gaw, ndai lam ni a majaw byin wa ai re.
Ndai ana a majaw byin ai lam hpe grau chye na lu na matu hte dai hpe tsi lajang lu na matu, matut nna sawk sagawn nga ai.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['N hkap la ai lam: tsi mawan']
['Ndai website gaw hpaji jaw ai hte chye na matu sha galaw da ai.']
['Ndai laika buk hta lawm ai lam ni hpe hkamja lam hte seng nna, ana jep ai (sh) tsi ai lam hta n mai jai lang ai.']
['Ga san ni a mahtai hpe shapraw ya ai neural net gaw, ga shadawn, masha langai ngai hta ana langai ngai mu lu ai lam ni hpe madun ai shaloi, n hkrak ai.']
['Ndai website hta mu lu ai lam ni a majaw, tsi sarawun ni a hpaji jaw ga hpe galoi mung n madat ai sha, hpang hkrat ai lam n galaw u. Tsi hte seng nna, ra kadawn nga ai lam nga yang, 911 hpe shaga u. Ndai website hte dai hpe lang ai lam gaw, tsi sarawun hte machyi masha lapran hku hkau lam n nga ai. BioMedLib hte shi a bungli galaw masha ni, ndai website hta shang lawm ai ni kadai mung, ndai kaw lawm ai shiga hte seng nna, tsun mayu ai lam (sh) tsun ai hku nna, hpa ga sadi jaw ai lam n nga ai.']
['Hti na ahkang n nga ai: copyright']
['1998 ning Digital Millennium Copyright Act, 17 U.S.C. § 512 (the DMCA) gaw Internet kaw mara da ai lam ni gaw US copyright law npu na madu a ahkaw ahkang hpe tawt lai ai ngu kam ai copyright madu ni hpe ahkaw ahkang jaw da ai.']
['Anhte a website hte seng nna, (sh) anhte a magam bungli ni hte seng nna, mara shagun da ai lam ni gaw, na a copyright hpe tawt lai ai ngu nna nang kam ai nga yang, dai lam ni hpe dawm kau na matu (sh) dai ni hpe n mai lu hkra pat kau na matu, nang (sh) na a kasa gaw anhte hpe shana mai ai.']
['Dai shiga ni hpe laika hte ka nna email hte shagun ra ai (email address hpe "Contact" daw kaw mu lu na re).']
['Dai DMCA gaw, na a copyright tawt lai ai lam hpe shana ai hta lawu na lam ni lawm ra ai: (1) tawt lai ai ngu ai copyright lu ai bungli a lam; (2) tawt lai ai ngu ai lam hte seng ai lam hte dai hpe mu tam lu na matu anhte hpe karum ya ai lam; (3) na a matut mahkai lam, na a shara, phone number hte email hte seng ai lam ni; (4) nang mara shagun ai lam gaw copyright madu, shi a agent (sh) tara upadi hte n seng ai ngu ai hpe nang kam ai lam.']
['(5) N teng n man ai sakse hkam ai lam hte seng nna, nang masat da ai hte maren, shana ai lam hta lawm ai shiga ni gaw teng man ai hte, nang tawt lai ai ngu ai copyright hpe hkan sa na ahkang nga ai lam.']
['hte (6) copyright madu a mying hte galaw na ahkang lu ai wa a hkum hkrang (sh) electronic signature.']
['Lahta na lam ni hpe n ka bang ai rai yang, nang shagun ai laika hpe hpang hkrat ai hte n mai htang ai.']
['Matut Mahkai U']
['Gara hku mung san mayu yang email hte shana ya rit.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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Disclaimer: copyright
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['Hkrang']
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['Anhte gaw PubMed/Medline kaw na tsi hpaji hte seng ai laika buk wan 35 hte hpang ai. RefinedWeb kaw na mung re.']
