Tuwo mar Alzheimer en tuwo mar obwongo ma hinyo ng'ato ahinya, kendo mano miyo ng'ato ok nyal paro gik moko e yo maber, ok onyal ng'eyo gik moko e yo maber, kendo ok onyal timo gik moko e yo maber.
Ok ong'ere malong'o gimomiyo ng'ato bedo gi tuwo mar Alzheimer, kata kamano, ji paro ni tuwo mar Alzheimer en nikech gik mayudore e anyuola, chal mar alwora, kod kit ngima ng'ato.
Moko kuom gik manyalo miyo ng'ato obed gi tuwo mar Alzheimer gin kaka:
1. Genetics: Oseng'ere ni nitie gik moko ma miyo ng'ato bedo gi tuwo mar Alzheimer's, to moloyo gino miluongo ni apolipoprotein E (APOE).
2. Higni: Nyalo mar bedo gi tuwo mar Alzheimer medore kaka ng'ato medo bedo moti, kendo thoth ji yudo tuwo mar Alzheimer bang' bedo jahigini 65.
3. Weche motudore gi tuwo mar Alzheimer e anyuola: Bedo gi weche motudore gi tuwo mar Alzheimer e anyuola nyalo miyo ng'ato obed gi nyalo mar bedo gi tuwo mar Alzheimer.
4. Hinyruok ma ng'ato nyalo yudo e wiye: Hinyruok ma ng'ato nyalo yudo e wiye, to moloyo hinyruok ma ng'ato nyalo yudo ka ok ong'eyo, nyalo miyo ng'ato obed gi tuwo mar Alzheimer.
5. Tuwo mag adundo: Tuwo kaka high blood pressure, high cholesterol, kod diabetes nyalo miyo ng'ato obed gi tuwo mar Alzheimer.
6. Kit ngima: Ngima ma ng'ato ok nyal dakie, chiemo ma ok owinjore, koda bedo maonge gi paro mowinjore, nyalo miyo ng'ato obed gi tuwo mar Alzheimer.
7. Tuwo: Tuwo ma ok rum e obwongo nyalo miyo ng'ato obed gi tuwo mar Alzheimer.
8. Oxidative stress: Bedo ni ng'ato ok nyal geng'o gik ma miyo del bedo gi teko mar golo gik ma miyo del bedo gi teko mar geng'o gik ma miyo del bedo gi teko mar geng'o gik ma miyo del bedo gi teko mar geng'o gik ma miyo del bedo gi teko.
9. Amyloid plaques kod neurofibrillary tangles: Bedo gi amyloid plaques kod neurofibrillary tangles e obwongo en gima nyiso ni ng'ato nigi tuwo mar Alzheimer, kata kamano, ok ong'ere malong'o migawo ma gintiere e kelo tuwono.
Dwarore ni wang'e ni tuwo mar Alzheimer kelo gik mang'eny mopogore opogore, kendo nyalo bedo ni nitie gik mang'eny motudore gi tuwono.
Nonro pod dhi nyime mondo ng'ato ong'e kaka tuwono timore kendo mondo oyud yore mag thiedho tuwono.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
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(5) weche ma in iwuon iwacho, ma iketo e bwo buch kuong'ruok, ni weche manie kalatasno gin adier, kendo ni in gi teko mar tiyo gi ratiro mag ndiko ma ji wacho ni oketh;
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Ka ok iketo weche duto monyis malo kae, mano nyalo miyo idonj e kesno bang'e.
Wuo gi ng'at machielo
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What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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Chiegni ni
BioMedLib tiyo gi kompyuta ma tiyo gi masinde (ma iluongo ni machine-learning algorithms) e loso penjo gi dwoko.
Ne wachako gi buge milion 35 mag weche thieth miluongo ni PubMed/Medline. Bende, ne wachako gi buge mag RefinedWeb.
