What causes Alzheimer?

Isimgħu din il-paġna

X'jikkawża l- alzheimer?

Il- marda taʼ Alzheimer hija disturbu newrodegenerattiv progressiv li primarjament jaffettwa l- moħħ, u jikkawża telf taʼ memorja, tnaqqis konjittiv, u bidliet fil- mġiba.

Il- kawża eżatta tal- marda taʼ Alzheimer mhix mifhuma għalkollox, imma huwa maħsub li tirriżulta minn kombinazzjoni taʼ fatturi ġenetiċi, ambjentali, u taʼ l- istil taʼ ħajja.

Xi wħud mill- fatturi li jistgħu jikkontribwixxu għall- żvilupp tal- marda taʼ Alzheimer jinkludu:

1. Ġenetika: Ġew identifikati ċerti ġeni li jżidu r- riskju li jiżviluppaw il- marda taʼ Alzheimer, b'mod partikolari l- ġene tal- apolipoproteina E (APOE).

2. L- età: Ir- riskju li tiżviluppa l- marda taʼ Alzheimer jiżdied mal- età, u l- biċċa l- kbira tan- nies jiġu dijanjostikati wara l- età taʼ 65 sena.

3. Storja tal- familja: Li jkollok storja tal- familja tal- marda taʼ Alzheimer jistaʼ jżid ir- riskju li tiżviluppa din il- kundizzjoni.

4. Korrimenti fuq ir- ras: Storja taʼ korrimenti fuq ir- ras, speċjalment dawk li jirriżultaw f'telf tal- kuxjenza, jistgħu jżidu r- riskju tal- marda taʼ Alzheimer.

5. Fatturi taʼ riskju kardjovaskulari: Kondizzjonijiet bħal pressjoni għolja tad- demm, kolesterol għoli, u dijabete jistgħu jżidu r- riskju li tiżviluppa l- marda taʼ Alzheimer.

6. Fatturi dwar l- istil taʼ ħajja: Stil taʼ ħajja sedentarju, dieta ħażina, u nuqqas taʼ stimulazzjoni mentali jistgħu jżidu r- riskju tal- marda taʼ Alzheimer.

7. Inflammazzjoni: Inflammazzjoni kronika fil- moħħ tistaʼ tikkontribwixxi għall- żvilupp tal- marda taʼ Alzheimer.

8. Stress ossidattiv: Żbilanċ bejn il- produzzjoni taʼ radikali ħielsa u l- ħila tal- ġisem li jiddetossikahom jistaʼ jikkontribwixxi għall- żvilupp tal- marda taʼ Alzheimer.

9. Plaċċi amilojdi u tangles neurofibrillari: L- akkumulazzjoni ta ' plaċċi amilojdi u tangles neurofibrillari fil- moħħ hija karatteristika tal- marda ta ' Alzheimer, iżda r- rwol li għandhom fl- iżvilupp tal- kundizzjoni mhuwiex mifhum bis- sħiħ.

Huwa importanti li wieħed jinnota li l- kawża tal- marda taʼ Alzheimer hija kumplessa u x'aktarx li tinvolvi kombinazzjoni taʼ dawn il- fatturi.

Qed isir riċerka biex jinftiehmu aħjar il- mekkaniżmi sottostanti u jiġu żviluppati trattamenti effettivi għal din il- kundizzjoni devastanti.

Referenzi

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.

Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.

Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.

Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.

Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.

Tneħħija ta ' responsabbiltà: mediku

Dan is-sit tal-web huwa pprovdut għal skopijiet edukattivi u ta' informazzjoni biss u ma jikkostitwixxix l-għoti ta' parir mediku jew servizzi professjonali.

L- informazzjoni pprovduta m'għandhiex tintuża għad- dijanjosi jew it- trattament taʼ problema jew marda tas- saħħa, u dawk li jfittxu parir mediku personali għandhom jikkonsultaw maʼ tabib liċenzjat.

Jekk jogħġbok innota li n-netwerk newrali li jiġġenera t-tweġibiet għall-mistoqsijiet, huwa speċjalment mhux preċiż meta niġu għal kontenut numeriku. Pereżempju, in-numru ta' nies dijanjostikati b'marda speċifika.

Dejjem fittex il-parir tat-tabib tiegħek jew ta' fornitur tas-saħħa kwalifikat ieħor dwar kundizzjoni medika. Qatt ma tinjora l-parir mediku professjonali jew id-dewmien fit-tfittxija tiegħu minħabba xi ħaġa li qrajt f'dan il-websajt. Jekk taħseb li tista' jkollok emerġenza medika, ċempel 911 jew mur l-eqreb kamra tal-emerġenza immedjatament. L-ebda relazzjoni bejn tabib u pazjent ma tinħoloq minn dan il-websajt jew l-użu tagħha. La BioMedLib u lanqas l-impjegati tagħha, u lanqas kwalunkwe kontributur għal dan il-websajt, ma jagħmel xi rappreżentazzjonijiet, espressi jew impliċiti, fir-rigward tal-informazzjoni pprovduta hawnhekk jew għall-użu tagħha.

Riżerva ta' responsabbiltà: drittijiet tal-awtur

L-Att dwar id-Drittijiet tal-Awtur tal-Millennju Diġitali tal-1998, 17 U.S.C. § 512 (il-"DMCA") jipprovdi rikors għas-sidien tad-drittijiet tal-awtur li jemmnu li materjal li jidher fuq l-Internet jikser id-drittijiet tagħhom skont il-liġi tad-drittijiet tal-awtur tal-Istati Uniti.

Jekk temmen b'bona fidi li kwalunkwe kontenut jew materjal magħmul disponibbli b'rabta mal-websajt jew is-servizzi tagħna jikser id-drittijiet tal-awtur tiegħek, inti (jew l-aġent tiegħek) tista' tibgħatilna avviż li jitlob li l-kontenut jew il-materjal jitneħħa, jew li l-aċċess għalih jiġi mblukkat.

L-avviżi għandhom jintbagħtu bil-miktub bil-posta elettronika (ara t-taqsima "Kuntatt" għall-indirizz tal-posta elettronika).

Id-DMCA teħtieġ li n-notifika tiegħek ta' ksur allegat tad-drittijiet tal-awtur tinkludi l-informazzjoni li ġejja: (1) deskrizzjoni tax-xogħol protett bid-drittijiet tal-awtur li huwa s-suġġett ta' ksur allegat; (2) deskrizzjoni tal-kontenut allegatament li jikser id-drittijiet tal-awtur u informazzjoni suffiċjenti biex inkunu nistgħu nsibu l-kontenut; (3) informazzjoni ta' kuntatt għalik, inkluż l-indirizz tiegħek, in-numru tat-telefon u l-indirizz tal-email; (4) dikjarazzjoni minnek li għandek twemmin ta' fidi tajba li l-kontenut bil-mod li dwaru l-ilment ġie ppreżentat mhuwiex awtorizzat mid-detentur tad-drittijiet tal-awtur, jew mill-aġent tiegħu, jew mill-operazzjoni ta' kwalunkwe liġi;

(5) stqarrija minnek, iffirmata taħt il-piena ta' perjuri, li l-informazzjoni fin-notifika hija preċiża u li għandek l-awtorità li tinforza d-drittijiet tal-awtur li huma ddikjarati li ġew miksura;

u (6) firma fiżika jew elettronika tas-sid tad-drittijiet tal-awtur jew ta' persuna awtorizzata li taġixxi f'isem is-sid tad-drittijiet tal-awtur.

In-nuqqas li tinkludi l-informazzjoni kollha ta' hawn fuq jista' jwassal għal dewmien fit-trattament tal-ilment tiegħek.

Kuntatt

Jekk jogħġbok ibgħatilna email b'kull mistoqsija / suġġeriment.

What causes alzheimer?

Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.

The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.

Some of the factors that may contribute to the development of Alzheimer's disease include:

1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.

2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.

3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.

4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.

5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.

6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.

7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.

8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.

9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.

It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.

Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.

Disclaimer: medical

This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.

The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.

Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.

Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.

Disclaimer: copyright

The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.

Madwar

BioMedLib juża kompjuters awtomatizzati (algoritmi ta' tagħlim tal-magni) biex jiġġenera koppji ta' mistoqsijiet u tweġibiet.

Aħna nibdew b'35 miljun pubblikazzjoni bijomedika ta' PubMed/Medline. Ukoll, paġni tal-web ta' RefinedWeb.

Ara "Referenzi" ukoll "Disclaimer".