6. Kawsayninchik: Mana allinta kawsaspa, mana mikhuspa, mana yuyayninchikta llamkʼachispa ima, Alzheimer unquywan unqunanchikpaq, astawan tanqawanchik.
7. Inflamación: Alzheimerra unquyqa, uma ukhupi unayta unqusqanrayku rikhurin.
8. Alzheimerra unquyqa, mana allinchu kanman, chayraykutaq, mana allinchu kanman, chay unquymanta llimphuchakuyta atinapaq, mana allinchu kanman, chayraykutaq, mana allinchu kanman, chay unquywan unqunanchik.
Kay unquyqa, juk laya unquy, mayqinchus, juk runap yuyayninman chayan, chayrayku, mana allinchu.
Kay unquyqa, mana jukllachu, ichá kay unquykunawan khuska rikhurin.
Kay unquymanta astawan yachanapaq, imachus chay unquy kasqanta yachanapaq ima, astawan ukhunchakun.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Mana allinchu: unquy']
['Kay páginaqa, mana doctorman willanallapaqchu, nitaj ima unquymantapis jampichikunallapaqchu.']
['Kaypi willakusqanqa, mana unquykunata, unquykunata ima, yachanapaqchu, manaqa, unqusqasta yanapanapaq.']
['Kay tapuykunaman kutichiykunata churanapaq red neuronalqa, mana chiqanchu kachkan, chantapis yupaykunawan churanakun.']
["Kay sitio web nisqaqa mana ima unquymantapis jampichikunchu. BioMedLib, llamk'aqninkuna, manaqa kay sitio web nisqaman llamk'aqkuna, mana ima willaytapis, sut'ita chayri mana sut'ita, kaypi kaq willaykunata, apaykachayta ima, willankuchu."]
['Amachasqa: copyright']
['Kay Ley de Derechos de Autor del Milenio Digital de 1998, 17 U.S.C. § 512 (la DMCA) kamachiyqa, derechos de autorniyuqkunata, Internetpi kaqkuna, derechos de autor nisqata pʼakikusqankuta yuyaqkunata, jarkʼan.']
["Sichus allin yuyaywan yuyanki kay webpi utaq kay llamk'aykunapi ima qillqapas utaq ima llamk'achisqapas derechosniykita p'akikusqanta chayqa, qan (utaq wakichiqniykim) willawasunman chay qillqata utaq llamk'achisqata chinkachinanchikpaq utaq hark'ananchikpaq."]
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
Disclaimer: medical
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Disclaimer: copyright
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