Pathophysiology n heart attack, s isim net infarction n myocardial, eha anmizlay n azruf n idammen s achrut n muscle n ul, eway id tamattant n izirwan n ul
Awen eqqal fal as eɣaf n artery tan n corona s iddikud n idammen, eqqal harkuk a fal atherosclerosis, aqqal talɣa s iddikud n plaque daɣ artery tan
Alwaq wa id taqqimt n aratan tazzarat, addobat ad t awikkal as aratan n idammen ad aqqilan, addoben ad eɣzan artery tolas ad eɣzan idammen n oxygen i ad awḍen s aɣasan n ul
Awen war n ila oxygen eway id tisilwan n afissar n ul ad mmutent, eway id heart attack
Akayad n aśahat eqqal fal edag wa eha artery wa eɣlalan əd azzaman jir aśahat əd asafar
Isuj n aśahat n ul addoben ad ahan aśuhu meɣ aśuhu daɣ taɣissa, aśuhu n afanaz, aśuhu, aśuhu n aśuhu, əd aśuhu daɣ əfus, əɣaf, əšəɣəl meɣ aśuhu.
Asafar n aśahat n ul eha agudiyan asiwid n asiflid n idammen daɣ aśahat n ul s data s addobat, s asafar meɣ tikarsiwen chund angioplasty d stenting meɣ aśahat n aśahat n aśahat
Eqal assohen ad akayad as patophysiology n heart attack eqqal a ojjaran tolas ahanat haratan ajjotnen eha genetic, lifestyle d haratan n ahinzazaɣ
Haratan n aharoj n aśahat n ul ahan tanhat n idammen, cholesterol tanhat, asigar, aśahat n aratan, aśuhu, iban alxidmat n taɣissa d attarex n egadal n aśahat n ul
Aɣil n haratan win n ahinzazaɣ addobat ad eɣna asuj n ihuk n ul
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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['(5) almaɣna n awen, iktab daɣ azruf n alkad n tiditt, as isalan win isalan aqqalan tiditt tolas ilan tanaya n as artay alxaqqan win alxakum wi s allaɣen as ikna']
['Dǎɣ awa, əmik ən əmik ən alxaq meɣ awadim ən alxaq ən alxaq meɣ awadim atiwafan ye igi ən alxaq ən alxaq.']
['A fal war t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id']
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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