Oku kuliha eci ci koka uveyi wo cancer yo colon ci lomboloka oku kuliha ndomo uveyi waco u fetika kuenda oku amamako.
O cancer yo colon, yi tukuiwavo hati colorectal cancer, yi lomboloka uveyi wo cancer u fetikila vo colon ale vo rectum okuti onepa yimue yo intestino grosso.
Uvei waco u fetika poku kula kuetimba kuenje vokuenda kuotembo u pongoloka uvei wo kanser.
Oku kala kuolombuto vi kuete uveyi wo cancer yo colon ku tiamisiwila kovina vialua, oku kongelamo oku pongoloka kuovimatamata viuyali, oku kuatiwa luvei kuenda ovina vikuavo.
Oku pongoloka kuovimatamata viuyali ku pondola oku pita vo DNA yovimatamata viosingo, okuti ku tuala koku kula kuenda koku litepa kuovimatamata.
Ovio vi pondola oku nyõla olonepa vimue viekova vi kuatisa koku kula kuoselula, oku li tepa, kuenda oku tumbulula ekalo liayo.
Oku kuatiwa luvei wo colon, okuti u pondola oku kokiwa lovoveyi amue ndeci uveyi wo intestino, citava okuti u vokiyavo ohele yoku kuatiwa luvei wo colon cancer.
Nda omunu o kuete uveyi umue ka u kuete ovihemba, etimba liaye li fetika oku kuata uvei wo kanser.
Ovina vimue ndeci, okulia, ekalo liomuenyo kuenda oku kapiwa kuolombombo vimue, vi pondolavo oku koka uveyi wo kanser.
Oku lia calua ositu yikusuka kuenda yina ya pongiyiwa ciwa, ca siata oku nena uveyi wo cancer.
Kulivo ovina vikuavo vi vokiya ohele yoku kuatiwa luvei waco ndeci: Oku lẽla calua, oku sipa, kuenda oku linga olomapalo ka via sungulukile.
Eci uveyi wo cancer yo colon u fetika, citava okuti u pita vapuluvi alua, okupisa vapuluvi atete okuti uveyi waco u kasi lika vo colon toke vapuluvi ana okuti uveyi waco wa lisanduila kolonepa vikuavo vietimba.
O kanser yi pondola oku iñila vovimatamata vi kasi ocipepi kuenda yi lisanduilavo volosinga kuenda vosonde toke volonepa vikuavo ndeci, vowoño ale vapuvi.
Oku sakuiwa kuolombuto viocilenda ci lomboloka oku linga o cirurgia, o quimioterapia kuenda o radioterapia, ca tiamẽla kocitumãlo kuenda kocitumãlo co cancer.
Oku limbuka lonjanga kuenda oku sakula, oco ocina ca velapo koku mioñolola ovitangi, momo uveyi wo cancer yo colon olonjanja vialua u sakuiwa eci u limbukiwa votembo yaye yatete.
Oku linga akonomuiso amue ndeci, oku kũlĩhĩsa ombuto yuveyi waco, ci kuatisa oku limbuka uveyi waco osimbu ka wa fetikile.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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