Pathophysiology yo diabetes yi lomboloka oku lilongisa eci catiamẽla kovituwa viuhayele vi tuala koku kala luvei wo diabetes mellitus.
O diabetes mellitus ocimunga cimue coviveyi vi tukuiwa hati, metabolic diseases vi limbukiwila koku livokiya kuosukari vosonde (glucose) okuti yi tunda koku hongua kuoku kapiwa kuosukari, ale kovilinga viosukari.
Uvei wo diabete u tiamisiwila kovituwa vimue ndeci: Oku citiwa, ekalo lio vonjo kuenda ekalo liomuenyo womunu.
Ko diabetes yocituwa 1, pathophysiology yikongela oku nyõleha kuovimatamata viosonde vi panga o insulina vimo liokapa, okuti cilingisa ekambo lioku panga o insulin.
Eci ci koka okuti ka ci tẽliwa oku lava ciwa osonde, kuenje eci ci tuala koku kuata uvei wo hiperglicemia (oku livokiya kuosukari vosonde).
Oku tamalãla kuove ku insulin ku pita eci olosinga vietimba ka vi tambuluiya ciwa ku insulin, okuti ka vi tẽla oku talavaya ciwa lo glucose oco yi linge ongusu.
Eci ci vetiya ombuto yoku cita oco yi pange vali o insulina oco yi yule ocitangi caco, pole vokuenda kuotembo, ombuto yoku cita ka yi tẽla vali oku linga upange waco, kuenje yi tepulula o insulin.
Uvei wo diabete u tukuiwa hati, diabetes gestacional, u tunda kapongoloko o vo hormônio okuti, a nena esino lioku tambula o insulina.
O placenta yi panga olonepele vi pondola oku tateka upange wo insulina, kuenje yi vokiya osonde.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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