What is pathophysiology of Urticaria?

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什麼是麻疹的病理生理學?

麻疹是一種皮膚疾病, 皮膚上出現,凸起,紅色斑點.

麻疹的病理生理學涉及到肥大細胞釋放組胺和其他炎症介質,

針對特定過敏原 (如食物,藥物或昆蟲咬) 產生過敏反應,

造成血管內的血管外皮形成腫瘤.

慢性麻疹的病因通常不明,

在這種情況下,身體會產生自身抗體, 針對肥大細胞上的高親和力IgE受體 (FcεRI),

催產素的釋放會導致,發紅及腫等特徵性症狀.

抗體胺藥物通常能抑制組織胺的作用並減少症狀.

其他藥物如皮質類固醇或奧馬利祖瑪可能會被使用來控制病情.

['其他資料']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.

Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.

[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.

Marrouche N, Grattan C: Childhood urticaria. Curr Opin Allergy Clin Immunol. 2012, 12 (5): 485-90.

Brzoza Z, Grzeszczak W, Rogala B, Trautsolt W, Moczulski D: Possible contribution of chemokine receptor CCR2 and CCR5 polymorphisms in the pathogenesis of chronic spontaneous autoreactive urticaria. Allergol Immunopathol (Madr). , 42 (4): 302-6.

Sweeney TM, Dexter WW: Cholinergic urticaria in a jogger: ruling out exercise-induced anaphylaxis. Phys Sportsmed. 2003, 31 (6): 32-6.

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What is pathophysiology of urticaria?

Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.

The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.

In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.

This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.

In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.

In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.

In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.

Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.

In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.

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