Alzheimer yare no yɛ yare a ɛsan fa nkwammoaa mu na ɛsan fa amemene no mu, na ɛma obi werɛ fi nneɛma, n'adwene yɛ adwuma yiye, na ɔyɛ nneɛma bi a ɛmfata.
Wɔnte nea ɛde Alzheimer yare no ba no ase yiye, nanso wogye di sɛ efi awosu, nneɛma a atwa yɛn ho ahyia, ne asetra kwan mu.
Nneɛma bi a ebetumi ama obi anya Alzheimer yare no bi ne:
1. Awo mu nneɛma: Wɔahu awosu bi a ɛma obi nya Alzheimer yare no bi, titiriw awosu a ɛma obi nya apolipoprotein E (APOE) no.
2. Mfe: Asiane a ɛwɔ Alzheimer yare no mu yɛ kɛse bere a obi nyin no, na nnipa dodow no ara nya yare no bi bere a wɔadi mfe 65 akyi.
3. Abusua mu abakɔsɛm: Sɛ obi wɔ Alzheimer yare no bi wɔ n'abusua mu a, ebetumi ama yare no anya no kɛse.
4. Ti a wopira: Ti a wopira pɛn, titiriw nea ɛma obi yɛ hahaaha no betumi ama woanya Alzheimer yare no bi.
5. Komayare ho nneɛma: Nyarewa te sɛ mogya mmoroso, mogya mu srade a ɛwɔ mogya mu, ne asikreyare betumi ama obi anya Alzheimer yare no bi.
6. Asetra kwan: Sɛ obi nte faako, onni aduan pa, na onni nneɛma a ɛbɛkanyan n'adwene a, ebetumi ama wanya Alzheimer yare no bi.
7. Ntini a ɛpae: Ntini a ɛpae wɔ amemene mu betumi ama obi anya Alzheimer yare.
8. Adwennwen a ɛma nipadua no wosow ahoɔden: Sɛ ahoɔdennuru a ɛma nipadua no yɛ adwuma yiye no so tew na nipadua no ntumi nyi nnuru bɔne mfi mu a, ebetumi ama obi anya Alzheimer yare.
9. Amyloid plaques ne neurofibrillary tangles: Amyloid plaques ne neurofibrillary tangles a ɛboaboa ano wɔ amemene no mu no yɛ Alzheimer yare no ho sɛnkyerɛnne, nanso wɔnte dwuma a wodi wɔ yare no mu ase nwie.
Ɛho hia sɛ wohyɛ no nsow sɛ Alzheimer yare no de ɔhaw pii na ɛba na ɛhwehwɛ sɛ saa nneɛma yi nyinaa ka bom.
Wɔreyɛ nhwehwɛmu akɔ akyiri na ama yɛate yare no ase yiye na yɛanya ano aduru a ɛbɛboa no.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Abɔdin: ayaresa']
['Wɔayɛ wɛbsaet yi sɛ wɔmfa nkyerɛkyerɛ na wɔnka ho asɛm kɛkɛ, na ɛnyɛ sɛ wɔde ayaresa ho afotu anaa adwumakuw bi mmoa rema.']
['Ɛnsɛ sɛ wɔde nsɛm a wɔde ama no di dwuma de hwehwɛ yare anaa yare bi ho ayaresa, na ɛsɛ sɛ wɔn a wɔrehwehwɛ ayaresa ho afotu no ne oduruyɛfo a ɔwɔ tumi krataa di nkitaho.']
['Yɛsrɛ wo hyɛ no nsow sɛ amemene no mu mfiri a ɛma nsɛmmisa ho mmuae no nyɛ pɛpɛɛpɛ bere a ɛfa akontaahyɛde ho no. Sɛ nhwɛso no, nnipa dodow a wɔanya yare pɔtee bi.']
["Hwehwɛ afotu fi wo dɔkota anaa ayaresafo a wɔfata hɔ bere biara wɔ yareɛ ho. Nnyae ayaresa ho afotuo a wɔn a wɔn ho akokwaw de ma no ho adwenemu anaa twentwɛn wo nan ase sɛ worebɛhwehwɛ esiane biribi a woakenkan wɔ wɛbsaet yi so nti. Sɛ ɛyɛ wo sɛ wowɔ yareɛ ho nsɛmmisa a, frɛ 911 anaa kɔ ayaresabea a ɛbɛn wo pɛɛ ntɛm ara. Saa wɛbsaet yi anaa dwuma a wode di no mma wonnya oduruyɛfo ne ɔyarefo ayɔnkofa biara. BioMedLib anaa n'adwumayɛfoɔ anaa obiara a ɔde ne ho bɔ wɛbsaet yi ho dawuro biara nni mu, a ɛkyerɛ anaa enni mu, fa nsɛm a wɔde ama wɔ ha anaa dwuma a wɔde di no ho."]
['Disclaimer: copyright']
["Digital Millennium Copyright Act a w'atwe no afe 1998 wɔ U.S. Mmara 17 § 512 (DMCA) no ma wɔn a w'wɔ tumi sɛ wɔtwe wɔn ho fi nneɛma a wɔde agu intanɛt so ho kwan."]
['Sɛ wogye di sɛ wo nsɛm anaa nneɛma bi a ɛwɔ yɛn wɛbsaet anaa yɛn dwumadie mu no to wo mmara a woahyehyɛ no so a, wo (anaa wo dwumadifoɔ) bɛtumi de nkaebɔ ama yɛn de apɛ sɛ yɛyi nsɛm anaa nneɛma no firi hɔ anaa yɛsi ho kwan.']
["Ɛsɛ sɛ wɔde nkaebɔ fa e-mail so kɔma wɔn (hwɛ 'Contact' section ma e-mail address)."]
['DMCA hwehwɛ sɛ wo dawurubɔ a ɛfa mmara a obi abu so ho no de nsɛm a edidi so yi ka ho: (1) nkyerɛwde a ɛkyerɛ adwuma a mmara bɔ ho ban a obi abu so no; (2) nsɛm a wɔkyerɛ sɛ obi abu so no ne ɛho nsɛm a ɛfata a ɛbɛma yɛahunu faako a saa nsɛm no wɔ; (3) wo nkitahodi ho nsɛm, a wo address, fon number ne email ka ho; (4) krataa a woakyerɛ sɛ wowɔ gyidie pa sɛ nea woabɔ ho dawuru no nni mmara no wura anaa nea ɔhwɛ so anaa mmara biara tumi mu.']
['(5) sɛ wo de wo nsa ahyɛ krataa ase, na wohyɛ sɛ wobedi atoro, sɛ nsɛm a ɛwɔ krataa no mu yɛ nokware, na wowɔ tumi sɛ wode wo nsa bɛka nneɛma a obi akyerɛw abrɛ wo ase no;']
["و (6) physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner's behalf."]
['Sɛ woantwerɛ nsɛm a ɛwɔ soro yi nyinaa amfiri wo nkrataa no mu a, ɛbɛtumi ama wo kyɛfa no akyɛ.']
['Nkitahodi']
['Yɛsrɛ wo, fa nsɛmmisa anaa nyansahyɛ biara fa e-mail so brɛ yɛn.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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Disclaimer: copyright
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['Ɛfa ho']
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