Alzheimer bana ye bana ye min bɛ don mɔgɔ hakili la ni o bɛ mɔgɔ hakili ɲagami, k'a hakili tiɲɛ, k'a kɛ i n'a fɔ i tɛ foyi kalama.
A ka ca a la, o bana in sababu bɛ bɔ mɔgɔ wolo yɔrɔ, a sigida ani a ka ɲɛnamaya kɛcogo de la.
Fɛn minnu bɛ se ka kɛ sababu ye ka mɔgɔ bila sidabana la, olu dɔw filɛ:
1. joli siraw: Mɔgɔw ye joli sira dɔw dɔn minnu bɛ sumaya bila mɔgɔ la.
San 2nan: Ni mɔgɔ bɛka kɔrɔ ka taa, a ka teli ka kɛ ni a ka bana ye. A ka ca a la, o bɛ sɔrɔ a tigi san 65 ni kɔ de la.
3. Denbaya min ka mɔgɔ dɔ ye sidabana sɔrɔ: Ni sidabana bɛ mɔgɔ dɔ ka denbaya kɔnɔ, o bɛ se ka dɔ fara a ka bana sɔrɔli farati kan.
Kunkolo joginni: Kunkolo joginni minnu bɛ mɔgɔ hakili ɲagami, o bɛ se ka sumaya bila a tigi la.
5. Dusukun tantanni faratiw: Joli tansiyɔn yɛlɛlen, sukaro ani jolisegindumuni sɔrɔli faratiw bɛ se ka kɛ sababu ye ka mɔgɔ minɛ.
6. Balocogo: Mɔgɔ minnu ka ɲɛnamaya kɛcogo man ɲi, dumuni nafama dunbaliya ani hakili ɲagami, olu ka teli ka sumaya.
7. Faritɛnɛbana: Faritɛnɛbana min bɛ don mɔgɔ hakili la, o bɛ se ka laban ni sumaya ye.
8. Farikolo fununi: Farikolo fununi n'a tiɲɛni ka teli ka kɛ sababu ye ka mɔgɔ minɛ.
9. Amiyɔr pɔlikiw ani sɛmɛw: Amiyɔr pɔlikiw ni sɛmɛw minnu bɛ ɲɔgɔn lajɛn mɔgɔ hakili la, olu ye jolidɛsɛ taamasiɲɛ ye. Nka, u jɔyɔrɔ bɛ min na o dɔn man di.
A nafa ka bon ka a dɔn ko bana min bɛ wele ko alzheimers, o sababu ka ca wa a bɛ se ka kɛ nin fɛn ninnu caman de ye ɲɔgɔn sɔrɔ.
Sɛgɛsɛgɛli bɛ sen na walasa ka faamuyali ɲuman sɔrɔ bana in sɔrɔsababuw kan ani ka fura ɲumanw sɔrɔ a furakɛli la.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Kunnafoni nafama: kɛnɛya']
['Nin gafe in labɛnna kalan ni kunnafoni dɔrɔn de kama, a tɛ sɔrɔ dɔgɔtɔrɔ ka ladili wala kɛnɛyabaarakɛla ka baara kɛcogo la.']
["Kunnafoni minnu dira aw ma, aw man kan ka olu kɛ ka banaw furakɛ. Mɔgɔ minnu b'u yɛrɛ furakɛ, olu ka kan ka dɔgɔtɔrɔ dɔ ka dɛmɛ ɲini."]
["Aw ye aw janto nin na: ɛntɛrinɛti min bɛ jabi di ɲininkaliw jaabiliw kan, o dabali ka jugu kɛrɛnkɛrɛnnenya la n'a bɛ tali kɛ jatidenw de kan. Misali la, mɔgɔ hakɛ min ka bana kofɔra."]
['Aw bɛ ladilikan ɲini tuma bɛɛ aw ka dɔgɔtɔrɔ fɛ walima kɛnɛya baarakɛla dɔ wɛrɛ min bɛ se ka aw dɛmɛ ka aw ka bana furakɛ. Aw kana dɔgɔtɔrɔ ka ladilikan bila ka suma walima ka mɛn a ɲini na sabu aw ye fɛn dɔ kalan nin siti kan. Ni aw hakili la ko aw ka bana bɛ se ka juguya joona, aw ye 911 wele walima aw ka taa aw ka dɔgɔtɔrɔso la joona joona. Nin siti tɛ dɔkɔtɔrɔ ni banabagatɔ ka jɛɲɔgɔnya jira. BioMedLib ni a ka baaradenw, walima nin siti dɛmɛbaga si tɛ kuma si jira walima ka jira, nin kunnafoniw wala u labaarali kama.']
['Kunnafoni min lakodɔnna:']
["Digital Millennium Copyright Act san 1998, 17 U.S.C. § 512 (a DMCA) bɛ sariya sigi ka ɲɛsin mɔgɔ ma min b'a miiri ko fɛn min bɛ sɔrɔ intɛrinɛti kan, o bɛ tɔɲɔ a ka sariyaw la Ameriki jamana ka sariya kɔnɔ. "]
['Ni i dara a la kô i ka site web ni a baara tchogow bè i ka lakananifènw la, i (walima i ka lasigiden) bè se ka i yèrè ka lakananifènw bila ka o site web ni a baara tchogow bila kènèkan.']
['Waajibi don kunnafoniw ci kɛtɔ ka kɛ sɛbɛn ye e-mail fɛ (e-mail de kan ka lajɛ sɛbɛn ɲɛ Kunnafoniw lajɛ yɔrɔ la).']
["DMCA bɛ a ɲini i ka kunnafoni sɛbɛn bɔlɔlɔw kan kojugukɛ sɛbɛn bɛ sɛbɛn min kɔnɔ, o ka kan ka nin kunnafoniw fara a kan: (1) sɛbɛnni kɛtɔ ka sɛbɛnni kɛ min bɛ sɛbɛnni kɛ ni a ma kɛ kojugukɛ sɛbɛn ye; (2) sɛbɛnni kɛtɔ ka fɛn kofɔlen in kofɔ ani kunnafoni minnu bɛ a to an bɛ se ka fɛn kofɔlen in sɔrɔ; (3) i ka ladɛrɛsi, i ka ladɛrɛsi, telefɔni nimɔrɔ ani i ka ladɛrɛsi; (4) i ka kumaɲɔgɔnya sɛbɛn ko i dalen b'a la ko i bɛ a dɔn ko i bɛ ka fɛn kofɔlen in kɛ ni sariya min ye, o ma di sɛbɛnnikɛbaga ma, a ka ladɛrɛsi tigi, walima sariya wɛrɛ; "]
['(5) a ka seereyasɛbɛ sɛbɛntiya, ko seereyasɛbɛ minɛnen bɛ kojugubakɛlaw kama, ko kunnafoni min bɛ o sɛbɛntiya kɔnɔ, ko tiɲɛ don ani ko i yamaruyalen don ka yamaruya di ka kɛwalew kɛ minnu bɛ tiɲɛni kɛ; ']
['ani (6) sɛbɛn dɔ ka boloci walima ɛkitɔrɔniki sɛbɛn sɛbɛn tigi fɛ walima mɔgɔ min yamaruyara ka baara kɛ sɛbɛn tigi tɔgɔla. ']
['Ni kunnafoni fɔlen ninnu bɛɛ ma fara ɲɔgɔn kan, o bɛ se ka kɛ sababu ye ka mɛn baara kɛli la i ka sɛbɛn kɔnɔ.']
['Ɲɔgɔnkunbɛn']
['Sɛbɛn ɲɛ Ɲiningaliw / ladiliw']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
Disclaimer: medical
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Disclaimer: copyright
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['Ɲɔgɔn fɛ']
['BioMedLib bɛ baara kɛ ni ɔridinatɛri ye (mansin ka kalan aligoritimiw) walasa ka ɲininkaliw ni jaabiw di ɲɔgɔn ma.']
['An bɛ a daminɛ ni gafew miliyɔn 35 ye ka bɔ PubMed/Medline. Ani fana, ɛntɛrinɛti sitiw ka bɔ RefinedWeb.']
