Kɔnɔbara basigilan kansɛri bana kun ye fɛɛrɛw ye minnu bɛ kɛ sababu ye ka kansɛri lase mɔgɔ ma.
Kolotuguda kansɛri, min bɛ wele fana ko kansɛri min bɛ daminɛ kunsɛmɛ na walima kansɛri min bɛ daminɛ tonso bɔlen na, o ye biɲɛdimi suguya dɔ ye.
A ka ca a la a bɛ daminɛ i n'a fɔ sumuni min bɛ wele ko pɔlip, min bɛ se ka yɛlɛma ka kɛ kansɛri ye waati dɔ la.
Kɔnɔboli basigilen sababu ye fɛn caman ye, bana minnu sababu ye yɛlɛma ye joli fɛ, sumaya, ani lamini sababuw.
Genetiki yelemali be se ka sɔrɔ colon seliliw ka DNA kono, min be na ni seliliw ka cayali ani u tilani ma.
O yɛlɛmaw bɛ se ka sɔrɔ cɛya fɛ wala musoya fɛ, wa u bɛ se ka fiɲɛ don zɛni suguya caman na minnu bɛ se ka kɛ sababu ye ka seliliw falenfalen, ka tila ani ka u yɛrɛ labɛn.
Kɔnɔboli min bɛ sɔrɔ bana dɔw sababu la i n'a fɔ kɔnɔboli min bɛ sɔrɔ banakisɛ donni na, o fana bɛ se ka kansɛribana farati bonya.
Faritɛnɛbana juguman bɛ se ka na ni baga dɔw ye farikolo la minnu bɛ se ka kɛ sababu ye ka seliliw ka bonya ani ka u tila, ka laban kansɛri ma.
Farikolo lamini sababuw, i n'a fɔ dumuni, balocogo, ani bagaji dɔw tali, olu fana bɛ se ka kɛ sababu ye ka kansɛri lase mɔgɔ ma.
Misali la, dumuni min bɛ sogo mɔbali dun walima sogo kɛnɛba, o bɛ se ka kansɛri lase mɔgɔ ma.
Ko wɛrɛw bɛ se ka a to a tigi ka bana, i n'a fɔ ka bon kojugu, ka sigarɛti min ani ka farikoloɲɛnajɛ kɛ kojugu.
Ni kansɛri bɔra kɔkolo la, a bɛ se ka yɛlɛma ka kɛ bana caman ye, ka daminɛ a daminɛ na min bɛ sɔrɔ kɔkolo dɔrɔn de la ka taa a bila a ɲɛfɛla la ni a jɛnsɛnna ka se farikolo fan tɔw ma.
Kansɛri bɛ se ka seliliw kɛrɛfɛlanw ni farikolo yɔrɔw minɛ, ani fana a bɛ se ka jɛnsɛn farikolo yɔrɔw ni joli siraw fɛ yɔrɔjanw na, i n'a fɔ biɲɛ walima biɲɛ.
Kɔnɔboli kansɛri furakɛli bɛ tɛmɛ ni kirinni ye, kansɛri furakɛli ni kirinni juguman furaw ye ka a da a suguya kan ani a yɔrɔ.
A kɔrɔ ye ko bana minnu bɛ sɔrɔ banakisɛw tali fɛ, olu ka teli ka juguya.
Kɔnɔnatumuw sɛgɛsɛgɛli ka caya, i n'a fɔ kolosinsindimi, o bɛ se ka kansɛri daminɛni joona joona, a furakɛli ka nɔgɔ tuma min na.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Kunnafoni nafama: kɛnɛya']
['Nin gafe in labɛnna kalan ni kunnafoni dɔrɔn de kama, a tɛ sɔrɔ dɔgɔtɔrɔ ka ladili wala kɛnɛyabaarakɛla ka baara kɛcogo la.']
["Kunnafoni minnu dira aw ma, aw man kan ka olu kɛ ka banaw furakɛ. Mɔgɔ minnu b'u yɛrɛ furakɛ, olu ka kan ka dɔgɔtɔrɔ dɔ ka dɛmɛ ɲini."]
["Aw ye aw janto nin na: ɛntɛrinɛti min bɛ jabi di ɲininkaliw jaabiliw kan, o dabali ka jugu kɛrɛnkɛrɛnnenya la n'a bɛ tali kɛ jatidenw de kan. Misali la, mɔgɔ hakɛ min ka bana kofɔra."]
['Aw bɛ ladilikan ɲini tuma bɛɛ aw ka dɔgɔtɔrɔ fɛ walima kɛnɛya baarakɛla dɔ wɛrɛ min bɛ se ka aw dɛmɛ ka aw ka bana furakɛ. Aw kana dɔgɔtɔrɔ ka ladilikan bila ka suma walima ka mɛn a ɲini na sabu aw ye fɛn dɔ kalan nin siti kan. Ni aw hakili la ko aw ka bana bɛ se ka juguya joona, aw ye 911 wele walima aw ka taa aw ka dɔgɔtɔrɔso la joona joona. Nin siti tɛ dɔkɔtɔrɔ ni banabagatɔ ka jɛɲɔgɔnya jira. BioMedLib ni a ka baaradenw, walima nin siti dɛmɛbaga si tɛ kuma si jira walima ka jira, nin kunnafoniw wala u labaarali kama.']
['Kunnafoni min lakodɔnna:']
["Digital Millennium Copyright Act san 1998, 17 U.S.C. § 512 (a DMCA) bɛ sariya sigi ka ɲɛsin mɔgɔ ma min b'a miiri ko fɛn min bɛ sɔrɔ intɛrinɛti kan, o bɛ tɔɲɔ a ka sariyaw la Ameriki jamana ka sariya kɔnɔ. "]
['Ni i dara a la kô i ka site web ni a baara tchogow bè i ka lakananifènw la, i (walima i ka lasigiden) bè se ka i yèrè ka lakananifènw bila ka o site web ni a baara tchogow bila kènèkan.']
['Waajibi don kunnafoniw ci kɛtɔ ka kɛ sɛbɛn ye e-mail fɛ (e-mail de kan ka lajɛ sɛbɛn ɲɛ Kunnafoniw lajɛ yɔrɔ la).']
["DMCA bɛ a ɲini i ka kunnafoni sɛbɛn bɔlɔlɔw kan kojugukɛ sɛbɛn bɛ sɛbɛn min kɔnɔ, o ka kan ka nin kunnafoniw fara a kan: (1) sɛbɛnni kɛtɔ ka sɛbɛnni kɛ min bɛ sɛbɛnni kɛ ni a ma kɛ kojugukɛ sɛbɛn ye; (2) sɛbɛnni kɛtɔ ka fɛn kofɔlen in kofɔ ani kunnafoni minnu bɛ a to an bɛ se ka fɛn kofɔlen in sɔrɔ; (3) i ka ladɛrɛsi, i ka ladɛrɛsi, telefɔni nimɔrɔ ani i ka ladɛrɛsi; (4) i ka kumaɲɔgɔnya sɛbɛn ko i dalen b'a la ko i bɛ a dɔn ko i bɛ ka fɛn kofɔlen in kɛ ni sariya min ye, o ma di sɛbɛnnikɛbaga ma, a ka ladɛrɛsi tigi, walima sariya wɛrɛ; "]
['(5) a ka seereyasɛbɛ sɛbɛntiya, ko seereyasɛbɛ minɛnen bɛ kojugubakɛlaw kama, ko kunnafoni min bɛ o sɛbɛntiya kɔnɔ, ko tiɲɛ don ani ko i yamaruyalen don ka yamaruya di ka kɛwalew kɛ minnu bɛ tiɲɛni kɛ; ']
['ani (6) sɛbɛn dɔ ka boloci walima ɛkitɔrɔniki sɛbɛn sɛbɛn tigi fɛ walima mɔgɔ min yamaruyara ka baara kɛ sɛbɛn tigi tɔgɔla. ']
['Ni kunnafoni fɔlen ninnu bɛɛ ma fara ɲɔgɔn kan, o bɛ se ka kɛ sababu ye ka mɛn baara kɛli la i ka sɛbɛn kɔnɔ.']
['Ɲɔgɔnkunbɛn']
['Sɛbɛn ɲɛ Ɲiningaliw / ladiliw']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
Disclaimer: medical
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['Ɲɔgɔn fɛ']
['BioMedLib bɛ baara kɛ ni ɔridinatɛri ye (mansin ka kalan aligoritimiw) walasa ka ɲininkaliw ni jaabiw di ɲɔgɔn ma.']
['An bɛ a daminɛ ni gafew miliyɔn 35 ye ka bɔ PubMed/Medline. Ani fana, ɛntɛrinɛti sitiw ka bɔ RefinedWeb.']
