Bushe ubulwele bwa kansa ya ku prostate bwaba shani?
Icipimo ca malwele ya kansa ya kuli bongobongo cilosha ku fyo ubulwele bwaba.
Kansa ya prostate ni kansa yabipa iilefuma mu nsandesande sha prostate gland, umubili uunono uwapala walnut uwaba pa nshi ya bladder ku baume.
Umulopa wa prostate e ulenga amenshi ya bwaume, ayalenga umulopa wa bwaume ukukanaba sana.
Icilenga sana abantu ukukwata kansa ya kuli bongobongo tacalishibikwa bwino bwino, lelo kwaliba ifintu ifingi ifilenga abantu ukukwata sana ubu bulwele.
Pali ifi paba no mushinku, ulupwa, umushobo, ne fyabipa fimo ifingacitika ku muntu.
Ubulwele bwa kansa bwa prostate bwaba sana ku bakalamba, kabili ubulwele ubukalamba bwaba ku bakalamba abali ne myaka ukucila pali 65.
Na kabili, abaume abakwata balupwa abalwala kansa ya kuli bongobongo balafwa bwangu, nga filya fine caba ku baume abafuma ku Africa na ku bena Caribbean.
Ubulwele bwa kansa ya ku prostate bwaba pa kuti insandesande shilakula no kulekana ukwabula ukuleka.
Ici kuti cacitika pa mulandu wa kwaluka kwa nsandesande ishilenga ukuti ifilundwa fimo ifilenga umuntu ukukula filebomba sana nelyo pa mulandu wa kuti insandesande ishilenga umuntu ukukula shilabomba bwino.
Ifi fintu kuti fyalenga insandesande ukukula ukwabula ukuleka, ne ci kuti calenga ifilonda ukupanga.
Ilyo ili kansa lyatendeka ukukula, kuti lyatendeka ukwingila mu filundwa fya mubili ifyaba mupepi, pamo nga mu mala, mu mulomo, na mu nsandesande sha mumala ishaba mupepi.
Limo limo, insandesande sha kansa kuti shafuma ku tumor no kuya ku filundwa fimbi ifya mubili ukupitila mu mulopa nelyo mu nsandesande sha mu mala, kabili ici citwa ukuti metastasis.
Nga ca kuti kansa yasalangene, kuti cayafya sana ukuundapa.
Ubulwele bwa kansa ya ku prostate kuti bwaba no mulandu wa ma hormone, maka maka testosterone.
Testosterone kuti yalenga insandesande sha kansa ya kuli bongobongo ukulakulilako, kabili imiti iingi iya kundapa kansa ya kuli bongobongo yafwaya ukucefyako iyi hormone nelyo ukulesha ukubomba bwino.
Mu kwipifya, pa kuti umuntu alwale kansa ya ku prostate kano alekula no kulekana kwa nsandesande mu prostate gland, kabili kuti kwaba ifilenga umuntu alwale kansa.
Ukwishiba bwino bwino icilenga ubulwele ukucitika kwalicindama sana pa kuti umuntu engapanga umuti wa kuundapa bwino abalwele ba kansa ya kuli bongobongo.
Zobniw CM, Causebrook A, Fong MK: Clinical use of abiraterone in the treatment of metastatic castration-resistant prostate cancer. Res Rep Urol. 2014, 6 (): 97-105.
Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.
Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.
Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.
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What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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