Puɔ̈ɔ̈c de tuany ë puɔ̈u, man ye cɔl'myocardial infarction' ee rot looi të cenë riɛm ya tek ku bï ya lɔ në biäk de cuäŋ yic, ku bï ya naŋ kë ye yen cɔk thou.
Kän ee rot looi të cenë cuäŋ de cuäŋ ya thiöök në riɛm yic, man ye lac bɛ̈n në kë de atherosclerosis, ye rot looi të cenë cuäŋ ya thiöök në riɛm yic.
Kë ye cɔl plaque ee cholesterol, fatty substances, cellular waste products, calcium, ku fibrin.
Të cenë kë thiin tɔ̈ në puɔ̈u ya dhoŋ kɔ̈u, ke ka lëu bï riɛm ya thiɛ̈ɛ̈k, ago ya thiɛ̈ɛ̈k ku bï riɛm cïï thiäŋ në oxygen ya cɔk lɔ në puɔ̈u.
Kë cïn oxygen kënë ee cells ke puɔ̈u cɔk thou, ku ka ye naŋ kë ye cɔl heart attack.
Të ye tuaany rot lac looi thïn ee rot gɛɛi në ɣän ye tuɛnytuɛny ke riɛm ya dɔm thïn ku jɔl ya kaam ye tuɛnytuɛny ke riɛm dɔm ku jɔl ya kaam ye akïm kɔc kony.
Wal de tuany ë puɔ̈u ee rot lac looi në dhuk ë riɛm tënë puɔ̈u yic në kaam thiin koor yic, ke ye wal ka kuɛɛr cït angioplasty ku stenting ka coronary artery bypass surgery.
Kë thiekic ba nyic ee lɔnadɛ̈ ke puɔ̈ɔ̈c de tuaany ë puɔ̈u ee kë rilic apɛi ku ee yic naŋ käjuëc ye kek cɔk loi röt, agut cï kä ye kek dhiëëth, pïïrden ku jɔl ya kä ye kek rëër thïn.
Kä ye raan cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë.
Cɔl kä juääc kä aa lëu bïk yï kony ba ciɛ̈n kë ye yï cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë ye cɔl a nɔŋ kë.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
['Wɛ̈t cï lueel: wal']
['Wɛbthait kën ee piööc ku wël tɔ̈u thïn ku acie kë ye looi bï kɔc ke akïm ya lɛ̈k wala bï kɔc ke luɔi ya kony.']
['Wël cï kek ya lueel në ye athöör kënë yic acïï lëu bï kek ya kony agonë tuanytuɛny ya ŋic ku bï kek ya kony, ku kɔc wïc kuɔɔny de akïm adhil akïm de akïm ya thiëëc bï kek ya kony.']
['Tiɛ̈ŋë kä ye kuɔɔny de neural net looi, man ye kä ye bɛ̈n në kä cï kek ya thiëëc yiic, aye kek cɔk tɔ̈ ëke cïï kek ye deet yiic në thɛɛr juëc yiic. Cïmën de, ciin de kɔc cï kek yök ëke tuany.']
['Thïëc wɛ̈t akïmdu ka raan dɛ̈t wënë cï piöc ë pialgup wënë cï yök në biäk de tuaany. Duk wɛ̈t akïm ye kuɛ̈ɛ̈c nhom cɔk alɔn cï yen ya kë cï kueen në wɛbthait kënnë yic. Na ye tak ke yïn nɔŋ kë wïc bë yï kony në tuany, cɔl 911 ka lɔɔr në ɣön akïm tɔ̈u të thiääk ke yï në kaam thiin koor yic. Acïn akïm-tuany ye thiääk në yen në wɛbthait kënnë yic ka luɔ̈i de. BioMedLib ka kɔc cï luöi, ka raan ëbɛ̈n ye luui në yen në wɛbthait kënnë yic, acïn kë ye lueel, ye lueel ka ye lueel, në kë de wël cï kek gɔ̈ɔ̈r në ye athöör kënnë yic ka luɔ̈i de.']
['Wɛ̈t cï lueel: copyright']
['Lööŋ ke Digital Millennium Copyright Act de 1998, 17 U.S.C. § 512 (ye DMCA) ee kɔc ye käke kɔc gɔ̈t gam ke kä tɔ̈ në Internet aye käken tɔ̈ në U.S. copyright law dhoŋ kɔ̈th. ']
['Na ye gam në yith ke këriɛ̈ɛ̈c ka kä cï gɔ̈ɔ̈r tɔ̈ në wɛbthaitda ka kuɔɔnyda yic acï löŋ de gël de kä cï gɔ̈ɔ̈r dhoŋ kɔ̈u, ke yïn (ka raan ye yïn luɔ̈ɔ̈i) alëu ba ɣok tuɔ̈c wɛ̈t bï këriɛ̈ɛ̈c ka kä cï gɔ̈ɔ̈r nyaai, ka bï yïn pëën ba lɔ thïn. ']
["Wël aa bë ke tuɔɔc në athöör yic në email (tïŋ 'Rin de athöör' në biäk de email)."]
['DMCA ee wïc lɔnadɛ̈ ke kë ca lueel de kä cï keek gɔ̈ɔ̈r cï keek luɔ̈ɔ̈i anɔŋic wël cï keek gɔ̈ɔ̈r: (1) luɛɛl de kä cï keek luɔ̈ɔ̈i cï keek gɔ̈ɔ̈r cï keek luɔ̈ɔ̈i cï keek lueel; (2) luɛɛl de kä cï keek gɔ̈ɔ̈r cï keek lueel ku wël cï keek lueel bï keek cɔk lëu bïkë kä cï keek gɔ̈ɔ̈r yök; (3) wël bï yïn ke kɔn ya kɔɔr, nɔŋiic adrɛ̈ɛ̈rdu, namba de telepun ku adrɛ̈ɛ̈r de imeel; (4) wɛ̈t cï lueel në yï nhom lɔnadɛ̈ ke yïn ye gam ke kë cï gɔ̈ɔ̈r cï looi në kueer cï yïn ye gaam acie gam në raan de kä cï keek gɔ̈ɔ̈r cï keek luɔ̈ɔ̈i, ka raan de lööŋ, ka në luɔɔi de löŋ; ']
['(5) athöör cï yïn gätpiny në awuɔ̈c de wɛ̈t lueth, lɔnadɛ̈ ke wël tɔ̈ në athöör yic aye yith ku lɔnadɛ̈ ke yïn anɔŋ riɛl ba kä cï gɔ̈t ke lööŋ ke raan nɔŋ nhïïm ya looi në kë cï wuɔ̈ɔ̈c; ']
['Ku (6) ke ye kä cï gɔ̈t ke guɔ̈p ka kä cï gɔ̈t ke mɛ̈ɛ̈t de raan de kä cï luɔ̈ɔ̈i ka raan cï gäm riɛl bï luui në nyin de raan de kä cï luɔ̈ɔ̈i. ']
['Na këc wël cï gɔ̈ɔ̈r nhial tɔ̈u thïn kedhiɛ gɔ̈t, ke ka lëu bï këdun cï yïn gaam ya gääu.']
['Thiëc kek']
['Tuɔɔc email tënë ɣok na nɔŋ këdun thiëc/cï yïn tak.']
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
Disclaimer: medical
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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