A ka teli ka mɔgɔ fari yɔrɔ bɛɛ funu. A ka jugu ka mɔgɔ fari yɔrɔ bɛɛ funu.
A sababuw be se ka kɛ fɛɛn caaman ye, i n'a fɔ farilajidɛsɛ, banamisɛnniw, walima bana wɛrɛw.
A furakɛcogo be bɔ bana juguya hakɛ ni a sababu la.
A ka teli ka kɛ ko mɔgɔ dɔw ka teli ka kɛ ni nin bana suguya ye:
O furaw be fari funu ani ka ŋɛɲɛ ban. U be fari funu ka sɔrɔ ka farilajidɛsɛ bila mɔgɔ la.
Fura dɔw bɛ yen i tɛ mɔgɔ ci ka olu ta i n'a fɔ difenidaramini (Benadryl) wala loratadine (Claritin).
Ni bana juguyara, dɔgɔtɔrɔ be se ka dimimadafura di.
2. Kirikirimasiɲɛfuraw: Ni kirikirimasiɲɛfuraw ma se ka bana furakɛ, u be se ka kirikirimasiɲɛfura dɔw di i n'a fɔ peredinizɔni (prednisone) walisa ka bana nɔgɔya ani ka bana kɛlɛ.
A ka ca a la, u be o furaw ta ni mɔgɔ ye ani u be mɔgɔ dɛmɛ joona joona.
Fura minw be fanga di farikolo ma: Fura minw be fanga di farikolo ma, n'u tɛ bana furakɛ ka ɲɛ, u be se ka kɛ sikulɔsiporini (cyclosporine) wala metotɛrɛkisati (méthotrexate) ye.
4. omalizumab: O ye fura ye min bɛ se ka pikiri kɛ ani a bɛ se ka poroteyini immunoglobuline E (IgE) bali ka baara kɛ.
A ka teli ka kɛ ka sumaya jugumanw furakɛ minnu tɛ furakɛ ka ɲɛ.
5. Fura minw ka ɲi ka kɛ sababu ye ka kurukuru bila mɔgɔ la, aw kana olu ta: Fura minw ka ɲi ka kɛ sababu ye ka kurukuru bila mɔgɔ la ani aw kana u ta, o be se ka aw tanga a ma.
Tuma caaman na, dumuni dɔw, fura dɔw, fɛnɲɛnama dɔw ka kinni, funteni, nɛnɛ ani tile y'a to a be dimi.
6. Finimugu sumanin dɔ biri a kan: Finimugu sumanin dɔ biri a kan, o be se k'a to a tɛ funu ani a tɛ ŋɛɲɛ.
7. Fani jalan: Fani jalan be se ka kɛ ka ŋɛɲɛ nɔgɔya ani ka sumaya nɔgɔya.
O kɔ, u be fani jalan dɔ biri a kan walisa a kana funu.
8. Finimugu min be yeelen bɔ: Finimugu min be yeelen bɔ, o be se ka banabagatɔ fari tanga ani ka sumaya juguman dɔw nɔgɔya.
A ka ɲi i ka dɔgɔtɔrɔ dɔ ɲininka walisa ka kɔn ka furakɛli kɛ.
U be se k'a dɛmɛ k'a lɔn bana min b'a la ani k'a furakɛ cogo ɲuman na.
Deleanu D, Nedelea I, Petricau C, Leru P, Dumitrascu D, Muntean A: Clinical impact of omalizumab in refractory chronic urticaria: One centre experience. Exp Ther Med. 2019, 18 (6): 5078-5081.
Grundmann SA, Kiefer S, Luger TA, Brehler R: Delayed pressure urticaria - dapsone heading for first-line therapy? J Dtsch Dermatol Ges. 2011, 9 (11): 908-12.
Staumont-Sallé D, Piette F, Delaporte E: [Etiological diagnosis and treatment of chronic urticaria]. Rev Med Interne. 2003, 24 (1): 34-44.
Akashi R, Ishiguro N, Shimizu S, Kawashima M: Clinical study of the relationship between Helicobacter pylori and chronic urticaria and prurigo chronica multiformis: effectiveness of eradication therapy for Helicobacter pylori. J Dermatol. 2011, 38 (8): 761-6.
Calogiuri G, Nettis E, Mandurino-Mirizzi A, Di Leo E, Macchia L, Foti C, Vacca A, Kounis NG: Omalizumab for the Treatment of Persistent Drug Induced Urticaria Elicited by Thienopyridines: A Case Report. Antiinflamm Antiallergy Agents Med Chem. 2020, 19 (3): 335-339.
['Kunnafoni nafaman: furakɛli']
["O site web nin labɛnna walisa ka mɔgɔw kalan ani k'u kunnafoni dɔrɔn."]
["Mɔgɔ minw b'o gafew kalan, olu man kan k'u kɛ ka bana dɔw furakɛ."]
['Aw ye aw janto nin na: ɛntɛrinɛti min bɛ ɲiningaliw jaabiw labɛn, a tɛ se ka jatidenw fɔ ka ɲɛ. misali la, bana kɛrɛnkɛrɛnnen dɔ bɛ mɔgɔ minw na.']
["Tuma bɛɛ i ka kan ka ladili ɲini i ka dɔgɔtɔrɔ fɛ wala kɛnɛya baarakɛla dɔ fɛ bana dɔ koo la. I kana ban ka ladili ɲini dɔgɔtɔrɔ fɛ wala ka mɛɛn a ɲinili la sabu i ye koo dɔ kalan site web nin kan. N'i miirila ko bana dɔ be i kan, i ka kan ka teliya ka mɔgɔ wele 911 wala ka taga dɔgɔtɔrɔso la joona joona."]
['Kunnafoniw: sɛbɛkɔrɔ']
["Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (DMCA) ye sariya dɔ ye min b'a to mɔgɔ minw b'u ka gafew sɔrɔ ɛntɛrinɛti kan, olu ka se k'u yɛrɛ lafasa."]
["N'i lanin b'a la ko kunnafoni wala fɛɛn o fɛɛn be sɔrɔ an ka site web kan wala an ka baaraw kan, k'o be i ka sariya tiɲɛ, i (wala i ka lasigiden) be se k'o kunnafoni wala fɛɛn bɔ yen wala k'a bali k'a sɔrɔ."]
['I ka kan ka ci nin ci nin kɛ e-mail fɛ (i ka e-mail lajɛ yɔrɔ nin na)']
["DMCA b'a ɲini ko i ka kunnafoni nunu fara i ka kunnafoni kan: (1) kunnafoni min b'a yira ko i ye baara dɔ kɛ min ka kan ni sariya ye; (2) kunnafoni min b'a yira ko i ye baara dɔ kɛ min ma sariya labato ani kunnafoni wɛrɛw minw b'a to an be se k'o kunnafoni sɔrɔ; (3) i ka ladɛrɛsi, telefɔni nimɔrɔ ani e-mail; (4) i ka seereya dɔ ko i lanin b'a la ko i ye baara min kɛ, ko sariya t'o kɛ."]
["5. i ka sɛbɛ dɔ kɛ ka yira ko i ye tiɲɛn fɔ ani ko joo b'i fɛ ka joo dɔ latanga."]
["O kama, a ka ɲi i k'a ɲini k'a lɔn n'i ka ɲi k'o gafew wala videwo nunu jati k'u ye gafew ye minw labɛnna mɔgɔw ye."]
["N'i ma kunnafoni nunu bɛɛ di, a be se ka kɛ ko i ka ɲinini baara be mɛɛn."]
['Kumaɲɔgɔnya kɛ']
["N'i ka ɲiningaliw wala i ka ladiliw be yen, an ci e-mail fɛ."]
How to treat urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by itchy, raised welts that can appear on any part of the body.
It can be caused by various factors, including allergic reactions, infections, or underlying medical conditions.
Treatment for urticaria depends on the severity of the condition and the underlying cause.
Here are some common treatment options:
1. Antihistamines: These medications help to reduce itching and swelling by blocking the effects of histamine, a chemical released by the body during an allergic reaction.
Over-the-counter antihistamines such as diphenhydramine (Benadryl) or loratadine (Claritin) can be effective in mild cases.
In more severe cases, a doctor may prescribe stronger antihistamines.
2. Corticosteroids: In cases where antihistamines are not effective, corticosteroids such as prednisone may be prescribed to reduce inflammation and suppress the immune system.
These medications are usually taken orally and can provide quick relief.
3. Immunosuppressants: In cases of chronic urticaria that do not respond to other treatments, immunosuppressant drugs such as cyclosporine or methotrexate may be prescribed to suppress the immune system and reduce inflammation.
4. Omalizumab: This medication is an injectable monoclonal antibody that targets and blocks immunoglobulin E (IgE), a protein involved in allergic reactions.
It is typically used for chronic urticaria that does not respond to other treatments.
5. Avoid triggers: Identifying and avoiding triggers that cause urticaria can help prevent outbreaks.
Common triggers include certain foods, medications, insect bites, and exposure to heat, cold, or sunlight.
6. Cold compress: Applying a cold compress to the affected area can help reduce itching and swelling.
7. Wet wraps: Wet wraps can be used to soothe itching and reduce inflammation.
A damp cloth is wrapped around the affected area, and then a dry cloth is placed over it to retain moisture.
8. Phototherapy: Exposure to ultraviolet light can help suppress the immune system and reduce inflammation in some cases of chronic urticaria.
It is important to consult a healthcare professional for proper diagnosis and treatment of urticaria.
They can help determine the underlying cause and recommend the most appropriate treatment plan.
Disclaimer: medical
This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.
The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.
Disclaimer: copyright
The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.
['A ka kan ka kɛ']
['BioMedLib be baara kɛ ni ɔridinatɛri ye (mansin ka kalan aligoritimiw) ka ɲiningaliw ni jaabiliw dilan ɲɔgɔn fɛ.']
['An ye gafe miliyɔn 35 ta ka bɔ PubMed/Medline ani RefinedWeb kan.']
