Pathophysiology of urticaria jechuun maal jechuudha?
Urticaria, yeroo baay'ee hives jedhamuun kan beekamu, dhukkubni gogaa gogaa irratti mul'atu, kan ol ka'e, kan diimaa ta'e (wheels) dha.
Pathophysiology of urticaria release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues. Pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
Urticaria cimaa keessatti, histamine fi mediators kan biroon kan ba'an allergic reaction to a specific allergen, such as food, medication, or insect sting.
Kunis seelota maast seelii akka hin sochoone kan taasisu si'a ta'u, histamine fi mi'oota biroo kan dhiigaa akka dhangala'an taasisan kan gadhiisan si'a ta'u, kunis gara wheelsitti geessa.
Urticaria'n yeroo baay'ee sababni isaa kan hin beekamne yoo ta'u, garuu mala autoimmune'n kan wal qabate akka ta'e yaadama.
Haalli kun yoo ta'e, qaamni autoantibodies kan high-affinity IgE receptor (FcεRI) on mast cells irratti xiyyeeffatu ni uuma, kunis akka isaan socho'an fi histamine fi mediators biroo akka ba'an taasisa.
Urticaria cimaa fi yeroo dheeraa keessatti, histamine fi mediators biroon bilisa ba'uun mallattoowwan adda ta'an kan akka itching, redness, fi swelling fiduu danda'a.
Yaaliin wal'aansaa yeroo baay'ee dhiibbaa histamiinii dhabamsiisuu fi mallattoowwan hir'isuuf antihistamiinii fayyadamuu kan dabalatudha.
Dhukkubni kun baay'ee cimaa yoo ta'e, qorichoonni biroon kan akka kortikosterooyidii ykn omaliizuumaab dhukkubicha to'achuuf itti fayyadamuu danda'u.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numerical content. For example, the number of people diagnosed with a specific disease.
Yeroo hunda waa'ee dhukkuba tokkoo gorsa ogeessa fayyaa ykn ogeessa fayyaa ga'eessa ta'e biraa gaafadhu. Gorsa fayyaa ogeessaa hin tuffatin ykn waan weebsaayitii kana irratti dubbisteef isa barbaaduu hin daangeffatin. Yoo rakkoo fayyaa qabaachuu dandeessan yoo ta'e, hatattamaan 911 bilbilaa ykn gara kutaa ariifachiisaa dhihoo deemaa. Weebsaayitii kana ykn itti fayyadama isaatiin hariiroon ogeessa fayyaa fi dhukkubsataa hin uumamu. BioMedLib ykn hojjettoonni isaa, ykn namni weebsaayitii kana irratti gumaache kamiyyuu, odeeffannoo asitti kenname ykn itti fayyadama isaa ilaalchisee ibsa ykn yaada hin kennine hin kennine.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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