Ti Alzheimer's disease ket progresibo a sakit a mangdadael iti utek, a mangapektar iti utek, mangpakapuy iti memoria, mangpakapuy iti abilidad a mangilasin iti bambanag, ken mangbalbaliw iti kababalin.
Saan a naan-anay a maawatan ti eksakto a makagapu iti Alzheimer's disease, ngem patienda a gapu dayta iti kombinasion dagiti genetic, environmental, ken estilo ti panagbiag.
Dadduma kadagiti banag a mabalin a pakaigapuan ti panagsakit ti Alzheimer ket:
1. Genetics: Nailasin ti sumagmamano a gene a mangpakaro iti posibilidad a maaddaan iti Alzheimer's disease, nangruna ti gene nga apolipoprotein E (APOE).
2. Edad: Umad-adu ti agsakit iti Alzheimer's disease bayat ti panaglakay wenno panagbaket, ket kaaduan a tattao ti nadayagnos nga addaan iti dayta kalpasan ti edad a 65.
3. Pakasaritaan ti pamilia: Ti kaadda ti pakasaritaan ti pamilia iti Alzheimer's disease mabalin a mangnayon iti peggad ti panagsakit.
4. Pannakasugatan ti ulo: Ti napalabas a pannakadangran ti ulo, nangnangruna dagidiay agbanag iti pannakapukaw ti puot, mabalin a mangnayon iti peggad ti Alzheimer's disease.
5. Dagiti pakaigapuan ti panagsakit ti puso: Dagiti kasasaad a kas iti alta presion, nangato a kolesterol, ken diabetes mabalin a pakaruenna ti peggad ti panagsakit iti Alzheimer's disease.
6. Estilo ti panagbiag: Ti saan a nabannog a panagbiag, di umiso a taraon, ken kurang a panagpanunot mabalin a mangpakaro iti peggad ti Alzheimer's disease.
7. Pannaka-inflammation: Ti napaut a pannaka-inflammation ti utek mabalin a pakaigapuan ti Alzheimer's disease.
8. Oxidative stress: Ti di panagkatunosan ti pannakapataud dagiti free radical ken ti abilidad ti bagi a mangikkat iti sabidongda mabalin a pakaigapuan ti panagsakit ti Alzheimer.
9. Amyloid plaques ken neurofibrillary tangles: Ti panagurnong kadagiti amyloid plaques ken neurofibrillary tangles iti utek ket maysa a pakailasinan ti Alzheimer's disease, ngem saan a naan-anay a maawatan ti akemda iti itataud ti kasasaad.
Napateg a laglagipen a komplikado ti makagapu iti Alzheimer's disease ken mabalin a ramanenna ti panagtitipon dagitoy a banag.
Agtultuloy ti panagsirarak tapno ad-adda a maawatan dagiti kangrunaan a makagapu ken mapataud dagiti epektibo a panangagas iti daytoy makadadael a kasasaad.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
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What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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