Aṭṭan n sskeṛ yezmer ad iḥaz imdanen n yal leɛmer, n yal tawsit neɣ n yal azgen.
Maca, llan kra n yimgan zemren ad snernin amihi n waṭṭan n sskeṛ, gar-asen:
1. Amezruy n twacult: Ma yella yiwen seg imawlan- ik neɣ seg watmaten- ik yuḍen sskeṛ, d ayen ara yernun amihi n waṭṭan- agi.
2. Leɛmer: Amihi n waṭṭan n sskeṛ n ṣṣenf wis 2 yettzid skud imdanen ttimɣuren, ladɣa mbeɛd 45 n yiseggasen.
3. Taẓayt: Ma yella yiwen d azuran neɣ d afatan, yezmer ad t-yeṭṭef waṭṭan n sskeṛ n ṣṣenf wis sin.
4. Asebded n tikli: Asebded n tikli ur nṣubb ara yezmer ad yesnerni amihi n waṭṭan n sskeṛ n ṣṣenf wis 2.
5. Tawsit d tawsit: Kra n yegrawen d yegrawen am Imarikaniyen iberkanen, Imarikaniyen ispaniyen, Imarikaniyen inaṣliyen, Imarikaniyen n Asya d wid n tegzirin n Pasifik, sɛan amihi meqqren n waṭṭan n sskeṛ n ṣṣenf wis 2.
6. Aṭṭan n sskeṛ n tlawin yellan s tadist: Tilawin i yuḍnen aṭṭan n sskeṛ n tlawin yellan s tadist, yezmer lḥal ad tent-yeṭṭef sskeṛ n ṣṣenf wis 2 ticki ad ilint d timɣarin.
7. Aṭṭan n tmellalin timseksumin (SOPK): Tilawin yesεan SOPK, ttemɣayent deg umihi n waṭṭan n sskeṛ n ṣṣenf wis 2.
8. Tanezzayt: Wid yesεan tanezzayt sɛan idammen n sskeṛ yugaren win n tannumi maca ur εlay ara akken ad t-nḍeṛṛeb d win yesεan aṭṭan n sskeṛ.
Llan deg umihi meqqren n waṭṭan n sskeṛ n ṣṣenf wis 2.
9. Tanezzayt n yidammen: Ma tesεiḍ tanezzayt n yidammen (tanezzayt n yidammen) tezmer ad ternuḍ amihi n waṭṭan n sskeṛ n ṣṣenf wis 2.
10. Aswir n kulistṛul d win n triglisirid ur nṣeḥḥa ara: Aswir n kulistṛul d win n triglisirid yezmer ad yesnerni amihi n waṭṭan n sskeṛ n ṣṣenf wis 2.
11. Aṭṭanen n wul: Wid yesεan yakan aṭṭanen n wul, yezmer ad ten-yeṭṭef waṭṭan n sskeṛ wis sin.
D ayen yesɛan azal ad t-nẓer belli ɣas ma yella imgan-a zemren ad snernin amihi n waṭṭan n ssker, mačči d medden akk yesɛan imgan-a n umihi ara yesɛun aṭṭan-a.
Asebeddel n leεwayed n tudert, am wučči n tgella yelhan, aḥader n taẓayt yelhan, d uḥric n waddal s waṭas, yezmer ad yenqes umihi n waṭṭan n sskeṛ.
Swift S, White S: Could islet transplantation be a potential cure for diabetes? Nurs Times. , 99 (15): 48-9.
Anderson BJ: Who benefits from intensive therapy in type 1 diabetes? A fresh perspective, more questions, and hope. Diabetes Care. 2003, 26 (7): 2204-6.
Lisenby KM, Meyer A, Slater NA: Is an SGLT2 inhibitor right for your patient with type 2 diabetes? J Fam Pract. 2016, 65 (9): 587-93.
['Anfafad: amejjay']
['Asmel-a web yettunefk-d kan i yiswiyen n uselmed d usissen, ur d-yettak ara iwellihen imejjayen neɣ isefka udmawanen.']
['Isallen i d-yettunefken ur ilaq ara ad ttwasqedcen i usebded neɣ i usemḥi n kra n wugur n tezmert neɣ aṭṭan, yerna wid yettnadin ɣef yiwellihen imsujjiyen udmawanen yessefk ad ẓren amejjay yesɛan turagt.']
['Ttxil-k ẓer belli azeṭṭa amseddayan i d-yettaken tiririyin i tuttriwin, ur iṣeḥḥa ara mliḥ ma yella d ayen yerzan uḍḍun. D amedya, amḍan n yimdanen yettwaḍḍfen s kra n waṭṭan.']
['Ssutur dima ṛṛay n ṭṭbib-ik neɣ n kra n umejjay-nniḍen ara iwalmen ɣef waṭṭan-ik. Ɣur-k ad tḥeqreḍ ṛṛay n ṭṭbib amsadur neɣ ad tεeṭṭleḍ deg usuter-is ɣef lǧal n kra i teɣriḍ deg usmel-a. Ma tɣileḍ ahat tesεiḍ liḥala n tεeǧlanit deg tezmert, siwel 911 neɣ ṛuḥ ɣer texxamt n tεeǧlanit i d-iqerben. Ulac assaɣ gar ṭṭbib d umuḍin i d-yettnulfun s usmel-a neɣ aseqdec-is. BioMedLib neɣ ixeddamen-is, neɣ kra n win yettekkiyen deg usmel-a, ur d-ttaken ara yakk kra n yisumren, s wawal neɣ s usemres, ɣef ayen yerzan isallen i d-yettunefken dagi neɣ aseqdec-is.']
['Anekcum: azref n uḍfar']
['Asaḍuf n umeskar n yizerfan n wemdan n alfin (Digital Millennium Copyright Act) n 1998, 17 U.S.C. § 512 (DMCA) yettak-d ttawil i yimeskaren n yizerfan n wemdan i yumnen belli ayen i d-yettbanen deg Internet yettḍurru izerfan-nsen ddaw usaḍuf n umeskar n yizerfan n wemdan n Marikan. ']
['Ma tumneḍ s wul yelhan belli kra n taɣult neɣ taɣawsa yettwafken deg unermes-nneɣ neɣ deg yiqeddacen-nneɣ tegzem azref-ik n uḥraz, tzemreḍ (neɣ amsedday-ik) ad aɣ-d-tazneḍ izen akken ad nessuter asuffeɣ n taɣult neɣ n taɣawsa-nni, neɣ ad nessader tuqqna ɣer-s. ']
['Isental yessefk ad ttwazen s tira s yimayl (ẓer asebter "Contact" i tansa n yimayl).']
['DMCA teḥwaǧ isalli-inek n tukksa n yizerfan yettwanekṛen ad d-yeglu s yisallen-a: (1) asegzi n umahil yettwaḥerzen s yizerfan yettwanekṛen i d-yettwanekren; (2) asegzi n uḍris yettwanekren d yisallen ara aɣ-yeǧǧen ad d-naf uḍris-nni; (3) isallen n usiwel inek, ladɣa tansa-inek, uṭṭun n tilifun d tansa imayl; (4) awal sɣur-k d akken tumneḍ s wayen yelhan belli aḍris-nni s wamek i d-tettcetkiḍ ur yettwasemres ara sɣur bab n yizerfan, neɣ amesten-is, neɣ s useqdec n kra n usaḍuf; ']
['(5) yiwet n tseddi sɣur-k, yettwatten s uḥasef n lekdeb, d akken isallen yellan deg uzenzi d iṣeḥḥan u tesεiḍ azref ad tesseḍruḍ izerfan umeskar i d-yeqqaren ttwarẓen; ']
['akked (6) azmul n tfekka neɣ n tuqqna tagensit n bab n yizerfan neɣ n umdan yettwafernen ad yexdem s yisem n bab n yizerfan. ']
['Ma ur d-terriḍ ara isallen-agi akk, yezmer ad d-yeglu s uεeṭṭel deg usefrek n ucetki-inek.']
['Assaɣ']
['Ttxil-k azen-aɣ-d imayl ma tesεiḍ asteqsi / asumer.']
Who gets diabetes?
Diabetes can affect people of any age, race, or gender.
However, certain factors can increase the risk of developing diabetes, including:
1. Family history: Having a parent or sibling with diabetes increases the risk of developing the condition.
2. Age: The risk of type 2 diabetes increases as people get older, especially after the age of 45.
3. Weight: Being overweight or obese increases the risk of developing type 2 diabetes.
4. Physical inactivity: Lack of regular physical activity can increase the risk of type 2 diabetes.
5. Race and ethnicity: Certain racial and ethnic groups, such as African Americans, Hispanic/Latino Americans, Native Americans, Asian Americans, and Pacific Islanders, have a higher risk of developing type 2 diabetes.
6. Gestational diabetes: Women who have had gestational diabetes during pregnancy are at an increased risk of developing type 2 diabetes later in life.
7. Polycystic ovary syndrome (PCOS): Women with PCOS have an increased risk of developing type 2 diabetes.
8. Prediabetes: People with prediabetes have blood glucose levels higher than normal but not high enough to be diagnosed with diabetes.
They are at an increased risk of developing type 2 diabetes.
9. High blood pressure: Having high blood pressure (hypertension) can increase the risk of developing type 2 diabetes.
10. Abnormal cholesterol and triglyceride levels: High cholesterol and triglyceride levels can increase the risk of type 2 diabetes.
11. History of cardiovascular disease: People with a history of cardiovascular disease are at an increased risk of developing type 2 diabetes.
It is important to note that while these factors can increase the risk of developing diabetes, not everyone with these risk factors will develop the condition.
Making lifestyle changes, such as eating a healthy diet, maintaining a healthy weight, and engaging in regular physical activity, can help reduce the risk of developing diabetes.
Disclaimer: medical
This web site is provided for educational and informational purposes only and does not constitute providing medical advice or professional services.
The information provided should not be used for diagnosing or treating a health problem or disease, and those seeking personal medical advice should consult with a licensed physician.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
Always seek the advice of your doctor or other qualified health provider regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website. If you think you may have a medical emergency, call 911 or go to the nearest emergency room immediately. No physician-patient relationship is created by this web site or its use. Neither BioMedLib nor its employees, nor any contributor to this web site, makes any representations, express or implied, with respect to the information provided herein or to its use.
Disclaimer: copyright
The Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the “DMCA”) provides recourse for copyright owners who believe that material appearing on the Internet infringes their rights under U.S. copyright law. If you believe in good faith that any content or material made available in connection with our website or services infringes your copyright, you (or your agent) may send us a notice requesting that the content or material be removed, or access to it blocked. Notices must be sent in writing by email (see 'Contact' section for email address) . The DMCA requires that your notice of alleged copyright infringement include the following information: (1) description of the copyrighted work that is the subject of claimed infringement; (2) description of the alleged infringing content and information sufficient to permit us to locate the content; (3) contact information for you, including your address, telephone number and email address; (4) a statement by you that you have a good faith belief that the content in the manner complained of is not authorized by the copyright owner, or its agent, or by the operation of any law; (5) a statement by you, signed under penalty of perjury, that the information in the notification is accurate and that you have the authority to enforce the copyrights that are claimed to be infringed; and (6) a physical or electronic signature of the copyright owner or a person authorized to act on the copyright owner’s behalf. Failure to include all of the above information may result in the delay of the processing of your complaint.
['Ɣef']
['BioMedLib yesseqdac iselkimen imseddayen (algoritmes n ulmad n tnaεurt) i usnulfu n tuqqniwin n tuttriwin d tririyin.']
['Nebda s 35 imelyan n teẓrigin n tesnujjya tameddurt n PubMed/Medline. Rnu daɣen, isebtar n web n RefinedWeb.']