Mũrimũ wa Alzheimer nĩ mũrimũ ũthiĩte na mbere gũthũka wa tombo ũrĩa ũhutagia meciria mũno, ũgatũma mũndũ ariganĩrwo nĩ maũndũ, na ũgarũrũke mĩtugo-inĩ.
Kĩrĩa gĩtũmaga mũndũ arware mũrimũ wa Alzheimer gĩtiũĩ wega, no kwĩgeragĩrio atĩ ũrehagwo nĩ maũndũ ma kĩĩmerera, maũndũ marĩa matũrigicĩirie, na ũrĩa tũikaraga.
Mamwe ma maũndũ marĩa mangĩtũma mũndũ anyitwo nĩ mũrimũ wa Alzheimer nĩ:
1. Ũndũ wa kĩĩmerera: Tũhengereta tũmwe nĩ tũmenyekete atĩ nĩ tũingĩhaga mũrimũ wa Alzheimer, na makĩria tũhengereta tũrĩa tũrutaga wĩra wa gũthondeka proteini wĩtagwo apolipoprotein E (APOE).
2. Ũkũrũ: Ũgwati wa kũgĩa na mũrimũ wa Alzheimer wongererekaga o ũrĩa mũndũ arakũra, na andũ aingĩ makoragwo naguo thutha wa mĩaka 65.
3. Andũ a famĩlĩ arĩa marĩ na mũrimũ ũcio: Angĩkorũo thĩinĩ wa famĩlĩ yanyu nĩ kũrĩ mũndũ ũrĩ na mũrimũ wa Alzheimer, no ũkorũo ũrĩ ũgwati-inĩ mũnene wa kũgĩa naguo.
4. Kũgũthwo nĩ mũtwe: Mũndũ angĩkorũo aagũthĩtwo mũtwe, makĩria ũndũ ũngĩmũtiga atekwĩigua, no agĩe na ũgwati mũnene wa kũrũara mũrimũ wa Alzheimer.
5. Maũndũ marĩa mangĩtũma mũndũ anyitwo nĩ mũrimũ wa ngoro: Maũndũ ta kũhaica kwa thakame, cholesterol, na cukari no matũme mũndũ anyitwo nĩ mũrimũ wa Alzheimer.
6. Maũndũ megiĩ mĩikarĩre: Gũikara mũno, kwaga kũrĩa irio iria ciagĩrĩire, na kwaga kũhũthĩra meciria wega no kuongerere ũgwati wa kũrũara mũrimũ wa Alzheimer.
7. Kũimba: Kũimba kaingĩ thĩinĩ wa tombo no gũtũme mũndũ agĩe na mũrimũ wa Alzheimer.
8. Gũtukanio kwa mahĩndĩ na indo ingĩ iria ithũkagia mwĩrĩ: Kwaga ũigananĩru gatagatĩ-inĩ ka mahĩndĩ gũthondeka indo njũru na ũhoti wa mwĩrĩ wa gũcieheria no gũtũme mũndũ agĩe na mũrimũ wa Alzheimer.
9. Tũcunjĩ twa thakame twĩtagwo amyloid plaques na tũcunjĩ tũngĩ twĩtagwo neurofibrillary tangles: Gũcokanĩrĩra kwa tũcunjĩ twa thakame twĩtagwo amyloid plaques na tũcunjĩ tũngĩ twĩtagwo neurofibrillary tangles thĩinĩ wa tombo nĩ kĩmenyithia kĩa mũrimũ wa Alzheimer, no itemi rĩrĩa makoragwo narĩo harĩ gũkũria mũrimũ ũcio rĩtimenyekete wega.
Nĩ wega kũririkana atĩ mũrimũ wa Alzheimer's ũtwaranaga na maũndũ maingĩ na kwahoteka nĩ maũndũ maya mothe.
Ũtuĩria nĩ ũrathiĩ na mbere nĩguo ataũkĩrũo wega nĩ ũrĩa mũndũ aiguaga na athondeke njĩra cia kũmũhonia.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Ũkaana: thibitarĩ']
['Website ĩno ĩkoragwo ĩrĩ ya kũrutana na kũheana ũhoro tu na ti ya kũheana ũtaaro wa ũrigitani kana ũtungata wa kĩĩmwĩrĩ.']
['Ũhoro ũrĩa ũrĩ thĩinĩ wa broshua ĩyo ndwagĩrĩirũo kũhũthĩrũo gũthima kana kũrigita mũrimũ mũna, na arĩa marenda ũtaaro wa ũrigitani magĩrĩirũo gũcaria ũteithio wa ndagĩtarĩ.']
['No wone atĩ netiwaki ya neuron ĩrĩa ĩheanaga macokio ma ciũria icio, ndĩkoragwo na ũkinyanĩru mũno ũhoro-inĩ wĩgiĩ namba. Kwa ngerekano, mũigana wa andũ arĩa magwatĩtio mũrimũ mũna.']
['Hingo ciothe caria ũtaaro wa ndagĩtarĩ kana mũndũ ũngĩ wagĩrĩire ũgima-inĩ waku wa mwĩrĩ igũrũ rĩgiĩ mũrimũ. Ndũkaanahũthie ũtaaro wa ndagĩtarĩ kana ũcererũo kũũcaria nĩ ũndũ wa ũndũ ũthomete thĩinĩ wa website ĩno. Ũngĩkorũo ũrona ta wacemania na ũndũ mũhiũ, hũra thimũ 911 kana ũthiĩ thibitarĩ ya hakuhĩ na harĩa ũrĩ. Gũtirĩ ũrata wa ndagĩtarĩ na mũrwaru wonekaga nĩ ũndũ wa website ĩno kana kũhũthĩrũo kwayo. BioMedLib kana aruti ayo a wĩra, kana mũndũ ũngĩ wothe ũrutĩte wĩra thĩinĩ wa website ĩno, ndarĩ na ũira, wa ĩmwe kwa ĩmwe kana wa ĩmwe kwa ĩmwe, wĩgiĩ ũhoro ũrĩa ũheanĩtwo ho kana ũrĩa ũhũthĩrĩtwo.']
['Ũregani: wĩyathi wa kwandĩka']
['Watho wa Digital Millennium Copyright Act wa 1998, 17 U.S.C. § 512 (the DMCA) ũheanaga ũhoti wa ene a ihoto cia wandĩki arĩa metĩkĩtie atĩ ũhoro ũrĩa ũroneka intanetiinĩ nĩ ũtharĩtie ihooto ciao kũringana na watho wa U.S. wa ihoto cia wandĩki.']
['Ũngĩkorũo na wĩtĩkio atĩ ũhoro kana kĩndũ kĩna gĩtahingũrĩtwo ũhoro-inĩ wĩgiĩ website kana ũtungata witũ nĩ gĩgũthũkia ihooto ciaku, wee (kana mũndũ ũrĩa ũgũgũthondekera) no ũtũtũmĩre notithi ũkĩũria ũhoro kana kĩndũ kĩu kĩeherio, kana ũrigĩrĩrio ndũgacihũthĩre.']
["Marũa macio magĩrĩire gũtũmwo na njĩra ya kwandĩkwo na e-mail (rora ũhoro wa andirethi gĩcunjĩinĩ kĩa 'Maũndũ ma kwaranĩria')."]
['DMCA ĩbataraga atĩ notithi yaku ya kuuna ihooto cia wandĩki ĩkorwo na ũhoro ũyũ: (1) ũtaarĩria wa wĩra ũrĩa ũrĩ na ihooto cia wandĩki ũrĩa ũrarumwo; (2) ũtaarĩria wa ũhoro ũrĩa ũrarumwo na ũhoro mũiganu wa gũtũhotithia kũmenya kũrĩa ũhoro ũcio ũrĩ; (3) ũhoro waku wa kwaranĩria, hamwe na andirethi, namba ya thimũ na andirethi ya e-mail; (4) ndũmĩrĩri yaku atĩ wĩ na wĩtĩkio mwega atĩ ũhoro ũcio ũramenererio ndũrĩ na rũtha rwa mwene wa watho, kana mũrũgamĩrĩri, kana rwa watho o wothe; ']
['(5) nĩ mwandĩkanĩire, na nĩ mũkũheo mũkaana wa kũheenania, atĩ ũhoro ũrĩa ũrĩ kĩmenyithiainĩ kĩu nĩ wa ma na atĩ mũrĩ na ũhoti wa kũhingia ihooto iria mũreganĩte nacio;']
['na (6) kĩrore kĩa mwene kĩhoto kana kĩa mũndũ wĩtĩkĩritio gwĩtongoria handũ ha mwene kĩhoto.']
['Kwaga kwandĩka ũhoro ũcio wothe no gũtũme gũtangĩka gwaku kũhĩtũke.']
['Ũhoro wa Kwaranĩria']
['Tũma ndũmĩrĩri ya kũbucia kũgerera thimũ kana thimũ ya mohoro.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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['BioMedLib ĩhũthagĩra kombiuta cia gwĩthuurĩra (njĩra cia gũthoma macini) gũthondeka ciũria na macokio.']
['Twambĩrĩirie na mabuku milioni 35 megiĩ ũgima wa mwĩrĩ marĩa makoragwo thĩinĩ wa PubMed/Medline. Ningĩ, tũhũthagĩra ciarĩro cia RefinedWeb.']
