De pathofysiologie vaan longkanker verwies nao de veranderinge in de normale fysiologische percesse en mechanismes die veurkoume bij de oontwikkeling en progressie vaan longkanker.
Longkanker is 'n complexe krenkde die ontsteit door de ónbeheerste greuj en deiling van abnormale celle in de longen.
Dees celle kinne tumore vörme en ziech verspreie nao aander deile vaan 't lief, wat tot versjèllende symptome en complicaties leidt.
De pathofysiologie vaan longkanker behels versjèllende factore, boe-oonder genetische mutaties, milieufactoren en leefstijlkeuzes.
Genetische mutaties kinne in 't DNA vaan longcelle veurkoume, wat leid tot oonbeheerde celgroei en -deiling.
Dees mutaties kinne geërf of verworve weure en kinne weure veroorzaakt door blootstèlling aon kankerverwekkende stoffe, wie tabaksrouk, radon, asbest en lochvervuiling.
Longkanker kin weure ingedeild in twie hoofsoorte: kleincellige longkanker (SCLC) en neet-kleincellige longkanker (NSCLC).
De pathofysiologie vaan dees soorte longkanker kinne versjèlle, umtot ze versjillende genetische mutaties höbbe en versjèlle in reactie op behandeling.
De pathofysiologie vaan longkanker behels ouch de interactie tösse kankercelle en 't umligkend weefsel, inclusief 't immuunsysteem.
Kankercelle kinne 't immuunsysteem ontvluchte, zoetot ze kinne greuje en zich ónbeheersbaar kinne verspreie.
Daoneve kin de tumormikro-omgeving de tumorgroei en metastase bevordere door 'n ondersteunende umgeving te geve veur kankercelle.
De pathofysiologie vaan longkanker is 'n complex en dynamisch perces, en oonderzeukers wèrke continu um de oonderligkende mechanismes beter te begriepe um effectievere behandelinge te oontwikkele en de oetkomste vaan patiënte te verbetere.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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