En ang'o matimore sama ng'ato nigi tuwo mar urticaria?
Urticaria, ma kinde mang'eny iluongo ni hives, en tuwo mar dend ng'ato ma nyiso ka ng'ato ong'ol, ka ng'ato ong'ol, kendo ka ng'ato ong'ol.
Tuwo mar urticaria oriwo golo histamine kod gik mamoko ma miyo ng'ato bedo gi tuwo mar inflammatory mediators kowuok e mast cells, ma gin ng'injo mag geng'o tuoche mayudore e dend ng'ato.
Sama ng'ato nigi tuwo mar urticaria, histamine kod gik mamoko ma miyo ng'ato bedo gi tuwo mar urticaria, wuok nikech ng'ato nigi tuwo mar urticaria, kaka chiemo, yedhe, kata kute.
Kinde mang'eny, ok ong'ere gimomiyo ng'ato bedo gi tuwo mar urticaria, kata kamano, iparo ni en nikech tuwo mar autoimmune.
E chal ma kamano, dend ng'ato loso antibodies ma kedo gi IgE receptor ma nigi affinity mamalo (FcεRI) e ng'injo mag mast, to mano miyo ng'injo mag ng'injo mag mast tiyo kendo golo histamine kod gik mamoko.
E tuwo mar urticaria ma tekregi ng'eny koda ma tekregi ng'eny, histamine kod gik mamoko ma miyo ng'ato bedo gi tuwo mar urticaria miyo ng'ato bedo gi tuwo mar itching, redness, kod swelling.
Kinde mang'eny, thieth oriwo tiyo gi yedhe ma geng'o Histamine mondo ogeng' gik ma Histamine timo kendo dwok chien tuoche.
E kinde ma tuwono ng'eny ahinya, inyalo ti gi yedhe mamoko kaka corticosteroids kata omalizumab e geng'o tuwono.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Brzoza Z, Grzeszczak W, Rogala B, Trautsolt W, Moczulski D: Possible contribution of chemokine receptor CCR2 and CCR5 polymorphisms in the pathogenesis of chronic spontaneous autoreactive urticaria. Allergol Immunopathol (Madr). , 42 (4): 302-6.
Sweeney TM, Dexter WW: Cholinergic urticaria in a jogger: ruling out exercise-induced anaphylaxis. Phys Sportsmed. 2003, 31 (6): 32-6.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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