Bãag ning sẽn boond tɩ alzayimeerã yaa bãag sẽn wat ne yĩn-gãong bãas sẽn namsd nebã, tɩ b yamã sãamdẽ, tɩ b yamã boogdẽ, tɩ b manesmã toeemdẽ.
B pa mi bãagã sẽn wat ne bũmb ningã takɩ ye.
Bõn-kãens kẽer tõe n wa kɩtame tɩ bãagã paam neda:
1. Zamaan rog-n-mikã: B bãnga rog-n-mik kẽer sẽn paasd bãagã sẽn tõe n wa ne wã, sẽn yɩɩd fãa rog-n-mik ning sẽn yaa apolipoprotéin E wã.
2. Yʋʋmã: Bãagã tõe n wa paama ned yʋʋm sẽn vẽsg yĩnga.
3. Zak pʋgẽ bãaga: Zak pʋgẽ ned sã n zoe n bẽe bãagã, tõe n kɩtame tɩ bãagã paam-a n paase.
4. Zĩig ning ned sẽn pogl a zugã: Ned sã n zoe n pogla a zugã, sẽn yɩɩd fãa sã n pogl-a n pa mi a sẽn maandg ye, tõe n kɩtame t'a bẽed bãag ning b sẽn boond tɩ alzayimeerã n paasdẽ.
5. Sũur bãase: Bãas wala zɩɩm sẽn tar pãng wʋsgo, zɩɩm ning sẽn be yĩngẽ wã n yɩɩd sẽn segdã, la diabɛt tõe n kɩtame tɩ bãag ning b sẽn boond tɩ alzayimeerã paam neda.
6. Vɩɩmã manesem: Ned sã n pa vɩ n tʋmdẽ, a sã n pa rɩt sõma, la a sã n pa tar bũmb wʋsg sẽn kõt a yamã pãnga, tõe n kɩtame t'a bẽed bã-kãngã n paasdẽ.
7. Zũnzũy bãase: Zũnzũy bãas sẽn kaoosd wʋsg tõe n kɩtame tɩ ned bẽed bãag ning b sẽn boond tɩ alzayimeerã.
8. Sũ-sãams sẽn tɩ loe ne pemsmã: Yĩngã sã n pa tõe n yiisd bõn-wẽns nins sẽn wat ne bãasã tɩ zemsdẽ, tõe n wa kɩtame tɩ bãagã paam-a.
9. Sũyã bãas sẽn tar bãas la b sẽn boond tɩ plaques amyloïdes ne neurofibrilles: Sũyã bãas sẽn tar bãas la b sẽn boond tɩ plaques amyloïdes sẽn tigimd taab to-to wã yaa bãnd sẽn wilgd tɩ ned bẽeda Alzheimɛɛr bãaga.
Yaa tɩlae tɩ d bãng tɩ bãagã sẽn wat ne bũmb ninsã yaa wʋsgo, la tɩ tõe tɩ yaa yɛl a woto buud n naag taaba.
B ket n maanda vaeesg sẽn na yɩl n bãng bãagã sẽn wat ne yɛl ninsã sõma, la b bao tɩbsg sẽn na n sõng-a.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Bãngr-gomde: logtoeemdã']
['Yaa zãmsg la kibay kũun yĩng bal la b yiisd sit kãngã, la pa logtoeemdã wɛɛngẽ sagls bɩ tʋʋm-sõng maaneg yĩng ye.']
['B pa segd n tũnug ne kɩbay nins b sẽn kõ wã n bao bãag bɩ zu-loɛɛg tɩɩm ye. Sẽn dat-b tɩbsg b toor segd n tɩ yãa logtor sẽn tar sor n tõe n tɩp-ba.']
['D tẽeg tɩ b sẽn maand to-to n leokd sogsgã to-to wã pa zems ye.']
['Y sã n wa karemd bũmb sẽn be sɩtã pʋgẽ, bɩ y ra tol n yĩm tɩ y segd n tɩ gesa logtor ye. Y sã n tagsdẽ tɩ y tara yĩn-wɩsgr yell bɩ y bool 911 wall y kẽng logtor yirã.']
['Bas-m-yam: sɛbã zãab wɛɛngẽ']
['Digital Millennium Copyright Act sẽn yaa yʋʋmd 1998 soabã, 17 U.S.C. § 512 (DMCA) kõta sor tɩ ned sã n tẽed tɩ bũmb sẽn be ẽntɛrnetã pʋgẽ kɩɩsda a sẽn tar sor n tõe n maan bũmb ninsã, a tõe n tɩ kos n paam n lebse.']
['Yãmb sã n tẽed ne pʋ-peelem tɩ bũmb sẽn be tõnd sɩt wɛɛbã pʋgẽ bɩ tõnd tʋʋm-noyã pʋsẽ n sãamd yãmb dʋrwa wã, yãmb (bɩ y tʋʋm-tʋmdã) tõe n toola tõnd koɛɛg n kos tɩ d yiis bũmbã wall d gɩdg tɩ y ra paam n kẽ ye.']
['B segd n tʋma koees ne ẽtɛrnetã (Ges-y ẽtɛrnetã adɛrs sẽn be babg ning sẽn yet tɩ "Tõnd sõsg zĩigã").']
['DMCA wã baoodame tɩ yãmb sẽn na n togs ned tɩ b maan-a-la bũmb sẽn kɩɩsd a sẽn tar sor n tõe n maan bũmb ningã, bɩ y wilg-a bũmb nins sẽn pʋgdã: 1) bũmb ning sẽn kɩt tɩ b maan-a bũmb ningã, 2) bũmb ning sẽn kɩt tɩ b maan-a bũmb ningã, la y wilg-d bũmb ning sẽn kɩt tɩ d tõe n bãng a sẽn be zĩig ninga. 3) y sẽn tõe n paam ned n gom ne-a to-to, n paas y adɛrsã, telefõnnã nimero, la y e-mailã. 4) y sã n yeel tɩ y kɩsa sɩd tɩ bũmb ning sẽn kɩt tɩ y maan bũmb ningã pa ned ning sẽn tar sor n tõe n maan bũmbã, bɩ a tʋm-tʋmdã, bɩ laloa wã sẽn kõ sor tɩ y maan ye.']
['5) Y sã n wa rat n wilg tɩ y pa tar sor n na n kɩɩs ned a to, bɩ y gʋls sebr n wilg tɩ y sẽn togsã yaa sɩda, la tɩ y tara sor n na n wilg tɩ nedã sẽn maan bũmb ning n kɩɩs yãmb dʋrwa rãmbã yaa sɩda.']
['La (6) sɛb nins sẽn tar-b sor n na n yiis sɛbã, bɩ ned sẽn tar sor n na n tʋm sɛbã yiisg yĩngã.']
['Y sã n pa gʋls kibay nins sẽn be yĩngrã, tõe n kɩtame tɩ y yẽgengã kaoos n pa sa ye.']
['Sõsg ne neda']
['Y sã n tar sokr bɩ y sẽn dat n bãnge, bɩ y gʋls-d lɛtr n tool-do.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
Disclaimer: medical
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Disclaimer: copyright
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['Sẽn zems']
['BioMedLib tũnugda ne ordinatɛɛr dãmb sẽn maand b toor n maand sogsg la b leokd taaba.']
['D sɩngda ne sɛb milyõ 35 sẽn tik Biiblã zug n yiisd tɩbsg wɛɛngẽ.']
