Kodi n'chiyani chimene chimayambitsa matenda a Alzheimer's?
Matenda a Alzheimer's ndi matenda owononga ubongo amene makamaka amakhudza ubongo, ndipo amachititsa kuti munthu asakumbukire bwino zinthu, kuzindikira bwino zinthu, ndi kusintha khalidwe lake.
Sitikudziŵa bwinobwino chimene chimayambitsa matenda a Alzheimer's, koma anthu amakhulupirira kuti matendawa amayamba chifukwa cha chibadwa, malo okhala, ndi moyo wa munthu.
Zina mwa zinthu zimene zingachititse kuti munthu akhale ndi matenda a Alzheimer's ndi izi:
1. Chibadwa: Anthu apeza majini ena amene amachititsa kuti munthu akhale ndi matenda a Alzheimer's, makamaka majini a apolipoprotein E (APOE).
2. Zaka: Chiopsezo cha matenda a Alzheimer's chimawonjezeka ndi ukalamba, ndipo anthu ambiri amapezeka ndi matendawa atakwanitsa zaka 65.
3. Mbiri ya m'banja: Kukhala ndi mbiri ya m'banja ya matenda a Alzheimer's kungawonjezere chiopsezo cha kukhala ndi matendawa.
4. Kuvulala m'mutu: Kuvulala m'mutu m'mbuyomu, makamaka kuvulala kumene kumachititsa kuti munthu ataya chikumbumtima, kungawonjezere chiopsezo cha matenda a Alzheimer.
5. Zochita zoopsa za matenda a mtima ndi mitsempha: Matenda monga kuthamanga kwa magazi, cholesterol yapamwamba, ndi matenda a shuga angawonjezere chiopsezo cha matenda a Alzheimer's.
6. Mkhalidwe wa moyo: Mkhalidwe wosayenda bwino, kudya bwino, ndi kusakhala ndi zinthu zolimbikitsa maganizo kungawonjezere chiopsezo cha matenda a Alzheimer's.
7. Kutupa: Kutupa kosatha kwa ubongo kungathandize kuti munthu akhale ndi matenda a Alzheimer's.
8. Kupanikizika kwa oxidative: Kusagwirizana pakati pa kupanga kwa ma free radicals ndi kuthekera kwa thupi kuwachotsa poizoni kungathandize kuti munthu akhale ndi matenda a Alzheimer's.
9. Amyloid plaques ndi neurofibrillary tangles: Kuchuluka kwa amyloid plaques ndi neurofibrillary tangles mu ubongo ndi chizindikiro cha matenda a Alzheimer's, koma mbali imene ali nayo m'kukula kwa vutoli siimamvetsetsedwa bwino.
Nkofunika kudziŵa kuti chifukwa cha matenda a Alzheimer's n'chovuta kumvetsa ndipo mwachionekere chimaphatikizapo zinthu zimenezi pamodzi.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
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Chonde titumizireni imelo ndi funso / lingaliro lililonse.
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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