Matenda a urticaria, amene anthu ambiri amawatcha kuti chikuku, ndi matenda a khungu amene amaoneka ngati makoswe ofiira, okwera, ndi odetsa khungu.
Pathophysiology ya urticaria imaphatikizapo kumasulidwa kwa histamine ndi othandizira ena otupa kuchokera ku maselo a mast, omwe ndi maselo a chitetezo cha m'thupi omwe amapezeka pakhungu ndi minofu ina.
Mu urticaria yoopsa, kumasulidwa kwa histamine ndi othandizira ena kumayambitsidwa ndi chifuwa cha allergen inayake, monga chakudya, mankhwala, kapena kulumidwa ndi tizilombo.
Zimenezi zimachititsa kuti maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo a maselo.
M'matenda a urticaria aakulu, kaŵirikaŵiri chifukwa chake sichidziŵika, koma akuganiza kuti chimakhudzana ndi matenda a autoimmune.
Pankhaniyi, thupi limapanga ma autoantibodies omwe amalimbana ndi high-affinity IgE receptor (FcεRI) pa maselo a mast, zomwe zimapangitsa kuti azigwira ntchito komanso kumasula histamine ndi othandizira ena.
M'matenda onse a urticaria aakulu ndi aakulu, kutuluka kwa histamine ndi ma mediator ena kumabweretsa zizindikiro za kuyabwa, kufiira, ndi kutupa.
Kaŵirikaŵiri chithandizocho chimaphatikizapo kugwiritsa ntchito mankhwala a antihistamines kuti aletse zotsatira za histamine ndi kuchepetsa zizindikiro.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Chonde dziŵani kuti ma neural net amene amapanga mayankho a mafunso, ndi osayenerera makamaka pankhani ya manambala. Mwachitsanzo, chiŵerengero cha anthu odwala matenda enaake.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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