E fisiopatologia di un atake di kurason, tambe konosi komo infarto di miokard, ta enserá e interupshon di fluho di sanger na un parti di e músculo di kurason, ku ta kondusí na morto di e sèlnan di kurason.
Esaki ta sosodé normalmente pa motibu di un obstrukshon di un arteria koronario dor di un trombus, ku hopi biaha ta resultado di aterosklerosis, un kondishon kaminda plaka ta akumulá den e arteria.
E plak ta konsistí di kolesteròl, supstansia di vet, produktonan di sushi di sèl, kalsio i fibrina.
Ora un plak kibra, e por kousa un koagulo di sanger ku ta blòkia e arteria i ta stroba sanger riku na oksígeno di yega na e múskulo di kurason.
E falta di oksígeno ta kousa morto di e sèlnan di e múskulo di kurason, loke ta kondusí na un atake di kurason.
E grado di daño ta dependé di e área ku e arteria blokia ta suministrá i e tempu entre e atake i tratamentu.
Sintomanan di un atake di kurason por inklui doló òf inkómodo na pechu, falta di rosea, náusea, sintimentu di kabes bira lihé i doló den brasa, garganta, mandibula òf lomba.
Tratamentu pa un atake di kurason generalmente ta enserá restorá e fluho di sanger na e músculo di kurason mas lihé posibel, sea pa medio di remedi òf prosedura manera angioplastia i stent òf operashon di bypass di e arteria koronario.
Ta importante pa nota ku e fisiopatologia di un atake di kurason ta kompleho i ta enbolbí faktornan múltiple, inkluyendo faktornan genétiko, estilo di bida i medio ambiente.
Faktornan di riesgo pa atake di kurason ta preshon haltu, kolesteròl haltu, humamentu, diabétis, obesidat, falta di aktividat físiko i un historia familiar di malesa di kurason.
Manehá e faktornan di riesgo aki por yuda redusí e chèns di haña un atake di kurason.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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