Kay cáncer de pulmón pathophysiologíaqa, imaymana factoresmanta kachkan, kaykunataq: mutaciones genéticas, factores ambientales, imayna kawsayninchikmanta ima.
Genetic mutations nisqaqa ADN nisqapi rikhurin, chaytaq mana controlasqa wiñayta, t'aqakuyta ima apakun.
Kay mutacionesqa, herenciajina chayri mana herenciajina kanman, chaytaq cigarro qʼosñi, asbesto, aire chʼichichasqa ima, chaykunata apaykachasqanrayku rikhurin.
Kay cáncer de pulmón iskay laya: cáncer de pulmón de células pequeñas (SCLC) y cáncer de pulmón de células no pequeñas (NSCLC).
Kay cáncer de pulmón nisqakunaqa, mana kikinchu kanku, imaraykuchus mana kikinchu kanku mutaciones genéticas nisqakuna, mana kikinchu kanku tratamiento nisqaman kutichisqanku.
Kay cáncer de pulmón nisqa patología fisiológica nisqapiqa, células cancerosaswan, chay ukhu tejidowan, sistema inmunológicowan ima, khuska llamkʼanku.
Kay unquyqa, mana unqusqasta unquymanta jarkʼayta atinchu.
Kay pathophysiología del cáncer de pulmón nisqaqa juk proceso complejo, dinámico, jinamanta investigadoresqa, astawan allinta entiendenankupaq llamk'achkanku, kay tratamientos aswan eficaces kananpaq, jinamanta unqusqasta aswan allinta qhawanankupaq.
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Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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