Indwara ya Alzheimer ni indwara ifatira mu tugingo ngengabuzima igenda irunyuka, ikaba ifata canecane ubwonko, igatuma umuntu yibagira, ubwenge bwiwe bugaca bugabanuka, inyifato yiwe na yo igahinduka.
Ibitera iyo ndwara ntibitomoye neza, mugabo bivugwa ko biterwa n'ibintu bitandukanye, nk'utugingo ngengakamere, ibidukikije be n'uburyo umuntu abayeho.
Bimwe mu bintu bishobora gutuma umuntu arwara indwara ya Alzheimer ni ibi:
1. Ingaruka zo mu tugingo ngengakamere: Hari utugingo ngengakamere tumwetumwe twabonetse dutuma umuntu arushiriza kugeramirwa n'indwara ya Alzheimer, canecane utugingo ngengakamere twitwa apolipoprotéine E (APOE).
2. Imyaka: Ingeramizi yo kurwara indwara ya Alzheimer irongerekana uko umuntu agenda arakura, kubera ko abantu benshi bayirwara bamaze gushikana imyaka 65.
3. Ingorane zo mu muryango: Kugira umuntu arwaye indwara ya Alzheimer mu muryango birashobora gutuma arushiriza kugeramirwa n'iyo ndwara.
4. Gukomereka ku mutwe: Umuntu yarigeze gukomereka ku mutwe, canecane agakomereka ku buryo ata ubwenge, arashobora kurushiriza kugeramirwa n'indwara ya Alzheimer.
5. Ingeramizi z'umutima: Ingorane nk'ivumbuka ry'umurindi w'amaraso, ibinure vyinshi mu maraso be n'igisukari birashobora gutuma umuntu arushiriza kurwara indwara yo mu bwonko yitwa Alzheimer.
6. Ingene umuntu abayeho: Kuba umuntu yigira gusa, gufungura nabi no kubura ivyo akoresha mu bwenge vyoshobora gutuma arushiriza kurwara ya ndwara yonona ubwonko yitwa Alzheimer.
7. Indwara y'ibisebe: Indwara y'ibisebe idakira mu bwonko yoshobora gutuma umuntu arwara indwara yo mu mutwe yitwa Alzheimer.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Urupapuro rwemeza ko ata ruhusha:']
["Uru rubuga rw'ivy'ubuvuzi ruratangwa ku bw'inyigisho no ku bw'ukumenyesha amakuru gusa, ntirutanga impanuro mu vy'ubuvuzi canke ngo rube urwego rw'abahinga mu vy'ubuvuzi."]
["Itegeko ryo mu 1998 ryerekeye uburenganzira umuntu afise mu vyo akoresha mu guhanahana amakuru (Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the DMCA) ritanga uburenganzira ku bantu bafise uburenganzira bwo guhanahana amakuru babona ko ibintu vyerekanwa kuri Internet bihonyanga uburenganzira bwabo hisunzwe amategeko ya Leta Zunze Ubumwe za Amerika ajanye n'uburenganzira umuntu afise. "]
['Mu gihe wibaza ko hari ibintu canke ibikoresho vyabonetse ku rubuga rwacu canke ku mbuga zacu bibangamiye uburenganzira bwawe, wewe (canke uwukugenzura) urashobora kuturungikira ubutumwa busaba ko ivyo bintu canke ivyo bikoresho bikurwaho canke ko bitagisubira kuboneka. ']
['Amatangazo ategerezwa kurungikwa mu nyandiko hakoreshejwe ubutumwa bwa "email" (raba ahavuga ngo "Kwitaba" kugira ngo umenye aderese ya "email").']
["Itegeko rya DMCA risaba ko umenyesha ko hari uwugomba guhonyanga uburenganzira bwawe mu gutanga amakuru akurikira: (1) idondorwa ry'igikorwa gikingiwe n'amategeko kiriko kiragirizwa guhonyanga uburenganzira bwawe; (2) idondorwa ry'ibiri mu vyo bavuga ko bihonyanga uburenganzira bwawe be n'amakuru ahagije atuma dushobora kumenya aho biri; (3) amakuru y'ukuntu twokwifatanya nawe, harimwo aderese yawe, inomero za telefone n'ikete ryawe ryo kurungikiranira ubutumwa kuri internet; (4) ivyemeza ko wemera udakeka ko ivyo bintu biri mu buryo uriko uridodombera bitahawe uburenganzira n'uwabitunganije, canke uwubiserukira, canke n'itegeko iryo ari ryo ryose; "]
["(5) urwandiko rwanditswe n'uwatanze iyo notifica-tion, rwemeza ko amakuru ari muri iyo notifica-tion ari ay'ukuri kandi ko afise ububasha bwo gukurikirana ivy'uwo muntu avuga ko yahonyanze uburenganzira bwiwe; "]
['Turakwinginze uturungikire ubutumwa kuri email ufise ikibazo / iciyumviro.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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