Ingorane zijanye n'indwara ya kanseri y'uruti rw'umugongo ni zo zituma iyo ndwara ikwiragira.
Kanseri y'ibere, izwi kandi ko ari kanseri y'ibere, ni ubwoko bwa kanseri itangurira mu kibuno canke mu muhogo, ivyo bikaba ari ibihimba vy'amara manini.
Mu bisanzwe bitangura ari uduhimba twitwa polypes, mu nyuma tukaba tumwe mu bwoko bwa kanseri.
Ingorane zitera kanseri y'uruti rw'umugongo ziterwa n'ibintu bitari bike, harimwo amahinduka aba mu tugingo ngengakamere, ugucibwamwo be n'ibintu vyo mu bidukikije.
Hari aho haba amahinduka mu tugingo ngengakamere tw'utugingo ngengabuzima two mu nda, bigatuma dukura kandi tugaca twigwiza cane.
Iyo kanseri y'uruti rw'umugongo imaze gushika, irashobora gutera intambwe zitandukanye, uhereye ku ntango, aho usanga ikiri mu rwagasha, gushika ku ntambwe yo mu nyuma, aho usanga yarakwiragiye mu bindi bihimba vy'umubiri.
Utwo tugingo ngengabuzima turashobora gutera ibihimba be n'utugingo ngengabuzima biri hafi y'aho tuba, tukongera tukandukira mu maraso no mu maraso tukagera kure nko mu gitigu canke mu mahaha.
Mu bisanzwe, mu kuvura kanseri y'ibere harimwo kubagwa, imiti ya kanseri be n'imiti ifasha kuvura indwara mu buryo bwa rayons, ivyo bikaba bivana n'aho iyo kanseri igeze be n'aho iri.
Gutahura indwara hakiri kare no kuyivura ni ntahara kugira ngo ivyiza biboneke, kuko kanseri y'uruti rw'umugongo akenshi ishobora kuvurwa iyo ifashwe hakiri kare.
Kwisuzumisha kenshi, nk'ugupima mu nda, birashobora gufasha kumenya kanseri yo mu nda hakiri kare, igihe ishobora kuvurwa neza.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Urupapuro rwemeza ko ata ruhusha:']
["Uru rubuga rw'ivy'ubuvuzi ruratangwa ku bw'inyigisho no ku bw'ukumenyesha amakuru gusa, ntirutanga impanuro mu vy'ubuvuzi canke ngo rube urwego rw'abahinga mu vy'ubuvuzi."]
["Itegeko ryo mu 1998 ryerekeye uburenganzira umuntu afise mu vyo akoresha mu guhanahana amakuru (Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the DMCA) ritanga uburenganzira ku bantu bafise uburenganzira bwo guhanahana amakuru babona ko ibintu vyerekanwa kuri Internet bihonyanga uburenganzira bwabo hisunzwe amategeko ya Leta Zunze Ubumwe za Amerika ajanye n'uburenganzira umuntu afise. "]
['Mu gihe wibaza ko hari ibintu canke ibikoresho vyabonetse ku rubuga rwacu canke ku mbuga zacu bibangamiye uburenganzira bwawe, wewe (canke uwukugenzura) urashobora kuturungikira ubutumwa busaba ko ivyo bintu canke ivyo bikoresho bikurwaho canke ko bitagisubira kuboneka. ']
['Amatangazo ategerezwa kurungikwa mu nyandiko hakoreshejwe ubutumwa bwa "email" (raba ahavuga ngo "Kwitaba" kugira ngo umenye aderese ya "email").']
["Itegeko rya DMCA risaba ko umenyesha ko hari uwugomba guhonyanga uburenganzira bwawe mu gutanga amakuru akurikira: (1) idondorwa ry'igikorwa gikingiwe n'amategeko kiriko kiragirizwa guhonyanga uburenganzira bwawe; (2) idondorwa ry'ibiri mu vyo bavuga ko bihonyanga uburenganzira bwawe be n'amakuru ahagije atuma dushobora kumenya aho biri; (3) amakuru y'ukuntu twokwifatanya nawe, harimwo aderese yawe, inomero za telefone n'ikete ryawe ryo kurungikiranira ubutumwa kuri internet; (4) ivyemeza ko wemera udakeka ko ivyo bintu biri mu buryo uriko uridodombera bitahawe uburenganzira n'uwabitunganije, canke uwubiserukira, canke n'itegeko iryo ari ryo ryose; "]
["(5) urwandiko rwanditswe n'uwatanze iyo notifica-tion, rwemeza ko amakuru ari muri iyo notifica-tion ari ay'ukuri kandi ko afise ububasha bwo gukurikirana ivy'uwo muntu avuga ko yahonyanze uburenganzira bwiwe; "]
['Turakwinginze uturungikire ubutumwa kuri email ufise ikibazo / iciyumviro.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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