Pathophysiology n diabetes eqqal teɣare n immikan n physiological tan war n oɣed wi eway nen efes n diabetes mellitus
Diabetes mellitus eqqal taqqimt n ihishkan n metabolic s ilanen iddikud n iddikud n blood sugar (glucose) ijjan s ihishkan n insulin secretion, insulin action meɣ issin
Pathophysiology n diabetes eha anmizlay n genetic, ahinzazaɣ d dumu n tamudre wi ahanen edag daɣ efes n aṭṭan
Daɣ type 1 diabetes, pathophysiology eha anmiyiz n autoimmune n beta cells tan win insulin daɣ pancreas, eway id iban n insulin
Awen eway id war addobat n akayad n iddikud n glucose daɣ achareɣa, eway id hyperglycemia (high blood sugar)
Daɣ type 2 diabetes, pathophysiology eqqal a ojjaran tolas ahan insulin resistance d insulin secretion
Asukni n insulin itaj alwaq wa id izirwan n taɣissa war n arhan s insulin s almaɣna n as war addoben ad axdaman glucose i assahat
Awen eway pancréas i ad ijji insulin ajjotnen i ad ijji anmizlay n anmizlay mucham daɣ azzaman, pancréas war addobat ad ijji anmizlay n asistan, eway id afanaz n ijji n insulin
Gestacional diabetes, wa id izjaran daɣ azzaman n tadist, eqqal fal asuj n hormonal tan wi id eway nen insulin resistance
Placenta t-id-issuj hormon tan addoben ad-assedmalan alxidmat n insulin, s awen id-issuknin tiw-at n iddikud n glucose daɣ idammen
Pathophysiology n diabetes eha deɣ efes n complications chund asuj n blood vessels, nerves d organ tan addoben ad awin s muchakil tan n assexat chund heart disease, stroke, kidney disease d vision loss
Afahəm ən patofisiologie ən diabete eqal aśohen fal igi ən adabara tan wi ahanen ad tikarsiwen ən asiwad ye aśahat wen aśohatnen.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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