What is pathophysiology of Prostate cancer?

Awa eqqal patophysiology n cancer n prostate?

Pathophysiology n prostate cancer eqqal fal sistem tan d tikarsiwen n aru id eway nen efes d efes n aṭṭan

Cancer wan prostate eqqal tumur n tumast tazzarat daɣ izirwan n prostate gland, eqqal organ madroynen n dumu n walnut eqqal nen ddaw n bladder daɣ irgazan

Prostate gland t-id-issuj aman n seminal, wa isaknen d isaknen sperm

Albab n cancer n prostate war eqqal a issnen hullan mucham illanat titbaqqa ajjotnen issanen as issiwaden aharoj n a hin ewad a

Awen ahan tiw-at, attarex n tawset, tawset d imutiyan n etari iyyad

Cancer wan prostate eqqal a oharan daɣ irgazan wi ogaran daɣ awatay tolas ijjit n haratan ahanen daɣ irgazan wi ogaran daɣ awatay wan 65

Daɣ asiwad, irgazan ilan attarex n tawset n cancer tan prostate ahanen daɣ aharoj hullan chund irgazan win afro-americain d irgazan n Caribbean

Pathophysiology n prostate cancer eha tiw-at war n ila alxakum d tazunt n izirwan daɣ prostate gland

Awen addobat ad ijru fal amutti n etari s eway id asuj n ihuk n ich-ikil n efes n etari meɣ asuj n etari tan n tumors

Imuttiyan win addoben ad awin id efes war n oɣed n izirwan, eway id efes n tumor

Alwaq wa id tumor ittimɣur addobat ad ikcem iɣasan d izirwan n tamazduq chund bladder, rectum d lymph nodes n tamazduq

Daɣ ihandagan iyyad, izirwan n cancer addoben ad arunen daɣ tumor wa ezzaran tolas ad azun s ichrutan iyyad n taɣissa s achareɣa n achareɣa n achareɣa meɣ lymphatic system, tamatikwayt s isim net metastasis

Alwaq wa id cancer izjaran, addobat ad eqqil a ojjaran ad t awid

Cancer tan prostate addoben deɣ ad aqqilan fal hormonal factors hullan androgen hormone testosterone

Testosterone addobat ad issiwid efes n izirwan n cancer n prostate tolas ajjotnen n asafar n cancer n prostate ahan ittus n afanaz n hormon wen meɣ afanaz n alxidmat net

Daɣ akayad, patofisiology n prostate cancer eha tiw-at war n ila alxakum d tazunt n izirwan daɣ prostate gland, addobat nen ad eqqil fal genetic, hormonal d haratan n ahinzazaɣ

Afahəm ən təbiɗawt tan tabarat ən aśahat eqal aśohat fal igi ən adabara tan wi ahanen ad assəmutiy ən iba ən imuḍan ən cancer tan prostate.

['Isalan wi illanen']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Zobniw CM, Causebrook A, Fong MK: Clinical use of abiraterone in the treatment of metastatic castration-resistant prostate cancer. Res Rep Urol. 2014, 6 (): 97-105.

Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.

Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.

Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.

Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.

Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.

Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.

['Asukni: medikal']

['Web site wen eqqal i isalan n teɣare d isalan ɣas tolas war eqqel asiwil n asafar meɣ alxidmat tan n musnat']

['Isalan wi ikfan war addoben ad axdaman i asifham meɣ asafar n aśahat meɣ aśahat tolas wi aramanen asiwil n asafar n awadim ilzam ad aruman asiwil n asafar n awadim']

['Dǎɣ asikin, neural net wa iknanen tirmawen ən assestan, eqal aśohen aśohat dǎɣ alwaq wa id itawan dǎɣ maɗin ən maɗinan. sund, iddikud ən adinat wi atiwasananen as aśohat atiwasanan.']

['Harkuk arha majrad n doctor inek meɣ n awadim n assexat n tazmert n addinat fal aśahat n aśahat. War t-id-t-igdel majrad n aśahat n awadim meɣ azzaman daɣ aśahat fal as harat awa iktabem fal site wen. Afal as ad t-illa aśahat n aśahat n aśahat, asiwel 911 meɣ aru s ehan n aśahat n aśahat wa oharnen daɣ azzaman. War t-illa majrad n aśahat n awadim d aśahat fal site wen meɣ axadam net. BioMedLib meɣ inaxdiman net meɣ awadim wa iknanen fal site wen, war t-illa majrad, iknan meɣ iknan, fal isalan wi dawanen meɣ axadam net']

['Asukni: copyright']

['Digital Millennium Copyright Act wan 1998, 17 U.S.C. § 512 (DMCA) eha almaɣna i kel alxaq n alxaq n alxaq wa allaɣen as isuj win illanen fal Internet ikna alxaq nasan daɣ achareɣa n alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq wan alxaq']

['A fal tazamt s tiditt as isalan meɣ isuj win illanen daɣ asuj n asuj net meɣ alxidmaten iknan alxaq inek n alxaq, addobat ad aɣ-d-tazneḍ (meɣ agent inek) isalan meɣ isuj win ad n-ikkes meɣ ad n-isikkid alxidmat']

['Isalan ilzam ad ittusiknan s eɣaf s email (ikayad edag "Contact" i tansa n email)']

['DMCA t-assirɣas as isalan inek n asiwad n alxaq n alxoriya eha isalan win: (1) almaɣna n alxidmat eha alxaq n alxoriya eqqal harat n asiwad eha alxaq n alxoriya; (2) almaɣna n almaɣna eha alxoriya d isalan ajjotnen ijjan aɣ ad n afhaman almaɣna; (3) isalan n alɣisab inek, ahan tansa, amaṭṭaf n tilifun d tansa n email inek; (4) almaɣna inek n as ilan tumast n alxaq n alxoriya as almaɣna wa s eha alxaq eha alxaq eha alxaq war eha alxaq n alxaq eha alxoriya meɣ amatikki net meɣ s alxidmat n achareɣa fuk']

['(5) almaɣna n awen, iktab daɣ azruf n alkad n tiditt, as isalan win isalan aqqalan tiditt tolas ilan tanaya n as artay alxaqqan win alxakum wi s allaɣen as ikna']

['Dǎɣ awa, əmik ən əmik ən alxaq meɣ awadim ən alxaq ən alxaq meɣ awadim atiwafan ye igi ən alxaq ən alxaq.']

['A fal war t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id-t-id']

['Assahat']

['Ilzam aɣ e-mail s asteqsi meɣ asukni']

What is pathophysiology of prostate cancer?

The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.

Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.

The prostate gland produces seminal fluid, which nourishes and transports sperm.

The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.

These include age, family history, race, and certain genetic mutations.

Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.

Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.

The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.

This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.

These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.

As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.

In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.

Once the cancer has spread, it can be more difficult to treat.

Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.

Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.

In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.

Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.

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