Urticaria, s isim net net n addinat eqqal aqqal aqqal n achareɣa n taɣissa s tijja n ittus, tiw-at, tiw-at tazeggaɣt (wheals) fal taɣissa
Pathophysiology n urticaria eha asiflid n histamine d inflammatory mediators tan iyyad daɣ mast cells, aqqalan immune cells illanen daɣ taɣissa d tissus tan iyyad
Daɣ urticaria ta tazzarat, asuj n histamine d mediator tan iyyad eqqal s allergic reaction i allergen n ichikilan chund učči, asafar meɣ aśuhu n ihishkan
Awen eway mast cells i ad aɣin, issufuɣ histamine d mediator tan iyyad wi isawadan i isuj n idammen ad aqqilan a hinan, s eway id efes n wheals
Daɣ urticaria ta tazzayat, s alwaq iyyan war itwassan s eɣaf net mucham eqqal as ijja fal sistem n autoimmune
Daɣ almaɣna wen, taɣissa t-id-t-id-issuknan autoantibodies ijjan IgE receptor (FcεRI) fal mast cells, s-ikkaten efes nasan d-assufeɣ n histamine d mediators iyyad
Daɣ urticaria tan ajjotnen d win aru, asuj n histamine d mediator tan iyyad eway id samalan n ittus, redness d swelling
Asafar eqqal alwaq iyyan iji n antihistamines i ad eqqil n alxidmat tan histamine d afanaz n samalan
Daɣ ihishkan ajjotnen, isuj n iyyad chund corticosteroids meɣ omalizumab addobat nat axadam i akayad n talɣa
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Brzoza Z, Grzeszczak W, Rogala B, Trautsolt W, Moczulski D: Possible contribution of chemokine receptor CCR2 and CCR5 polymorphisms in the pathogenesis of chronic spontaneous autoreactive urticaria. Allergol Immunopathol (Madr). , 42 (4): 302-6.
Sweeney TM, Dexter WW: Cholinergic urticaria in a jogger: ruling out exercise-induced anaphylaxis. Phys Sportsmed. 2003, 31 (6): 32-6.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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