Nye ci lomboloka o patofisiologia yuveyi wo cancer yovapuvi?
Oku kuliha eci ci koka uveyi wo kanser yovapuvi ci lomboloka apongoloko kovopange kuenda kovisimilo viekalo vi pita vokuenda kuoku kula kuenda oku amamako kuvei wo kanser yovapuvi.
O cancer yovapuvi uveyi umue wa tatama okuti u tunda koku kula kuenda koku litepa kuovimatamata ka via sungulukile vovapuvi.
Kuenje, vi fetika oku livokiya calua.
Oku kala ciwa kuolombuto viukãi kuenda oku kala ciwa kuolombuto vialume.
Oku pongoloka kuovimatamata viuyali ku pondola oku pita vo DNA yovimatamata viapuvi, okuti ku tuala koku livokiya kuenda koku litepa kuovimatamata.
Ocitangi caco ci pondola oku tunda kovimatamata viuyali ndeci: Owisi wakaya, owuya u tunda volonjo, kuenda ofela ya liña.
O cancer yovapuvi citava okuti yi tepiwa volonepa vivali vinene: small cell lung cancer (SCLC) kuenda non-small cell lung cancer (NSCLC).
Oku kuliha eci ci koka uveyi wo kanser yovapuvi ci kongelavo oku litokeka kuovimatamata viocilenda kuenda ovimatamata vikuavo, oku kongelamo o sistema imunológico.
Olomakina vi sakula uvei wo kanser, vi pondola oku tateka asokoluilo oku liteyuila kovovei aco.
Handi vali, ocitumãlo citito colomuma ci pondola oku vetiya oku kula kuolomuma kuenda oku li sanduila kovimatamata vikuavo poku eca ocitumãlo cimue ciwa kovimatamata viocilenda.
Uloño woku sakula uveyi wo cancer yovapuvi u kuete esilivilo lialua kuenda olonoño viamamako oku likolisilako oku kuata elomboloko liwa oco va sange onjila yoku sakula uveyi waco kuenda oku mioñolola ekalo liava va vela.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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['(5) ukanda umue wa sinaliwa love okuti u lekisa okuti esapulo liaco liocili kuenda o kuete omoko yoku teyuila omoko yove yoku soneha.']
['Kuenda (6) ondimbukiso yocikuata ale yo komputador yomunu ukuete omoko yoku linga upange waco ale yomunu umue o kuete omoko yoku linga upange waco vonduko yomunu ukuete omoko yaco. ']
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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