Uvei wo urticaria, okuti wa kulihiwa okuti, uvei umue wocipa okuti u limbukiwila koku molẽha kuovilundu vi kusuka, vi kuete evalo, kuenda vi kula.
Ocitukiko ci koka oku tunda ku histamina kuenda ovina vikuavo vi koka oku kuatiwa kuvei u tukuiwa hati, mastocytes.
Voku kuata uveyi waco, o histamina kuenda ovina vikuavo vi kuatisa oku kuata uveyi waco, vi tunda kovina vimue ndeci, okulia, ovihemba ale oku lumaniwa lovipuka.
Kuenje, eci ci vetiya olosinga viosonde oku kuata uvei umue u tukuiwa hati, histamina.
Voku kala lombuto ka ya kulile, esunga liaco ka lia kulihiwile, pole ca siata oku simawa okuti ca tiamẽla kovimatamata viuyaki.
Vepuluvi eli, etimba li panga ovihemba vi tukuiwa hati autoantibodies okuti vi kuata unene woku tambula o IgE (FcεRI) vovitukiko vi tukuiwa hati mast cells, okuti vi vetiya oku kuata ongusu kuenda oku tundisa o histamine kuenda ovina vikuavo.
Ci kale okuti uveyi waco u kasi oku amamako ale sio, eci ku tunda o histamina kuenda ovina vikuavo vi koka uveyi waco, ku molẽha evalo, evalo li kusuka kuenda evalo.
Ovihemba viaco vi kuatisa oku tutuiya ovihemba vi tukuiwa hati, histamina oco vi tepulule evalo.
Olonjanja vimue, ovihemba vikuavo ndeci o corticosteroide ale omalizumabe vi pondola oku kapiwako oco vi kuatise oku lava uvei waco.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Brzoza Z, Grzeszczak W, Rogala B, Trautsolt W, Moczulski D: Possible contribution of chemokine receptor CCR2 and CCR5 polymorphisms in the pathogenesis of chronic spontaneous autoreactive urticaria. Allergol Immunopathol (Madr). , 42 (4): 302-6.
Sweeney TM, Dexter WW: Cholinergic urticaria in a jogger: ruling out exercise-induced anaphylaxis. Phys Sportsmed. 2003, 31 (6): 32-6.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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