Jàngoroy kansar bu mag bi dafay tekki ay jëfekaay ak ay doxalin yuy tax ba kansar bu mag bi di law.
Kanser bu mag bu ndaw, ñu koy wax it kansër bu mag bu ndaw, ab xeetu kansër la buy tàmbalee ci biir bu mag bi walla bu déjj bi, di ay cér ci biir bu mag bi.
Dafa di dañuy tàmbalee ci polip bu man a màgg ba nekk jàngoroy kansaar ci ay at.
Jàngoroy colon dafay ànd ak ay sabab yu bari, niki ay gene yuy soppi jikko, ay feebar ak ay sabab yuy jur ag dund.
Ay coppite yu aju ci gene mën nañ a am ci ADN bu selil colon, di indi yokkute ak séddale selil buñ mën a saytu.
Yëngu-yëngu yooyu mën nañu leen a donn walla ñu am leen, te mën nañu laal gene yu wuute yi bokk ci yokkute, séddale ak yeesal selil yi.
Feebar bu am ay feebar yuy tax ba yaram di metti ci biir buy jur feebar bu am ay feebar yuy tax ba yaram di metti ci biir, loolu itam mën na yokk sa gaawu jàngoroy colon.
Xëpp bu yàgg mën na jur ay kemikal yuy yokk màggug yaram ak séddale ko, loolu mën na jur jàngoroy kansaar.
Yëngu-yënguy wërlaay yi, niki lekk, dund ak luñ laal ay kemikal, mën nañu indi itam jàngoroy colon.
Diis bu am ay yàpp yu xonq yu bari ak yu ñu defare, ci misaal, ñu jàpp ne dafay yokk jàngoroy kansar ci biir buy.
Yeneen sabab yi, niki màggat, saxar, ak ñàkk jëf, mën nañu yokk it jafe-jafe bi.
Bu jàngoroy colon bi amee, mën na dem ba ci ay wàll, dale ci bu njëkk bi, bi mu yem ci colon bi, dem ba ci bu gën a yàgg bi, bi mu law ci yeneen wàll yi ci yaram wi.
Selil cancer yi mën nañ dugg ci yaram yi nekk ci wet gi, mën nañ itam law ci sistem bu lymphatic bi walla ci deret ji ba ci béréb yu sore, niki ci biir yaram wala ci pooj yi.
Jëfandikoo ci faj jàngoroy colon dafay ànd ak ay jéego, ay kemoterap ak ay radioterap, ci lu aju ci màggug jàngoro ji ak fu mu nekk.
Xam ak faj ci njëlbéen ga am na solo lool ngir gën a wér, ndax jàngoroy colon mën nañ koo faj bu ñu ko xamee ci njëlbéen ga.
Seetlu bu safaanu, niki colonoscopie, mën na la dimbali nga xam kansar bu njëkk bi, bu ko gën a mën a faj.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Waxtaan: wér-gu-yaram']
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['Seetal ne jàmbaar gii di sàkk tontu yi ci laaj yi, dafa ñàkk solo lool ci lu jëm ci limu limu nit ñi, niki limub ñi ñu jàngal ab jàngoro.']
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['Séddo: sañ-sañu jëfandikoo']
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['Sudee gëm nga ci lu wér ne lenn ci li nekk ci sunu dal bi walla ci sunuy liggéey dafa moy say sañ-sañ, yaw (mbaa sa jawriñ) mën nga nu yónnee ab bataaxal di laaj ñu dindi li nekk ci dal bi walla ci liggéey bi, walla ñu téye sa jàll ci moom. ']
['Bind nañu ay yëgle ci mbind, ci ab bataaxal (Xoolal "Contact" ngir xam màkkaanu bataaxal bi).']
['DMCA dafa digle ne sa bataaxal bu jëm ci jàddug sañ-sañ bu ñu sos war na ëmb li ci topp: (1) xët wu jëm ci liggéey bi ñu sos ne jàdd nañ ko; (2) xët wu jëm ci li ñu sos ne jàdd nañ ko ak ay xibaar yu doy ngir may nu nu nu man a gis li mu ëmb; (3) ay xibaar yu jëm ci yaw, boole ci sa màkkaanu dal, sa limu telefóni ak sa màkkaanu imeel; (4) ab kàddu bu jóge ci yaw bu lay xamal ne am nga yaakaar bu wér ne li nga sos ci anam wi ñu la ko sosu, moom boroom sañ-sañ bi, walla ki ko dénk, walla benn yoon, nanguwu ko; ']
['(5) ab bataaxal bu ñu la jox, nga dëggal ci sa loxo ne li nga bind dëgg la te am nga sañ-sañu sàmm sañ-sañu jëfandikoo sañ-sañu bind bi ñu la sosal ne yàqu na;']
['ak (6) benn màndarga buy firndeel walla buy wone ay màndarga yuy wone ne moom la sañ-sañu jëfandikoo walla mu ngi koy jëfandikoo ci turu moom. ']
['Suñ la ci dugalul lépp lu ñu wax ci kaw, mën na tax ba say tawat di gaaw a jàppale.']
['Waxtaan']
['Yónneel nu ab imeel bu la laaj walla nga am ay xalaat.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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['Nu tàmbali ci 35 milyoŋi téerey xam-xamu jëmm ci PubMed/Medline. Te itam ci xëti web yi ci RefinedWeb.']
