Patofisiologi kanker usus rayeuk mengacu bak mekanisme dan proses yang mendasari yang mengarah bak perkembangan dan perkembangan kanker usus rayeuk.
Kanker usus besar, juga dituri sebagoe kanker kolorektal, adalah jenis kanker yang dimulai di usus besar atau rektum, yang merupakan bagian dari usus besar.
Biasa jih dimulai seubagai pertumbuhan yang dikhen polip, yang jeut berkembang jeut kanker seiring wate.
Patofisiologi kanker usus rayeuk melibatkan padum-padum boh faktor, termasuk mutasi genetik, inflamasi, dan faktor lingkungan.
Mutasi genetik jeut terjadi bak DNA sel usus besar, yang jeut keu pertumbuhan sel yang hana terkontrol dan pembelahan.
Mutasi nyo jeut diwariskan atau di teumeng, dan jeut mempengaruhi berbagai gen yang terlibat dalam pertumbuhan sel, pembelahan, dan perbaikan.
Peradangan bak usus rayeuk, nyang jeut ji peuget le kondisi lage peunyaket radang usus, jeut ji tingkatkan resiko kanker usus rayeuk.
Peradangan kronis jeut geu peuget pelepasan bahan kimia yang geu peuget pertumbuhan sel dan pembelahan, yang jeut geu peuget perkembangan kanker
Faktor lingkungan, lage diet, gaya udep, dan paparan bahan kimia tertentu, jeut chit berkontribusi bak perkembangan kanker usus besar.
Makanan yang le asoe mirah dan olahan, miseu jih, ka ji kaitkan deungen peningkatan resiko kanker usus besar.
Faktor laen, lage obesitas, merokok, dan kurang aktifitas fisik, jeut cit ji tingkatkan resiko.
Watee kanker usus rayeuk ka berkembang, jeut berkembang melalui padum-padum boh tahap, dari kanker tahap awai yang terbatas bak usus rayeuk hingga tahap leubeh lanjut di pat kanker ka menyebar ke bagian laen tuboh.
Sel kanker jeut menyerang jaringan ngen organ yang na di toe, dan jeut menyebar melalui sistem limfatik atau aliran darah keu teumpat yang jioh, lage hati atau paru-paru.
Perawatan untuk kanker usus besar biasa jih melibatkan kombinasi operasi, kemoterapi, dan terapi radiasi, tergantung bak tahap dan lokasi kanker.
Deteksi dan perawatan dini adalah kunci untuk peuget hasil yang leubeh get, kareuna kanker usus rayek seureng jeut ji peu ubat meunyoe ji teume bak tahap awai.
Pemeriksaan rutin, lage kolonoskopi, jeut membantu mendeteksi kanker usus besar bak tahap awai jih, watee nyan paleng jeut diobati.
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Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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