Urtikaria, biasa jih dituri seubagoe urtikaria, adalah kondisi kulet yang ditanda deungen deuh jih gatal, meulangue, bintik mirah (wheels) bak kulet.
Patofisiologi urtikaria melibatkan pelepasan histamin dan mediator inflamasi laen dari sel mast, yang merupakan sel kekebalan yang na bak kulet dan jaringan laen.
Dalam urtikaria akut, pelepasan histamin dan mediator laen dipeuget oleh reaksi alergi terhadap alergen tertentu, lage makanan, obat, atau sengatan serangga.
Hal nyo menyebabkan sel mast degranulate, peulheuh histamin dan mediator laen yang peuget pembuluh darah jeut bocor, yang meuakibat keu terbentuk roda.
Bak urtikaria kronis, penyebab jih seureng hana dituri, tapi di pike terkait deungen mekanisme autoimun.
Lam kasus nyo, tuboh geu produksi autoantibodi nyang target reseptor IgE afinitas tinggi (FcεRI) bak sel mast, geu peu aktifkan dan geu peulheuh histamin ngon mediator laen jih.
Wahlgren CF: Pathophysiology of itching in urticaria and atopic dermatitis. Allergy. 1992, 47 (2 Pt 1): 65-75.
Raap U, Liekenbröcker T, Wieczorek D, Kapp A, Wedi B: [New therapeutic strategies for the different subtypes of urticaria]. Hautarzt. 2004, 55 (4): 361-6.
[Recommendations for the diagnosis and treatment of urticaria in children]. Arch Argent Pediatr. 2021, 119 (2): S54-S66.
Brzoza Z, Grzeszczak W, Rogala B, Trautsolt W, Moczulski D: Possible contribution of chemokine receptor CCR2 and CCR5 polymorphisms in the pathogenesis of chronic spontaneous autoreactive urticaria. Allergol Immunopathol (Madr). , 42 (4): 302-6.
Sweeney TM, Dexter WW: Cholinergic urticaria in a jogger: ruling out exercise-induced anaphylaxis. Phys Sportsmed. 2003, 31 (6): 32-6.
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What is pathophysiology of urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by the appearance of itchy, raised, red welts (wheals) on the skin.
The pathophysiology of urticaria involves the release of histamine and other inflammatory mediators from mast cells, which are immune cells found in the skin and other tissues.
In acute urticaria, the release of histamine and other mediators is triggered by an allergic reaction to a specific allergen, such as food, medication, or insect sting.
This causes the mast cells to degranulate, releasing histamine and other mediators that cause blood vessels to become leaky, leading to the formation of wheals.
In chronic urticaria, the cause is often unknown, but it is thought to be related to an autoimmune mechanism.
In this case, the body produces autoantibodies that target the high-affinity IgE receptor (FcεRI) on mast cells, leading to their activation and the release of histamine and other mediators.
In both acute and chronic urticaria, the release of histamine and other mediators leads to the characteristic symptoms of itching, redness, and swelling.
Treatment typically involves the use of antihistamines to block the effects of histamine and reduce symptoms.
In severe cases, other medications such as corticosteroids or omalizumab may be used to control the condition.
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