Pathophysiology of colon cancer kyerɛ kwan a yare no fa so nya nkɔso.
Kokoram yɛ yarewa bi a ɛtumi fi ase wɔ obi nsono mu anaa ne berɛmo mu.
Mpɛn pii no, efi ase sɛ ade a wɔto a wɔfrɛ no polyp, a ebetumi adan kokoram bere rekɔ so.
Pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Yareɛ a ɛde yare ba tumi kɔso wɔ DNA mu wɔ nkwammoaa a ɛwɔ nsono mu, na ɛde nkwammoaa no nyin ne ne kyekyɛ ba a wɔnhwɛ so.
Wobetumi anya saa nsakrae yi afi awo mu anaa wɔanya no, na ebetumi aka awosu ahorow a ɛka nkwammoaa nyin, wɔn kyekyɛ, ne wɔn nsiesie ho.
Sɛ obi nya nsaneyadeɛ wɔ ne nsono mu a, ɛbɛtumi ama wanya nsaneyadeɛ mu kokoram.
Sɛ yareɛ no kɔ so tena obi mu a, etumi ma ɔtwe nnuru a ɛma nkwammoaa no nyin na ɛpaepae, na etumi ma onyaa kokoram.
Nneɛma bi te sɛ aduan a wodi, sɛnea wɔbɔ wɔn bra, ne nnuru bi a wɔde di dwuma nso betumi ama obi anya nsono mu kokoram.
Sɛ nhwɛso no, wohwɛ nnuan a mogyanam ne nam a wɔadi ho dwuma wom a, etumi ma obi nya kokoram.
Nneɛma foforo te sɛ kɛseyɛ mmoroso, sigaretnom, ne apɔwmuteɛteɛ a wɔyɛ no daa betumi ama asiane no ayɛ kɛse.
Sɛ kokoram ba obi nsono mu a, ɛtumi kɔ mpuntuo pii mu, firi kokoram a ɛwɔ ne mpuntuo mu de kɔ mpuntuo a ɛrekɔ n'anim mu a ɛtrɛ akɔ nipadua no fa bi.
Kokoram nkwammoaa no betumi ahyɛ nipadua no akwaa a ɛbɛn ho no so, na ebetumi atrɛw afa mogya mu anaa mogya ntini mu akɔ mmeae a ɛwɔ akyirikyiri te sɛ berɛbo anaa ahrawa mu.
Ayaresa a wɔde yɛ kokoram no taa hwehwɛ sɛ wɔyɛ obi oprehyɛn, ma no nnuru a ano yɛ den (chemotherapy), na wɔsan ma no nnuru a ano yɛ den (radiation therapy), a egyina kokoram no fa ne baabi a ɛwɔ so.
Sɛ wohu ne ho yareɛ no ntɛm a, ɛboa paa ma wo yareɛ no kɔ yiye.
Nhwehwɛmu a wɔyɛ daa te sɛ colonoscopy betumi aboa ma wɔahu kokoram a ɛwɔ nsono mu ntɛm bere a ɛyɛ mmerɛw sɛ wɔbɛsa no.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
['Abɔdin: ayaresa']
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['Nkitahodi']
['Yɛsrɛ wo, fa nsɛmmisa anaa nyansahyɛ biara fa e-mail so brɛ yɛn.']
What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
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