Kɔnɔboli, min bɛ wele tubabukan na eritoromisini, ye farila bana ye min bɛ sɔrɔ kuruw fɛ minnu bɛ mɔgɔ fari fan bɛɛ kalaya.
A sababu bɛ se ka kɛ fɛn caman ye, i n' a fɔ farilajidɛsɛ, banamisɛnniw, walima bana wɛrɛw.
Kɔnɔboli furakɛli bɛ bɔ bana juguya ni a sababu la.
A furakɛcogo dɔw filɛ:
1. antistaminiw: Nin fura ninnu bɛ ŋɛɲɛ ni sumaya nɔgɔya ka a sababu kɛ u bɛ farikolo ka baarakɛ cogo bali.
Banakisɛfagalan minnu bɛ se ka ta ka a sɔrɔ furakisɛ ma olu ye difenidaramini (Benadryl) walima loratadine (Claritin) ye, olu bɛ se ka bana nɔgɔmanw furakɛ.
Ni a ka jugu kosɛbɛ, dɔgɔtɔrɔ bɛ se ka dimimadafura farimanw di a ma.
Kɔnɔboli: Ni kɔnɔboli basigilen don, aw bɛ a lajɛ ka banakisɛfagalan ta.
Nin fura ninnu ka teli ka ta da la ani u bɛ se ka mɔgɔ dɛmɛ joona.
Furakisɛ minnu bɛ farikolo farilajidɛsɛ kɛlɛ: Ni sumaya jugumanba don, ni fura wɛrɛ ma se ka a furakɛ, aw bɛ faritanabana kɛlɛfura dɔw ta i n'a fɔ sikulɔsiporini (cyclosporine) walima metotɛrɛkisati (méthotrexate) walasa ka farilajidɛsɛ kɛlɛ ani ka sumaya kɛlɛ.
4. omalizumab: Nin fura in ye banakisɛfagalan pikirilama ye min bɛ se ka monoklonal antibody kɛlɛ min bɛ se ka immunoglobuline E (IgE) kɛlɛ ani ka a bali, poroteyini min bɛ faritanabanaw kɛlɛ.
A ka ca a la, a bɛ kɛ ka sumayabana jugumanba furakɛ min tɛ sɔn furakɛli suguya wɛrɛw ma.
5. Aw ye aw yɛrɛ tanga faritanabanaw ma: Ka faritanabanaw dɔn ani ka u yɛrɛ tanga, o bɛ se ka bana bali ka wuli.
Deleanu D, Nedelea I, Petricau C, Leru P, Dumitrascu D, Muntean A: Clinical impact of omalizumab in refractory chronic urticaria: One centre experience. Exp Ther Med. 2019, 18 (6): 5078-5081.
Grundmann SA, Kiefer S, Luger TA, Brehler R: Delayed pressure urticaria - dapsone heading for first-line therapy? J Dtsch Dermatol Ges. 2011, 9 (11): 908-12.
Staumont-Sallé D, Piette F, Delaporte E: [Etiological diagnosis and treatment of chronic urticaria]. Rev Med Interne. 2003, 24 (1): 34-44.
Akashi R, Ishiguro N, Shimizu S, Kawashima M: Clinical study of the relationship between Helicobacter pylori and chronic urticaria and prurigo chronica multiformis: effectiveness of eradication therapy for Helicobacter pylori. J Dermatol. 2011, 38 (8): 761-6.
Calogiuri G, Nettis E, Mandurino-Mirizzi A, Di Leo E, Macchia L, Foti C, Vacca A, Kounis NG: Omalizumab for the Treatment of Persistent Drug Induced Urticaria Elicited by Thienopyridines: A Case Report. Antiinflamm Antiallergy Agents Med Chem. 2020, 19 (3): 335-339.
['Kunnafoni nafama: kɛnɛya']
['Nin gafe in labɛnna kalan ni kunnafoni dɔrɔn de kama, a tɛ sɔrɔ dɔgɔtɔrɔ ka ladili wala kɛnɛyabaarakɛla ka baara kɛcogo la.']
["Kunnafoni minnu dira aw ma, aw man kan ka olu kɛ ka banaw furakɛ. Mɔgɔ minnu b'u yɛrɛ furakɛ, olu ka kan ka dɔgɔtɔrɔ dɔ ka dɛmɛ ɲini."]
["Aw ye aw janto nin na: ɛntɛrinɛti min bɛ jabi di ɲininkaliw jaabiliw kan, o dabali ka jugu kɛrɛnkɛrɛnnenya la n'a bɛ tali kɛ jatidenw de kan. Misali la, mɔgɔ hakɛ min ka bana kofɔra."]
['Aw bɛ ladilikan ɲini tuma bɛɛ aw ka dɔgɔtɔrɔ fɛ walima kɛnɛya baarakɛla dɔ wɛrɛ min bɛ se ka aw dɛmɛ ka aw ka bana furakɛ. Aw kana dɔgɔtɔrɔ ka ladilikan bila ka suma walima ka mɛn a ɲini na sabu aw ye fɛn dɔ kalan nin siti kan. Ni aw hakili la ko aw ka bana bɛ se ka juguya joona, aw ye 911 wele walima aw ka taa aw ka dɔgɔtɔrɔso la joona joona. Nin siti tɛ dɔkɔtɔrɔ ni banabagatɔ ka jɛɲɔgɔnya jira. BioMedLib ni a ka baaradenw, walima nin siti dɛmɛbaga si tɛ kuma si jira walima ka jira, nin kunnafoniw wala u labaarali kama.']
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['Ni i dara a la kô i ka site web ni a baara tchogow bè i ka lakananifènw la, i (walima i ka lasigiden) bè se ka i yèrè ka lakananifènw bila ka o site web ni a baara tchogow bila kènèkan.']
['Waajibi don kunnafoniw ci kɛtɔ ka kɛ sɛbɛn ye e-mail fɛ (e-mail de kan ka lajɛ sɛbɛn ɲɛ Kunnafoniw lajɛ yɔrɔ la).']
["DMCA bɛ a ɲini i ka kunnafoni sɛbɛn bɔlɔlɔw kan kojugukɛ sɛbɛn bɛ sɛbɛn min kɔnɔ, o ka kan ka nin kunnafoniw fara a kan: (1) sɛbɛnni kɛtɔ ka sɛbɛnni kɛ min bɛ sɛbɛnni kɛ ni a ma kɛ kojugukɛ sɛbɛn ye; (2) sɛbɛnni kɛtɔ ka fɛn kofɔlen in kofɔ ani kunnafoni minnu bɛ a to an bɛ se ka fɛn kofɔlen in sɔrɔ; (3) i ka ladɛrɛsi, i ka ladɛrɛsi, telefɔni nimɔrɔ ani i ka ladɛrɛsi; (4) i ka kumaɲɔgɔnya sɛbɛn ko i dalen b'a la ko i bɛ a dɔn ko i bɛ ka fɛn kofɔlen in kɛ ni sariya min ye, o ma di sɛbɛnnikɛbaga ma, a ka ladɛrɛsi tigi, walima sariya wɛrɛ; "]
['(5) a ka seereyasɛbɛ sɛbɛntiya, ko seereyasɛbɛ minɛnen bɛ kojugubakɛlaw kama, ko kunnafoni min bɛ o sɛbɛntiya kɔnɔ, ko tiɲɛ don ani ko i yamaruyalen don ka yamaruya di ka kɛwalew kɛ minnu bɛ tiɲɛni kɛ; ']
['ani (6) sɛbɛn dɔ ka boloci walima ɛkitɔrɔniki sɛbɛn sɛbɛn tigi fɛ walima mɔgɔ min yamaruyara ka baara kɛ sɛbɛn tigi tɔgɔla. ']
['Ni kunnafoni fɔlen ninnu bɛɛ ma fara ɲɔgɔn kan, o bɛ se ka kɛ sababu ye ka mɛn baara kɛli la i ka sɛbɛn kɔnɔ.']
['Ɲɔgɔnkunbɛn']
['Sɛbɛn ɲɛ Ɲiningaliw / ladiliw']
How to treat urticaria?
Urticaria, commonly known as hives, is a skin condition characterized by itchy, raised welts that can appear on any part of the body.
It can be caused by various factors, including allergic reactions, infections, or underlying medical conditions.
Treatment for urticaria depends on the severity of the condition and the underlying cause.
Here are some common treatment options:
1. Antihistamines: These medications help to reduce itching and swelling by blocking the effects of histamine, a chemical released by the body during an allergic reaction.
Over-the-counter antihistamines such as diphenhydramine (Benadryl) or loratadine (Claritin) can be effective in mild cases.
In more severe cases, a doctor may prescribe stronger antihistamines.
2. Corticosteroids: In cases where antihistamines are not effective, corticosteroids such as prednisone may be prescribed to reduce inflammation and suppress the immune system.
These medications are usually taken orally and can provide quick relief.
3. Immunosuppressants: In cases of chronic urticaria that do not respond to other treatments, immunosuppressant drugs such as cyclosporine or methotrexate may be prescribed to suppress the immune system and reduce inflammation.
4. Omalizumab: This medication is an injectable monoclonal antibody that targets and blocks immunoglobulin E (IgE), a protein involved in allergic reactions.
It is typically used for chronic urticaria that does not respond to other treatments.
5. Avoid triggers: Identifying and avoiding triggers that cause urticaria can help prevent outbreaks.
Common triggers include certain foods, medications, insect bites, and exposure to heat, cold, or sunlight.
6. Cold compress: Applying a cold compress to the affected area can help reduce itching and swelling.
7. Wet wraps: Wet wraps can be used to soothe itching and reduce inflammation.
A damp cloth is wrapped around the affected area, and then a dry cloth is placed over it to retain moisture.
8. Phototherapy: Exposure to ultraviolet light can help suppress the immune system and reduce inflammation in some cases of chronic urticaria.
It is important to consult a healthcare professional for proper diagnosis and treatment of urticaria.
They can help determine the underlying cause and recommend the most appropriate treatment plan.
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