Bushe ubulwele bwa kansa ya ku mukoshi bwaba shani?
Icipimo ca kansa ya mu mala cilosha ku fyo icilonda cicita pa kuti umuntu alwale kansa ya mu mala.
Kansa ya mu mala, iyo beta no kuti colorectal cancer, yaba musango wa kansa itendekela mu mala nelyo mu musana, utwaba mu mala ayakalamba.
Ilingi line utushiishi tulatendeka fye no kukula kabili kuti twaya tupanga kansa.
Icilenga kansa ya mu mala kuti yaba ne fintu ifingi pamo nga ukufulungana kwa nsandesande, ukufimba, ne fintu fimbi.
Ifyabipa ifingacitika ku nsandesande sha mu mala kuti fyalenga insandesande ukulakula no kulekana.
Ifi fintu kuti fyaba fya mu cisoolo nelyo fyacitika fye umuntu nga afyalwa, kabili kuti fyalenga insandesande ukulakula, ukulekana, no kuwamya ifilundwa fya nsandesande.
Ukufimba mu mala, ukwingalenga umuntu ukulwala kansa ya mala, kuti kwalenga no kuti alwale kansa ya mala.
Ukupola kwa mutatakuya kuti kwalenga insandesande ukulakula no kulekana, kabili kuti kwalenga umuntu ukulwala kansa.
Ifilenga umuntu ukukwata kansa ya ku mukoshi, fintu pamo nga ifya kulya alya, ifyo acita ilyo ali no bumi, e lyo no kuba ku miti imo imo.
Ku ca kumwenako, abantu abalya sana inama ishakashika ne shapangwa, balafwa ku bulwele bwa kansa.
Fimbi pamo nga ukufina, ukupeepa, no kukanabomba bwino kuti fyalenga ubulwele ukubipilako.
Nga ca kuti kansa ya mu mala yatendeka, kuti yapita mu fiputulwa ifingi, ukutendekela pa ciputulwa ca kubalilapo ica kansa iyaseeka fye mu mala ukufika ku ciputulwa ca ku ntanshi ica kansa iyaseeka mu filundwa fimbi ifya mubili.
Insandesande sha kansa kuti shaingila mu filundwa fya mubili ifyaba mupepi, kabili kuti shafika na ku filundwa fya mubili fimbi ifyaba ukutali pamo nga ilibu nelyo bapwapwa ukupitila mu mulopa.
Umuti wa kubomfya pa kundapa kansa ya mu mala utwalekanalekana, pamo nga ukulepula, ukubomfya imiti ya kucefyako amalwele ya kansa, no kubomfya umwela wa radiation, ukulingana no mushinku wa kansa na uko yabela.
Ukuishiba bwangu no kuundapa kwalicindama pa kuti ubulwele bwishipola, pantu ilingi line ubulwele bwa kansa bwa ku mukoshi kuti bwapola nga bwaishibikwa bwangu.
Ukuceeceeta lyonse, pamo nga ukuceeceeta mu mala, kuti kwafwa ukusanga kansa ya mu mala ilyo fye yatendeka, ilyo ilingile ukuundapwa.
Vidal-Vanaclocha F: The liver prometastatic reaction of cancer patients: implications for microenvironment-dependent colon cancer gene regulation. Cancer Microenviron. 2011, 4 (2): 163-80.
Yagi T, Kubota E, Koyama H, Tanaka T, Kataoka H, Imaeda K, Joh T: Glucagon promotes colon cancer cell growth via regulating AMPK and MAPK pathways. Oncotarget. 2018, 9 (12): 10650-10664.
Sharma SH, Thulasingam S, Nagarajan S: Terpenoids as anti-colon cancer agents - A comprehensive review on its mechanistic perspectives. Eur J Pharmacol. 2017, 795 (): 169-178.
Keshk WA, Zineldeen DH, Wasfy RE, El-Khadrawy OH: Fatty acid synthase/oxidized low-density lipoprotein as metabolic oncogenes linking obesity to colon cancer via NF-kappa B in Egyptians. Med Oncol. 2014, 31 (10): 192.
Dongfeng D, An C, Shujia P, Jikai Y, Tao Y, Rui D, Kai T, Yafeng C, Jianguo L, Xilin D: Explanation of colon cancer pathophysiology through analyzing the disrupted homeostasis of bile acids. Afr Health Sci. 2014, 14 (4): 925-8.
Tammali R, Ramana KV, Srivastava SK: Aldose reductase regulates TNF-alpha-induced PGE2 production in human colon cancer cells. Cancer Lett. 2007, 252 (2): 299-306.
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What is pathophysiology of colon cancer?
The pathophysiology of colon cancer refers to the underlying mechanisms and processes that lead to the development and progression of colon cancer.
Colon cancer, also known as colorectal cancer, is a type of cancer that begins in the colon or rectum, which are parts of the large intestine.
It typically starts as a growth called a polyp, which can develop into cancer over time.
The pathophysiology of colon cancer involves several factors, including genetic mutations, inflammation, and environmental factors.
Genetic mutations can occur in the DNA of colon cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can affect various genes involved in cell growth, division, and repair.
Inflammation in the colon, which can be caused by conditions such as inflammatory bowel disease, can also increase the risk of colon cancer.
Chronic inflammation can lead to the release of chemicals that promote cell growth and division, potentially leading to the development of cancer.
Environmental factors, such as diet, lifestyle, and exposure to certain chemicals, can also contribute to the development of colon cancer.
A diet high in red and processed meats, for example, has been linked to an increased risk of colon cancer.
Other factors, such as obesity, smoking, and lack of physical activity, can also increase the risk.
Once colon cancer develops, it can progress through several stages, from early-stage cancer that is confined to the colon to more advanced stages where the cancer has spread to other parts of the body.
The cancer cells can invade nearby tissues and organs, and may also spread through the lymphatic system or bloodstream to distant sites, such as the liver or lungs.
Treatment for colon cancer typically involves a combination of surgery, chemotherapy, and radiation therapy, depending on the stage and location of the cancer.
Early detection and treatment are key to improving outcomes, as colon cancer is often curable when caught in its early stages.
Regular screening, such as colonoscopy, can help detect colon cancer at its earliest stages, when it is most treatable.
Disclaimer: medical
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