Who gets Alzheimer?

Pwy sy'n cael alzheimer?

Mae clefyd Alzheimer yn anhwylder niwrodegeneradol raddol sy'n effeithio'n bennaf ar oedolion hŷn.

Mae'r risg o ddatblygu clefyd Alzheimer yn cynyddu gyda'r oedran, ac mae'n fwyaf cyffredin mewn pobl dros 65 oed.

Fodd bynnag, gall Alzheimer sy'n dechrau'n gynnar ddigwydd mewn pobl yn eu 30au, 40au, a 50au, er bod hyn yn llai cyffredin.

Mae geneteg hefyd yn chwarae rhan yn datblygiad clefyd Alzheimer.

Mae pobl sydd â hanes teuluol o'r clefyd yn wynebu mwy o berygl o'i ddatblygu eu hunain.

Yn ogystal, gall rhai mudiadau genetig, fel y gen APOE-e4, gynyddu'r tebygolrwydd o ddatblygu clefyd Alzheimer.

Mae ffactorau risg eraill ar gyfer clefyd Alzheimer yn cynnwys hanes o anafiadau pen, clefyd cardiofasgwlaidd, a ffactorau ffordd o fyw fel ysmygu, gordewdra, a diffyg ymarfer corff.

Mae'n bwysig nodi, er bod rhai ffactorau'n gallu cynyddu'r risg o ddatblygu clefyd Alzheimer, nad yw'n rhan arferol o heneiddio, ac ni fydd pawb sy'n heneiddio yn datblygu'r clefyd.

Cyfeiriadau

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.

Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.

Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.

Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.

Bakhtiari S, Moghadam NB, Ehsani M, Mortazavi H, Sabour S, Bakhshi M: Can Salivary Acetylcholinesterase be a Diagnostic Biomarker for Alzheimer? J Clin Diagn Res. 2017, 11 (1): ZC58-ZC60.

Gwaharddiad cyfrifoldeb: meddygol

Mae'r wefan hon yn cael ei ddarparu at ddibenion addysgol a gwybodaeth yn unig ac nid yw'n darparu cyngor meddygol neu wasanaethau proffesiynol.

Ni ddylid defnyddio'r wybodaeth a ddarperir i ddiagnosio neu drin broblem neu glefyd iechyd, a dylai'r rhai sy'n ceisio cyngor meddygol personol ymgynghori â meddyg trwyddedig.

Sylwch fod y rhwydwaith niwrol sy'n cynhyrchu atebion i'r cwestiynau, yn arbennig o anghywir pan ddaw i gynnwys rhifol. Er enghraifft, nifer y bobl sy'n cael diagnosis o glefyd penodol.

Ceisiwch gyngor eich meddyg neu ddarparwr iechyd cymwys arall bob amser ynghylch cyflwr meddygol. Peidiwch byth ag anwybyddu cyngor meddygol proffesiynol neu ohirio ei geisio oherwydd rhywbeth rydych chi wedi ei ddarllen ar y wefan hon. Os ydych chi'n meddwl y gallai fod gennych argyfwng meddygol, ffonwch 911 neu ewch i'r ystafell brys agosaf ar unwaith. Nid oes unrhyw berthynas meddyg-cleifion yn cael ei greu gan y wefan hon na'i ddefnydd. Nid yw BioMedLib na'i weithwyr, na unrhyw gyfrannwr i'r wefan hon, yn gwneud unrhyw gynrychiolaeth, yn glir neu'n awgrymol, mewn perthynas â'r wybodaeth a ddarperir yma na'i ddefnydd.

Gwrthod cyfrifoldeb: hawlfraint

Mae Deddf Hawlfraint y Mileniwm Digidol o 1998, 17 U.S.C. § 512 (y DMCA) yn darparu adnodd i berchnogion hawlfraint sy'n credu bod deunydd sy'n ymddangos ar y Rhyngrwyd yn torri eu hawliau o dan gyfraith hawlfraint yr Unol Daleithiau.

Os ydych chi'n credu mewn ffydd da bod unrhyw gynnwys neu ddeunydd a wnaed ar gael mewn cysylltiad â'n gwefan neu'n gwasanaethau yn torri eich hawlfraint, gallwch chi (neu'ch asiant) anfon hysbysiad atom yn gofyn i'r cynnwys neu'r ddeunydd gael ei ddileu, neu fod mynediad ato wedi'i rwystro.

Rhaid i rybuddion gael eu hanfon yn ysgrifenedig trwy e-bost (gweler adran "Cyflwyniad" am gyfeiriad e-bost).

Mae'r DMCA yn gofyn i'ch hysbysiad o dorri hawlfraint honedig gynnwys y wybodaeth ganlynol: (1) disgrifiad o'r gwaith hawlfraint sy'n destun y dorri hawlfraint honedig; (2) disgrifiad o'r cynnwys sy'n dorri hawlfraint honedig a gwybodaeth ddigonol i'n galluogi i ddod o hyd i'r cynnwys; (3) gwybodaeth gyswllt i chi, gan gynnwys eich cyfeiriad, rhif ffôn a chyfeiriad e-bost; (4) datganiad gan chi bod gennych gred ffyddlon nad yw'r cynnwys yn y ffordd sy'n cwyno amdano wedi'i awdurdodi gan berchennog yr hawlfraint, neu ei asiant, neu gan weithredu unrhyw gyfraith;

(5) datganiad gennych chi, wedi'i lofnodi o dan gosb llygredd, bod y wybodaeth yn y hysbysiad yn gywir ac bod gennych chi'r awdurdod i orfodi'r hawlfraint sy'n cael eu troseddu;

a (6) llofnod corfforol neu electronig o berchennog y hawlfraint neu berson wedi'i awdurdodi i weithredu ar ran perchennog y hawlfraint.

Gall methu â chynnwys yr holl wybodaeth uchod arwain at oedi wrth brosesu'ch cwyn.

Cysylltwch

Anfonwch e-bost i ni gyda unrhyw gwestiwn / awgrym.

Who gets alzheimer?

Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects older adults.

The risk of developing Alzheimer's disease increases with age, and it is most common in people over the age of 65.

However, early-onset Alzheimer's can occur in people in their 30s, 40s, and 50s, although this is less common.

Genetics also play a role in the development of Alzheimer's disease.

People with a family history of the disease are at a higher risk of developing it themselves.

Additionally, certain genetic mutations, such as the APOE-e4 gene, can increase the likelihood of developing Alzheimer's.

Other risk factors for Alzheimer's disease include a history of head injuries, cardiovascular disease, and lifestyle factors such as smoking, obesity, and lack of exercise.

It is important to note that while certain factors can increase the risk of developing Alzheimer's disease, it is not a normal part of aging, and not everyone who ages will develop the disease.

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