Alzheimer ƒe dɔlélea nye dɔléle si gblẽa nu le ahɔhɔ̃a ƒe dɔwɔwɔ ŋu si gblẽa nu le ahɔhɔ̃a koŋ ŋu, eye wònana ame ƒe susu megawɔa dɔ nyuie o, eƒe susu me gblẽna, eye eƒe nuwɔna trɔna.
Womenya nusi tututu hea Alzheimer dɔlélea vɛ o, gake wosusui be domenyiŋusẽfianuwo, nuto si me ame le, kple alesi wonɔa agbee gbɔe wòtsona.
Nusiwo ate ŋu ana Alzheimer-dɔlélea nadze egɔme dometɔ aɖewoe nye:
1. Domenyiŋusẽfianuwo: Wode dzesi domenyiŋusẽfianu aɖewo siwo naa ame léa Alzheimer-dɔ, vevietɔ domenyiŋusẽfianu si nye apolipoprotein E (APOE).
2. Tsitsi: Ne ame le tsitsim la, eƒe dɔlélea ƒe nugbegblẽa nɔa dzidzim ɖe edzi, eye wokpɔa ame akpa gãtɔ le woƒe ƒe 65 xɔxɔ megbe.
3. Ƒomea me tɔwo ƒe dɔléle: Ne Alzheimer dɔlélea le ame aɖe ƒe ƒomea me la, ate ŋu ana wòate ŋu adze dɔ bɔbɔe.
4. Taɖuame: Taɖuame siwo ame xɔ kpɔ, vevietɔ esiwo nana ame buna ɖe eɖokui la, ate ŋu ana wòadze Alzheimer-dɔ bɔbɔe.
5. Dzidɔ ƒe nugbegblẽ le ame ŋu: Nɔnɔmewo abe ʋusɔgbɔdɔ, ʋumemi, kple suklidɔ ate ŋu ana be ame nava lé Alzheimer dɔlélea bɔbɔe.
6. Agbenɔnɔ ƒe akpa aɖewo: Agbenɔnɔ si me ame meʋãna le o, nuɖuɖu manyomanyo, kple susumeŋusẽ ƒe anyimanɔmanɔ ate ŋu ana wòadze dɔ bɔbɔe.
7. Dzidede: Dzidede si nɔa ahɔhɔ̃a me ate ŋu ana woadze Alzheimer-dɔléle.
8. Nu siwo naa lãmetsiwo gblẽna le ame ŋu: Ne lãmetsi siwo naa lãmetsiwo gblẽna le ame ŋu ƒe agbɔsɔsɔme kple alesi ŋutilãa te ŋu ɖea aɖi le wo ŋu ƒe agbɔsɔsɔme mesɔ o la, ate ŋu ana Alzheimer-dɔ nadze ame dzi.
9. Amyloid-kpewo kple lãmeka siwo wɔa dɔ le ahɔhɔ̃ me ƒe ƒuƒoƒo: Dɔléle sia ƒe dzesi ɖekae nye be kpewo kple lãmekawo va ƒoa ƒu ɖe ahɔhɔ̃a me, gake womese akpa si wowɔna le dɔlélea ƒe tsitsi me la gɔme bliboe o.
Ele vevie be míade dzesii be nusi gbɔ Alzheimer-dɔlélea tsona la de ŋgɔ eye ɖewohĩ nu siawo katãe wɔa dɔ ɖekae.
Wole numekuku dzi yim be woase nu si gbɔ dɔlélea tso gɔme nyuie wu eye woato edzi adae.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Mɔxeɖenu: atikewɔwɔ']
['Taɖodzinu siwo nye nufiafia kple nyatakaka koe le nyatakakadzraɖoƒe sia ŋu eye menye atikewɔwɔ ŋuti ɖaŋuɖoɖo alo dɔwɔnawo ƒe nya gblɔm wole o.']
['Mele be woazã nyatakaka siwo wona la atsɔ ada dɔléle alo dɔ aɖe o, eye ele be amesiwo di be yewoakpɔ atikewɔwɔ ŋuti ɖaŋuɖoɖo na yewo la nabia ɖɔkta si xɔ mɔɖegbalẽ.']
['De dzesii be neural network si wɔa nyabiabiawo ƒe ŋuɖoɖowo la meɖia o vevietɔ ne wotsɔ xexlẽdzesiwo wɔe. Le kpɔɖeŋu me, ne wotsɔ ame siwo ŋu dɔléle aɖe le ƒe xexlẽme wɔ dɔe.']
['Bia wò ɖɔkta alo lãmesẽdɔwɔla bubu si dze ƒe aɖaŋuɖoɖo ɣesiaɣi le lãmesẽkuxi aɖe ŋu. Mègaŋe aɖaba ƒu aɖaŋuɖoɖo si ɖɔktawo ɖo na wò alo gbɔ dzi ɖi le exexlẽ me le nyatakakadzraɖoƒe sia ta o. Ne èsusu be ɖewohĩ lãmesẽkuxi aɖe le fu ɖem na ye la, ke yɔ 911 alo yi ɖe afisi wokpɔa nɔnɔme kpatawo gbɔ le.']
['Copyright: Copyright']
['Digital Millennium Copyright Act of 1998, 17 U.S.C. § 512 (the DMCA) na mɔ siwo dzi copyright ƒe nunɔlawo ate ŋu ato awɔ nu ɖe nu siwo wo xɔ se be wo le Internet dzi la ŋu.']
['Ne èxɔe se kple kakaɖedzi be nyatakakadzraɖoƒe alo dɔwɔnu aɖe si míetsɔ wɔ míaƒe nyatakakadzraɖoƒea alo dɔwɔnawo la nye dada le wò gome la, wò ŋutɔ (alo wò dɔtɔ) ate ŋu aŋlɔ agbalẽ aɖo ɖe mí abia be míaɖe nyatakakadzraɖoƒea alo dɔwɔnua ɖa alo axe mɔ ɖe ezazã nu.']
['Ele be woana nyatakakawo to e-mail dzi (kpɔ "Kpekpeɖeŋunaƒe" ƒe akpa si nye e-mail adrɛs). ']
['DMCA bia be nàŋlɔ nyatakaka siwo gbɔna ɖe wò nyatakaka si nèŋlɔ be woada le copyright dzi la me: (1) nuŋɔŋlɔ si fia be copyright le dɔ si ŋu wole nu ƒom le la ŋu; (2) nyatakaka si fia be nuŋɔŋlɔa le eme eye wòade mía nu be míake ɖe eŋu; (3) nyatakaka siwo ana míake ɖe ŋuwò, siwo dometɔ aɖewoe nye wò adrɛs, kaƒodzesi kple e-mail adrɛs; (4) wò nya si fia be èxɔe se kple kakaɖedzi be copyright ƒe ame si tɔe nyatakakaa nye, alo eƒe dɔtɔ, alo se aɖeke meɖe mɔ ɖe eŋu o; ']
['(5) Wò ŋutɔ nàŋlɔ agbalẽ si dzi nàde asii, si me nàde se be ne mèwɔe o la, àda alakpa, atsɔ aɖo kpe edzi be nyatakaka siwo le nyatakakaa me la de pɛpɛpɛ eye be ŋusẽ le asiwò be nàʋli agbalẽ siwo ŋu wogblɔ le be woda le la ta.']
['Eye (6) ame si tɔe agbalẽa nye alo ame si wona ŋusẽe be wòawɔ dɔ le ame si tɔ ŋkɔ me la ƒe asinuŋɔŋlɔ alo eƒe asinuŋɔŋlɔ si le mɔ̃ dzi. ']
['Ne mèŋlɔ nyatakaka siwo katã le etame ɖe agbalẽa me o la, ate ŋu ana be wò nyatoƒoe me dzodzro natsi megbe.']
['Kadodo']
['Taflatse ɖo email ɖe mí ne nyabiabia alo aɖaŋuɖoɖo aɖe le asiwò.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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['Le Eŋu']
['BioMedLib zãa kɔmpiuta siwo wɔa dɔ le wo ɖokui si (mɔ̃ siwo zãa mɔ̃ɖaŋunuwo tsɔ srɔ̃a nu) tsɔ wɔa nyabiabia kple ŋuɖoɖowo.']
