Na pathophysiology ni kenisa ni yatevuso e vakaibalebaletaki ena veisau ni ivakarau ni bula kei na ka e dau yaco ena gauna e tubu tiko kina na kenisa ni yatevuso.
Na kenisa ni yatevuso e mate vereverea e vu mai na tubu kei na kena tawasei na sela ni yatevuso.
Na sela oqo e rawa ni tubu me kenisa, qai tete ina so tale na tikiniyago, e vakavuna me levu na mate kei na leqa.
Na pathophysiology ni kenisa ni yatevuso e okati kina e vica na ka, okati kina na genetic mutations, environmental factors, kei na ivakarau ni bula.
E rawa ni yaco na veisau ni gene ena DNA ni sela ni yatevuso, e vakavuna me tubu qai tawase na sela.
Na veisau oqo e rawa ni dewa mai na dua na itubutubu se na kena e dewavi koya ga, qai rawa ni vu mai na kena ceguvi na tavako, na kasi gaga na radon, na kasi na asbestos, kei na cagi gaga.
Na kenisa ni yatevuso e rawa ni wasei ina rua na mataqali: na kenisa ni yatevuso e vakatokai na small cell lung cancer (SCLC) kei na kenisa ni yatevuso e vakatokai na non-small cell lung cancer (NSCLC).
E rawa ni duidui na mataqali kenisa ni yatevuso oqo, ni duidui na kena gene e dewa, qai duidui tale ga na kena isau.
Na pathophysiology ni kenisa ni yatevuso e okati tale ga kina na nodrau cakacaka vata na sela ni kenisa kei na tikiniyago e wavoliti rau, wili kina na sotia ni yago.
E rawa ni levea na sotia ni yago na sela ni kenisa, me tubu qai tete.
Kena ikuri, na microenvironment ni tumour e rawa ni vakavuna na tubu ni tumour kei na metastasis ena kena vakarautaki e dua na vanua vinaka me bula kina na sela ni kenisa.
Na pathophysiology ni kenisa ni yatevuso e vereverea qai toso tiko ga, ra qai saga tiko ga na dauvakadidike mera kila vinaka na ka e vakavuna me rawa ni yaga kina na veiqaravi e caka kei na kena vinaka na kedra ituvaki na tauvimate.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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