Pathophysiology of diabetes jechuun qo'annoo adeemsa qaamaa wal faallessu kan dhibee sukkaaraa (diabetes mellitus) akka uumamu taasisu kan argisiisudha.
Dhibeen sukkaaraa (diabetes mellitus) garee dhukkuboota metaboliikii sukkaara dhiigaa (glucose) ol'aanaa ta'een kan mul'atu yoo ta'u, kun ammoo hanqina insulin secretion, insulin action, ykn lamaanuu irraa kan ka'edha.
Dhukkubni sukkaaraa (diabetes) baay'ee kan walqabatu si'a ta'u, dhukkubni kun akka babal'atu kan taasisu wantoota jiinii, naannoofi akkaataa jireenyaa wajjin walqabatanidha.
Dhibee sukkaaraa gosa 1 keessatti, pathophysiology'n balleessaa autoimmune of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production kan dabalatuudha.
Kunis sadarkaa gulukoosii dhiigaa sirreessuu dadhabuu fi hyperglycemia (sukkaara dhiigaa ol ka'uu) fiduu danda'a.
Dhibee sukkaaraa gosa 2 keessatti, pathophysiology baay'ee walxaxaa kan ta'e yoo ta'u, insulin resistance fi impaired insulin secretion ni dabalata.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numerical content. For example, the number of people diagnosed with a specific disease.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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