Pathophysiology of prostate cancer (pathophysiology of prostate cancer) tooftaa fi adeemsa bu'uura ta'e kan guddina fi guddina dhukkubichaa geessu kan argisiisudha.
Pathophysiology of prostate cancer kun guddinaa fi qoqqoobbii seelota prostate gland keessatti to'atamuu hin dandeenye kan dabalatuudha.
Kun kan ta'u sababa jijjirama jiiniitiin kan ka'e baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina baay'ina.
Jijjiiramni kun guddina seelii to'atamaa hin taane akka uumamu kan taasisu si'a ta'u, kunimmoo kaansarichi akka uumamu gochuu danda'a.
Dhukkubni kun yommuu guddatu, qaamota fi qaamota naannoo sana jiran, kan akka burqaa, reektamiifi laafaa naannoo sana jiran irratti dhiibbaa geessisuu dandaʼa.
Teestoostirooniin guddina seelii kaansarii proostaatii kakaasuu kan danda'u yoo ta'u, wal'aansi kaansarii proostaatii baay'een sadarkaa hormoonii kana hir'isuuf ykn bu'aa isaa dhabamsiisuuf kan qophaa'edha.
Walumaagalatti, pathophysiology of prostate cancer kun guddinaa fi qoqqoodama seelota prostate gland keessatti to'atamuu hin dandeenye kan dabalatu yoo ta'u, kun ammoo genetic, hormonal, fi environmental factors'n dhiibbaa geessisuu danda'a.
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Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.
Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.
Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numerical content. For example, the number of people diagnosed with a specific disease.
Yeroo hunda waa'ee dhukkuba tokkoo gorsa ogeessa fayyaa ykn ogeessa fayyaa ga'eessa ta'e biraa gaafadhu. Gorsa fayyaa ogeessaa hin tuffatin ykn waan weebsaayitii kana irratti dubbisteef isa barbaaduu hin daangeffatin. Yoo rakkoo fayyaa qabaachuu dandeessan yoo ta'e, hatattamaan 911 bilbilaa ykn gara kutaa ariifachiisaa dhihoo deemaa. Weebsaayitii kana ykn itti fayyadama isaatiin hariiroon ogeessa fayyaa fi dhukkubsataa hin uumamu. BioMedLib ykn hojjettoonni isaa, ykn namni weebsaayitii kana irratti gumaache kamiyyuu, odeeffannoo asitti kenname ykn itti fayyadama isaa ilaalchisee ibsa ykn yaada hin kennine hin kennine.
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What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.
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