Tha galar Alzheimer na eas-òrdugh neurodegenerative adhartach a bheir buaidh air an eanchainn sa chiad àite, ag adhbhrachadh call cuimhne, crìonadh inntinneil, agus atharrachaidhean giùlain.
Chan eil fios dè dìreach a tha ag adhbhrachadh galar Alzheimer, ach thathas a 'creidsinn gu bheil e mar thoradh air measgachadh de fhactaran ginteil, àrainneachd, agus dòigh-beatha.
Tha cuid de na factaran a dh 'fhaodadh cur ri leasachadh galar Alzheimer a' toirt a-steach:
1. Ginteil: Chaidh ginean sònraichte a chomharrachadh a tha a'meudachadh cunnart galar Alzheimer a leasachadh, gu sònraichte an gine apolipoprotein E (APOE).
2. Aois: Bidh an cunnart bho bhith a 'leasachadh galar Alzheimer a' meudachadh le aois, leis a'mhòr-chuid de dhaoine air an dearbhadh às deidh aois 65.
3. Eachdraidh teaghlaich: Ma tha eachdraidh teaghlaich de ghalar Alzheimer ann faodaidh e an cunnart a bhith a 'leasachadh an t-suidheachaidh a mheudachadh.
4. Leòntan cinn: Faodaidh eachdraidh de leòntan cinn, gu sònraichte an fheadhainn a tha a 'leantainn gu call mothachaidh, an cunnart bho ghalar Alzheimer a mheudachadh.
5. Factaran cunnart cardiovascular: Faodaidh tinneasan leithid bruthadh-fala àrd, cholesterol àrd, agus tinneas an t-siùcair an cunnart a bhith a 'leasachadh galar Alzheimer.
6. Factaran dòigh-beatha: Faodaidh dòigh-beatha sedentary, droch dhaithead, agus dìth brosnachaidh inntinn an cunnart bho ghalar Alzheimer a mheudachadh.
7. Inflammation: Faodaidh sèid leantainneach san eanchainn cur ri leasachadh galar Alzheimer.
8. Strus oxidative: Dh 'fhaodadh mì-chothromachadh eadar cinneasachadh radain an-asgaidh agus comas a' chuirp an toirt air falbh a bhith a 'cur ri leasachadh galar Alzheimer.
9. Placaichean Amyloid agus tangles neurofibrillary: Tha cruinneachadh de phlacaichean amyloid agus tangles neurofibrillary san eanchainn na chomharra air galar Alzheimer, ach chan eil làn thuigse air a 'phàirt a tha aca ann a bhith a' leasachadh an t-suidheachaidh.
Tha e cudromach a bhith mothachail gu bheil adhbhar galar Alzheimer iom-fhillte agus is dòcha gu bheil e a 'toirt a-steach measgachadh de na factaran sin.
Tha rannsachadh a' dol air adhart gus tuigse nas fheàrr fhaighinn air na h-innealan bunaiteach agus leigheasan èifeachdach a leasachadh airson an t-suidheachaidh sgriosail seo.
Vellas B, Andrieu S, Cantet C, Dartigues JF, Gauthier S: Long-term changes in ADAS-cog: what is clinically relevant for disease modifying trials in Alzheimer? J Nutr Health Aging. , 11 (4): 338-41.
Braak E, Griffing K, Arai K, Bohl J, Bratzke H, Braak H: Neuropathology of Alzheimer's disease: what is new since A. Alzheimer? Eur Arch Psychiatry Clin Neurosci. 1999, 249 Suppl 3 (): 14-22.
Kovacs GG: Can Creutzfeldt-Jakob disease unravel the mysteries of Alzheimer? Prion. 2016, 10 (5): 369-376.
Cankurtaran M, Yavuz BB, Cankurtaran ES, Halil M, Ulger Z, Ariogul S: Risk factors and type of dementia: vascular or Alzheimer? Arch Gerontol Geriatr. , 47 (1): 25-34.
Mahami-Oskouei M, Hamidi F, Talebi M, Farhoudi M, Taheraghdam AA, Kazemi T, Sadeghi-Bazargani H, Fallah E: Toxoplasmosis and Alzheimer: can Toxoplasma gondii really be introduced as a risk factor in etiology of Alzheimer? Parasitol Res. 2016, 115 (8): 3169-74.
['Àicheadh: meidigeach']
["Tha an làrach-lìn seo air a thoirt seachad airson adhbharan foghlaim agus fiosrachaidh a-mhàin agus chan eil e a' toirt seachad comhairle mheidigeach no seirbheisean proifeiseanta."]
["Cha bu chòir am fiosrachadh a chaidh a thoirt seachad a chleachdadh airson duilgheadas slàinte no galar a dhearbhadh no a làimhseachadh, agus bu chòir dhaibhsan a tha a'sireadh comhairle meidigeach pearsanta co-chomhairle a chumail ri lighiche-sprèidh."]
["Thoiribh fa-near gu bheil an lìon neural a tha a' cruthachadh fhreagairtean do na ceistean, gu sònraichte neo-chruinneasach nuair a thig e gu susbaint àireamhach. Mar eisimpleir, an àireamh de dhaoine a chaidh a dhearbhadh le galar sònraichte."]
["Faigh comhairle an dotair agad no solaraiche slàinte eile a tha airidh air comhairle a thaobh tinneas meidigeach. Na dì-meas comhairle meidigeach proifeiseanta no dàil ann a bhith ga shireadh air sgàth rudeigin a leugh thu air an làrach-lìn seo. Ma tha thu a'smaoineachadh gum faodadh èiginn meidigeach a bhith agad, cuir fòn gu 911 no rachaibh chun t-seòmar èiginn as fhaisge sa bhad. Chan eil dàimh dotair-euslainteach air a chruthachadh leis an làrach-lìn seo no a chleachdadh. Chan eil BioMedLib no a luchd-obrach, no neach sam bith a tha a' cur ris an làrach-lìn seo, a 'dèanamh riochdachaidhean sam bith, fosgailte no a' toirt a-steach, a thaobh an fhiosrachaidh a tha air a thoirt seachad an seo no a chleachdadh."]
['Àicheadh: còraichean']
["Tha Achd Dlighe-sgrìobhaidh na Mìle Bliadhna Didseatach 1998, 17 U.S.C. § 512 (an DMCA) a' toirt ath-chuinge do shealbhadairean dlighe-sgrìobhaidh a tha den bheachd gu bheil stuth a tha a' nochdadh air an eadar-lìon a' briseadh an còraichean fo lagh dlighe-sgrìobhaidh na SA. "]
["Ma tha thu a' creidsinn ann an deagh chreideamh gu bheil susbaint no stuth sam bith a tha ri fhaotainn a thaobh ar làrach-lìn no ar seirbheisean a' briseadh d' dlighe-sgrìobhaidh, faodaidh tu fhèin (no an riochdaire agad) fios a chur thugainn ag iarraidh gun tèid an susbaint no an stuth a thoirt air falbh, no gun tèid cothrom a chur air. "]
["Feumar sanasan a chur ann an sgrìobhadh tro phost-d (faic an roinn 'Contact' airson seòladh puist-d). "]
["Tha an DMCA ag iarraidh gum bi am fiosrachadh a leanas anns an fhios agad mu bhriseadh còraichean-sgrìobhaidh a thathar ag ràdh: (1) tuairisgeul air an obair fo chòraichean-sgrìobhaidh a tha fo chasaid briseadh; (2) tuairisgeul air an t-susbaint a thathar ag ràdh a tha a 'briseadh agus fiosrachadh gu leòr gus leigeil leinn an t-susbaint a lorg; (3) fiosrachadh conaltraidh dhut, a' toirt a-steach do sheòladh, àireamh fòn agus seòladh puist-d; (4) aithris bhuat gu bheil creideas math agad nach eil an t-susbaint san dòigh a tha gearan air a cheadachadh le sealbhadair a 'chòraichean-sgrìobhaidh, no an neach-ionaid aige, no le gnìomh lagha sam bith; "]
['(5) Dearbhadh agaibh, air a shoidhnigeadh fo pheanas mionnachaidh, gu bheil am fiosrachadh san fhiosrachadh ceart agus gu bheil ùghdarras agaibh na còraichean-sgrìobhaidh a thathar ag ràdh a chaidh a bhriseadh a chur an gnìomh; ']
['agus (6) ainm-sgrìobhte corporra no eileagtronaigeach an t-sealbhadair dlighe-sgrìobhaidh no neach a tha ùghdarraichte a bhith ag obair às leth an t-sealbhadair dlighe-sgrìobhaidh. ']
["Ma dh'fhailicheas tu air a' ghearan agad a dheasachadh, faodaidh e dàil a chur air mura cuir thu a-steach am fiosrachadh gu lèir gu h-àrd."]
['Cuir fios thugainn']
['Cuir post-d thugainn le ceist / moladh sam bith.']
What causes alzheimer?
Alzheimer's disease is a progressive neurodegenerative disorder that primarily affects the brain, causing memory loss, cognitive decline, and behavioral changes.
The exact cause of Alzheimer's disease is not fully understood, but it is believed to result from a combination of genetic, environmental, and lifestyle factors.
Some of the factors that may contribute to the development of Alzheimer's disease include:
1. Genetics: Certain genes have been identified that increase the risk of developing Alzheimer's disease, particularly the apolipoprotein E (APOE) gene.
2. Age: The risk of developing Alzheimer's disease increases with age, with most people being diagnosed after the age of 65.
3. Family history: Having a family history of Alzheimer's disease may increase the risk of developing the condition.
4. Head injuries: A history of head injuries, particularly those that result in loss of consciousness, may increase the risk of Alzheimer's disease.
5. Cardiovascular risk factors: Conditions such as high blood pressure, high cholesterol, and diabetes may increase the risk of developing Alzheimer's disease.
6. Lifestyle factors: A sedentary lifestyle, poor diet, and lack of mental stimulation may increase the risk of Alzheimer's disease.
7. Inflammation: Chronic inflammation in the brain may contribute to the development of Alzheimer's disease.
8. Oxidative stress: An imbalance between the production of free radicals and the body's ability to detoxify them may contribute to the development of Alzheimer's disease.
9. Amyloid plaques and neurofibrillary tangles: The accumulation of amyloid plaques and neurofibrillary tangles in the brain is a hallmark of Alzheimer's disease, but the role they play in the development of the condition is not fully understood.
It is important to note that the cause of Alzheimer's disease is complex and likely involves a combination of these factors.
Research is ongoing to better understand the underlying mechanisms and develop effective treatments for this devastating condition.
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["Bidh BioMedLib a' cleachdadh choimpiutairean fèin-ghluasadach (algorithms ionnsachaidh innealan) gus paidhrichean ceist is freagairt a chruthachadh."]
["Bidh sinn a' tòiseachadh le 35 millean foillseachadh bith-leigheis de PubMed/Medline. Cuideachd, duilleagan-lìn de RefinedWeb."]
